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PATHO 370 Midterm Exam Version B Newest 2026
Actual Verified Exam 300 Questions And Correct
Detailed Answers (verified Answers) ||Already Graded
A+||Newest Exam!!!
A restrictive respiratory disorder is characterized by:
-increased total lung capacity
-decreased residual volume
-inspiratory wheezing
-expiratory wheezing - ANSWER-DEC RESIDUAL VOL
Restrictive respiratory disorders are characterized by
decreased residual volume. Restrictive respiratory
disorders are not characterized by increased residual
volume. Inspiratory and expiratory wheezing are not
characteristic of restrictive respiratory disorder.
Angiotensin-converting enzyme (ACE) inhibitors block the:
-release of rennin
-conversion of angiotensin I to angiotensin II
-conversion of angiotensinogen to angiotensin I
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-effect of aldosterone on the kidney - ANSWER-
CONVERSION OF ANGIOTENSIN I TO ANGIOTENSIN II
Angiotensin I is converted into angiotensin II while it is
circulating through the pulmonary vessels, by the
angiotensin-converting enzyme. ACE inhibitors block the
conversion of angiotension I to angiotension II. Renin
plays a role in the regulation of arterial blood pressure.
ACE inhibitors do not block the conversion of
angiotensinogen to angiotensin or the effect of
aldosterone on the kidney.
If an individual has a fully compensated metabolic
acidosis, the blood pH is:
-high
-low
-in the normal range
-either high or low, depending on the type of compensation
- ANSWER-IN THE NORMAL RANGE
The blood pH is in the normal range if an individual has
fully compensated for an acid-base imbalance. High blood
pH indicates alkalosis. Low blood pH indicates
uncompensated or partially compensated acidosis.
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Chronic bronchitis often leads to cor pulmonale because
of:
-ventricular hypoxia
-increased pulmonary vascular resistance
-left ventricular strain
-hypervolemia - ANSWER-INC PULMONARY VASCULAR
RESISTANCE
Chronic bronchitis often leads to cor pulmonale as a result
of increased pulmonary vascular resistance when right
ventricular end-diastolic pressure increases. Ventricular
hypoxia, left ventricular strain, and hypervolemia do not
lead to cor pulmonale.
An increase in organ size and function caused by
increased workload is termed:
-atrophy
-hypertrophy
-metaplasia
-inflammation - ANSWER-HYPERTROPHY
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Increased function of an organ such as the heart or
skeletal muscle results in organ hypertrophy because of
cellular enlargement. Atrophy refers to reduction in size of
an organ because of cellular shrinkage. Metaplasia refers
to replacement of one differentiated cell type with another.
Inflammation results from immune response rather than
workload.
Atherosclerotic plaques with large lipid cores are prone to
-dislodgement
-binding
-rupture
-attachment - ANSWER-RUPTURE
Rupture of atherosclerotic plaques with large lipid cores
initiates platelet aggregation and thrombus formation.
Dislodgement is not an occurrence of atherosclerotic
plaques with large lipid cores. Atherosclerotic plaques with
large lipid cores are not prone to binding. Large lipid cores
of atherosclerotic plaques are not prone to attachment.
A patient exhibiting respiratory distress as well as a
tracheal shift should be evaluated for:
PATHO 370 Midterm Exam Version B Newest 2026
Actual Verified Exam 300 Questions And Correct
Detailed Answers (verified Answers) ||Already Graded
A+||Newest Exam!!!
A restrictive respiratory disorder is characterized by:
-increased total lung capacity
-decreased residual volume
-inspiratory wheezing
-expiratory wheezing - ANSWER-DEC RESIDUAL VOL
Restrictive respiratory disorders are characterized by
decreased residual volume. Restrictive respiratory
disorders are not characterized by increased residual
volume. Inspiratory and expiratory wheezing are not
characteristic of restrictive respiratory disorder.
Angiotensin-converting enzyme (ACE) inhibitors block the:
-release of rennin
-conversion of angiotensin I to angiotensin II
-conversion of angiotensinogen to angiotensin I
,2|Page
-effect of aldosterone on the kidney - ANSWER-
CONVERSION OF ANGIOTENSIN I TO ANGIOTENSIN II
Angiotensin I is converted into angiotensin II while it is
circulating through the pulmonary vessels, by the
angiotensin-converting enzyme. ACE inhibitors block the
conversion of angiotension I to angiotension II. Renin
plays a role in the regulation of arterial blood pressure.
ACE inhibitors do not block the conversion of
angiotensinogen to angiotensin or the effect of
aldosterone on the kidney.
If an individual has a fully compensated metabolic
acidosis, the blood pH is:
-high
-low
-in the normal range
-either high or low, depending on the type of compensation
- ANSWER-IN THE NORMAL RANGE
The blood pH is in the normal range if an individual has
fully compensated for an acid-base imbalance. High blood
pH indicates alkalosis. Low blood pH indicates
uncompensated or partially compensated acidosis.
,3|Page
Chronic bronchitis often leads to cor pulmonale because
of:
-ventricular hypoxia
-increased pulmonary vascular resistance
-left ventricular strain
-hypervolemia - ANSWER-INC PULMONARY VASCULAR
RESISTANCE
Chronic bronchitis often leads to cor pulmonale as a result
of increased pulmonary vascular resistance when right
ventricular end-diastolic pressure increases. Ventricular
hypoxia, left ventricular strain, and hypervolemia do not
lead to cor pulmonale.
An increase in organ size and function caused by
increased workload is termed:
-atrophy
-hypertrophy
-metaplasia
-inflammation - ANSWER-HYPERTROPHY
, 4|Page
Increased function of an organ such as the heart or
skeletal muscle results in organ hypertrophy because of
cellular enlargement. Atrophy refers to reduction in size of
an organ because of cellular shrinkage. Metaplasia refers
to replacement of one differentiated cell type with another.
Inflammation results from immune response rather than
workload.
Atherosclerotic plaques with large lipid cores are prone to
-dislodgement
-binding
-rupture
-attachment - ANSWER-RUPTURE
Rupture of atherosclerotic plaques with large lipid cores
initiates platelet aggregation and thrombus formation.
Dislodgement is not an occurrence of atherosclerotic
plaques with large lipid cores. Atherosclerotic plaques with
large lipid cores are not prone to binding. Large lipid cores
of atherosclerotic plaques are not prone to attachment.
A patient exhibiting respiratory distress as well as a
tracheal shift should be evaluated for:
