Edapt Pathophysiology Test Bank| Complete questions and 100% correct answers| Latest Update
Edapt Pathophysiology Questions Week 1-4| Edapt Pathophysiology Test Bank| Complete questions and 100% correct answers| Latest Update Characteristics of Systolic HF: A. Heart failure with ejection fraction ≥ 50% B. Heart failure with ejection fraction ≤ 40% C. Pulmonary congestion without cardiomegaly on chest X-ray D. Pulmonary congestion with cardiomegaly on chest X-ray E. Increased size of left ventricle F. Decreased size of left ventricle G. S3 gallop H. S4 gallop B, D, E, G In a primary care office, a nurse practitioner (NP) assesses a client who reports taking a new medication 30 minutes before arrival. The NP is concerned that the client may be having a hypersensitivity reaction. Which clinical manifestations should the NP expect Hypertension Wheezing Bradycardia Diaphoresis Urticaria Vomiting Urticaria, wheezing, vomiting, and diaphoresis Immediate hypersensitivity is mediated by IgE antibodies, which result in an allergy, anaphylaxis, or atopic disease. The NP should expect the client to have a type 1 hypersensitivity to recent medication use, which can include these immediate reactions as clinical manifestations: urticaria, wheezing, vomiting, and diaphoresis. Hypertension and bradycardia are not associated with immediate hypersensitivity reactions. ____________ are the primary effector cells and responsible for initiating and mediating _____________ hypersensitivity. Eosinophils, Neutrophils, Mast cells, or T-cells Type 1, 2, 3, or 4 Mast cells are the primary effector cells and responsible for initiating and mediating type 1 hypersensitivity reactions. Characterized by the rapid release of proinflammatory mediators like histamine, leukotrienes, and cytokines in response to allergen exposure, mast cells are the primary effector cells responsible for initiating and mediating type 1 hypersensitivity reactions. ____________ hypersensitivity reactions involve the formation of _____________ that can deposit in tissues, leading to complement activation, inflammation, and tissue destruction. Type 1, 2, 3, or 4 IgE, IgM, IgG, immune complexes Type 3 hypersensitivity reactions involve the formation of immune complexes that can deposit in tissues, leading to complement activation, inflammation, and tissue destruction. Type 3 hypersensitivity reactions involve the formation of immune complexes that can deposit in tissues, leading to complement activation and inflammation. This process can cause tissue damage and is associated with systemic lupus erythematosus (SLE) and serum sickness. Type 1 reactions are mediated by IgE antibodies, and type 2 are mediated by IgG or IgM antibodies. Type 4 reactions are activated by T-helper cells. Highlight the finding(s) the nurse practitioner (NP) recognizes as risk factors that may contribute to the client's new diagnosis of allergic rhinitis. Select all that apply. Camille Rutherford, 45-years-old Chief Complaint: red, dry, itchy skin on arms and legs, shortness of breath, wheezing, and cough Medical History: presents with a nonproductive cough, expiratory wheezing, and shortness of breath upon exertion; reports a gradual onset of these symptoms and mentioned that they have been progressively worsening when walking their dog outside Past Medical History: eczema, hypertension Social History: lives at home with daughter and their dog cough, wheezing, SOB, gradual onset, outside, eczema, dog Allergic rhinitis attacks are related to ongoing exposure to specific offending agents. The strongest risk factor for developing asthma is a history of atopic disease (the client has eczema, a form of atopic dermatitis). Environmental factors and allergens—such as high humidity, cold, dry weather, house dust mites, pet fur, and pollen—can place a client at risk for a new diagnosis of allergic asthma. With prior exposure to allergens, Camille was sensitized. Chronic exposure to allergens mediated IgE antibodies to attach to sensitized cells, and with further exposure, IgE caused sensitized cells to degranulate. When degranulation occurs, inflammatory mediators like histamine, leukotrienes, and prostaglandins are released to produce several effects on the body, such as shortness of breath and wheezing. Constriction of bronchial smooth muscle also occurs, which explains her respiratory symptoms: shortness of breath, cough, and wheezing. The NP can diagnose the client with a type I hypersensitivity reaction based on localized and systemic symptoms. The client's age and history of hypertension are not risk factors. A 25-year-old presents to the emergency department (ED) with symptoms of ongoing weight loss, rapid heart rate, bilateral neck swelling, and hand tremors. Family medical history reveals a history of thyroid disorders. Physical examination and laboratory tests confirm the diagnosis of Graves' disease. Which mechanism below best explains the pathophysiology of Graves' disease? A. Delayed-type hypersensitivity response in the thyroid gland B. Activation of complement proteins leading to tissue damage
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