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NSG 3280 Exam 2 Complete Solutions 2026/2027 Updated | Instant Download | Integumentary & Infectious Disorders Exam Review

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This document contains NEW NSG 3280 Exam 2 complete solutions for the 2026/2027 academic year. It covers key pathophysiology concepts related to herpes simplex virus (HSV-1 and HSV-2), herpes zoster (shingles), antiviral treatments, shingles vaccination guidelines, clinical manifestations, fungal infections, and common integumentary disorders such as tinea pedis. The material is organized in a clear question-and-answer format to promote strong concept mastery and exam readiness. Ideal for nursing students preparing for Pathophysiology Exam 2, ATI-style assessments, and infectious disease and integumentary system testing aligned with current curriculum standards.

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NEW NSG 3280 EXAM 2 WITH COMPLETE SOLUTIONS 2026-2027!!



Etiology of herpes
Herpes virus contact withskin and mucous membranes; persists in latentform in
trigeminal nerve and other ganglia
HSV-1
Occurs above the waist; common on the lips,face, and mouth• Healing in 10 to 14
days;
persists in a latent form
Stress can exacerbate it
Contact transmission
no cure
HSV-2
Responsible for most infections in the genital region
sexually/contact transmitted
Clinical manifestations of HSV
Usually begins with burning or tingling sensation, followed by pustules, ulcers,
vesicles, and erythema; pain common
Is there a cure for herpes?
No but there is treament to keep breakouts under control or stop them completely
What is the treatment for HSV?
Antivirals like acyclovir.
Herpes zoster (shingles)
Acute inflammatory disease of a dermatomal segment of the skin
along the spinal nerves or spinal tract (Dorsal root ganglia)
Shingles vaccine is available and for what age?
50
What does shingles look like?
Cluster of fluid-filled blisters, often in a band
erythematous bases
usually unilaterally
typically in patients over 60 or people with weak immune support
Mid torso, shoulder/neck, or side of face
Shingles treatments
Gabapentin for severe cases
Fungal infections etiology
Microsporum, trichophyton, epidermophyton
Tinea pedis
Fungal infection of the foot; athlete's foot

, Tinea capitis
Ringworm of the scalp
Clinical manifestation of fungal infections
Erythematous•
Macules/papules or plaques with peripheral scalingand central clearing•
Vesicular lesions•
Nail thickening and discoloration
Yeast infections
Undiagnosed immunodeficiencydisorder of superficial infection of skin
and/ormucous membranes
Monitor overuse of antibiotics because __________
This can cause a disruption of normal flora
Thrush
Oral candidiasis in the newborn
can happen in adults too
Intertrigo
Chafing of the skin
Impetigo
Staphaureus or strep
Clinical manifestation of impetigo
O Bullous: veiscles, pustules → golden honey-colored crust, MC type

o Nonbullous: rapidly progresses from small to large bullae → rupture "varnish
like crusts" → fever, diarrhea

HIGHLY INFECTIOUS

Becomes non -infectious when it dries up
Seborrheic dermatitis
An inflammation that causes scaling and itching of the upper layers of the skin or
scalp
high oil production
Seborrheic dermatitis clinical manifestations
Various degrees of scaling and erythema in areas of high oil gland concentration;
for example cradle cap and dandruff

around nose and face areas
Psoriasis
Unknown cause, possibly autoimmune
chronic condition of a silvery, scaly white patches

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