NURS 143 EXAM REVIEW QUESTIONS WITH
VERIFIED SOLUTIONS
How do doctor treat hgih sodium
gradually reducing iso to hypo tonic solutions, so we don't risk cerebral edema, if DI vasopressin
might be ordered, diruretics also might be
nursing high sodium
thirst body temp mental status seziures twitches all vitals encourage fluids
potassium level
3.5-5
potassium level
3.5-5
what is potassium all about
muscle
where is potassium mostly
in cells, cell damage makes it go out
regulate K
kidneys, more likely to be lost in urine, vomit, diarrhea, sweat
what does alka do to k
moves it into cells
what med causes hypok
diuretics more so loop
what intervention can cause low k
suctioning
potassium lack symptoms
shallow breathing irritable confused/drowsy weak tired heart issues irregular and too fast or slow
lethargythready pulse gi issues n/v ileus in
what commonly comes with low k
metabolic alkalosis
, what should you antipate as the nurse for low k
cardiac monitoring
why should infusing kcl be carefully monitored
vesicant and likely cardiac effects
what medication is potentiated by low k
digitalis
hyperkalemia
cardiac arrest potential, could be from cell injury as k moves ito ECF or kidney injury or intake
symptoms of high potassium
irritable, twitching flaccid parlysis dysrrhythmia bradycardia, cramping diarrhea
acidosis increases
potassium levels (because hydrogen is kickiing potassium out)
what is the priority for monitoring high potassium
EC
med interventions hyperkalemia
calcium gluconate, sodium bicarb, iv insulin diuretics
hypocalcemia
less than 8.6 m
calcium levels
8.6-10.5
hypocalcemia
citrated blood, issues with parathyroid gland, pancreatitis, elevanted phosphate
when phosphorous is up
calcium is down
when calcium is up
phos is down
calcium low
cats go numb convulsions arrhthmis tetany spasms/stridor finger numbness
VERIFIED SOLUTIONS
How do doctor treat hgih sodium
gradually reducing iso to hypo tonic solutions, so we don't risk cerebral edema, if DI vasopressin
might be ordered, diruretics also might be
nursing high sodium
thirst body temp mental status seziures twitches all vitals encourage fluids
potassium level
3.5-5
potassium level
3.5-5
what is potassium all about
muscle
where is potassium mostly
in cells, cell damage makes it go out
regulate K
kidneys, more likely to be lost in urine, vomit, diarrhea, sweat
what does alka do to k
moves it into cells
what med causes hypok
diuretics more so loop
what intervention can cause low k
suctioning
potassium lack symptoms
shallow breathing irritable confused/drowsy weak tired heart issues irregular and too fast or slow
lethargythready pulse gi issues n/v ileus in
what commonly comes with low k
metabolic alkalosis
, what should you antipate as the nurse for low k
cardiac monitoring
why should infusing kcl be carefully monitored
vesicant and likely cardiac effects
what medication is potentiated by low k
digitalis
hyperkalemia
cardiac arrest potential, could be from cell injury as k moves ito ECF or kidney injury or intake
symptoms of high potassium
irritable, twitching flaccid parlysis dysrrhythmia bradycardia, cramping diarrhea
acidosis increases
potassium levels (because hydrogen is kickiing potassium out)
what is the priority for monitoring high potassium
EC
med interventions hyperkalemia
calcium gluconate, sodium bicarb, iv insulin diuretics
hypocalcemia
less than 8.6 m
calcium levels
8.6-10.5
hypocalcemia
citrated blood, issues with parathyroid gland, pancreatitis, elevanted phosphate
when phosphorous is up
calcium is down
when calcium is up
phos is down
calcium low
cats go numb convulsions arrhthmis tetany spasms/stridor finger numbness
