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NURS 300 Exam 2 – Questions With Trusted Solutions

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NURS 300 Exam 2 – Questions With Trusted Solutions

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NURS 300
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NURS 300

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NURS 300 Exam 2 – Questions With Trusted
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methylphenidate (concerta/ritalin)
-produces CNS and respiratory stimulation
-ADHD, narcolepsy
-cardiac abnormalities, hyperthyroidism
-increased attention span for ADHD
-hypertension, tachycardia, palpitations



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1 CNS stimulant key drug #3 2 histamine receptors



3 adrenergic decongestants MOA 4 antihistamine indications



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Terms in this set (146)

,histamine receptors causes vasodilation, increased GI and respiratory
secretions, and increased capillary permeability.
two main receptors are H1 and H2


antihistamines drugs that directly compete with histamine for
specific receptor sites and act as H1 blockers


Antihistamines MOA binding at unoccupied receptors and are more
effective at preventing histamine effects than
reversing them


antihistamine properties reduce capillary permeability, itching, wheal-and-
flare formation, have drying effects, and may cause
sedation


antihistamine indications used for palliative treatment of nasal allergies,
allergic rhinitis, motion sickness, PD, sleep disorders,
sneezing, and runny nose


antihistamines adverse effects dry mouth, difficulty urinating, constipation, vision
changes, and drowsiness


sedating antihistamines older antihistamines that work peripherally and
centrally and have anticholinergic effects, making
them more effective in some cases


example of sedating antihistamine Diphenhydramine (Benadryl)


nonsedating antihistamines developed to reduce unwanted sedation; work
peripherally with fewer CNS effects and have a
longer duration of action


examples of nonsedating Fexofenadine (Allegra)
antihistamines Loratadine (Claritin)
Cetirizine (Zyrtec)


what are the 3 main types of adrenergics, anticholinergics, corticosteroids
decongestants

, decongestants indications used for relief of nasal congestion from rhinitis,
common cold, sinusitis, hay fever, and other allergies


adrenergic decongestants MOA constrict small blood vessels in nasal tissues,
shrinking swollen mucous membranes and improving
drainage


oral adrenergic decongestants have prolonged effects but delayed onset, are less
potent than topical, and do not cause rebound
congestion


example of oral adrenergic Pseudoephedrine (Sudafed)
decongestants


topical adrenergic decongestants have prompt onset and are potent, but sustained use
causes rebound congestion


example of topical adrenergic Phenylephrine (Neo-Synephrine)
decongestants


Corticosteroid decongestants MOA Blocks local inflammatory response and decreases
congestion


examples of corticosteroids nasal Beclomethasone dipropionate, flunisolide (Nasalide),
decongestants fluticasone (Flonase)


Decongestants adverse effects nervousness, insomnia, palpitations, tremors, and
systemic effects; may cause mucosal dryness and
irritation


productive cough a congested cough that removes excessive
secretions


nonproductive cough a dry cough without mucus production


antitussives drugs that stop or reduce coughing and are used
only for nonproductive coughs or when coughing is
harmful

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