FINAL EXAMS PRN 1562 2026 FINAL
ASSESSMENT TESTED CONTENT WITH
ACCURATE SOLUTIONS
◉ left to right shunts. Answer: Congenital heart defects that cause
acyanotic congestive heart failure usually involve:
a. obstructive lesions
b. left to right shunts
c. right to left shunts
d. mixed lesions
◉ Which statement is true regarding warm autoimmune hemotlytic
anemia?
A) warm autimmune hemolytic anemia occurs primarily in men
B) it is self-limiting and rarely produces hemolysis
C) erythrocytes are bound to macrophages and sequestered in the
spleen
D) immunoglobulin M (IgM) coats erythrocytes and binds them to
receptors on monocytes. Answer: Ans: C- erythrocytes are bound to
macrophages and sequestered in the spleen
,◉ impairing alveolocapillary membrane diffusion. Answer:
Pulmonary edema and pulmonary fibrosis cause hypoxemia by:
a. creating alveolar dead space
b. impairing alveolocapillary membrane diffusion
c. creating a right to left shunt
d. decreasing the oxygen in inspired gas
◉ Hemolytic disease of the newborn (HDN) can occur if the mother:
A) is Rh-positive and the fetus is Rh-negative
B) is Rh-negative and the fetus is Rh-positive
C) has type A blood and the fetus has type O blood
D) has type AB blood and the fetus has type O blood. Answer: Ans: B-
is Rh-negative and the fetus is Rh-positive
◉ pulmonary emboli. Answer: Alveolar dead space is a result of:
a. atelectasis
b. pulmonary edema
c. pulmonary emboli
d. pneumonia
, ◉ capillary permeability. Answer: The cause of pulmonary edema in
ARDS is the result of increased:
a. oncotic pressure increase by fulminant trauma to the lungs
b. capillary hydrostatic pressure
c. levels of serum sodium and water
d. capillary permeability
◉ When diagnosed with hemolytic disease of the newborn (HDN),
why does the newborn develop hyperbilirubinemia after birth but
not in utero?
A) excretion of unconjugated bilirubin through the placenta into the
mother's circulation is no longer possible
B) hemoglobin does now break down inot bilirubin in the
intrauterin environment
C) the liver of the fetus is too immature to conjugate bilirubin from a
lipid-soluble form to a water-soluble form
D) the destruction of erythtocytes producing bilirubin is greater
after birth. Answer: Ans: A- excretion of unconjugated bilirubin
through the placenta into the mother's circulation is no longer
possible
◉ How does angiotensin II increase the workload of the heart after a
myocardial infarction (MI)?
ASSESSMENT TESTED CONTENT WITH
ACCURATE SOLUTIONS
◉ left to right shunts. Answer: Congenital heart defects that cause
acyanotic congestive heart failure usually involve:
a. obstructive lesions
b. left to right shunts
c. right to left shunts
d. mixed lesions
◉ Which statement is true regarding warm autoimmune hemotlytic
anemia?
A) warm autimmune hemolytic anemia occurs primarily in men
B) it is self-limiting and rarely produces hemolysis
C) erythrocytes are bound to macrophages and sequestered in the
spleen
D) immunoglobulin M (IgM) coats erythrocytes and binds them to
receptors on monocytes. Answer: Ans: C- erythrocytes are bound to
macrophages and sequestered in the spleen
,◉ impairing alveolocapillary membrane diffusion. Answer:
Pulmonary edema and pulmonary fibrosis cause hypoxemia by:
a. creating alveolar dead space
b. impairing alveolocapillary membrane diffusion
c. creating a right to left shunt
d. decreasing the oxygen in inspired gas
◉ Hemolytic disease of the newborn (HDN) can occur if the mother:
A) is Rh-positive and the fetus is Rh-negative
B) is Rh-negative and the fetus is Rh-positive
C) has type A blood and the fetus has type O blood
D) has type AB blood and the fetus has type O blood. Answer: Ans: B-
is Rh-negative and the fetus is Rh-positive
◉ pulmonary emboli. Answer: Alveolar dead space is a result of:
a. atelectasis
b. pulmonary edema
c. pulmonary emboli
d. pneumonia
, ◉ capillary permeability. Answer: The cause of pulmonary edema in
ARDS is the result of increased:
a. oncotic pressure increase by fulminant trauma to the lungs
b. capillary hydrostatic pressure
c. levels of serum sodium and water
d. capillary permeability
◉ When diagnosed with hemolytic disease of the newborn (HDN),
why does the newborn develop hyperbilirubinemia after birth but
not in utero?
A) excretion of unconjugated bilirubin through the placenta into the
mother's circulation is no longer possible
B) hemoglobin does now break down inot bilirubin in the
intrauterin environment
C) the liver of the fetus is too immature to conjugate bilirubin from a
lipid-soluble form to a water-soluble form
D) the destruction of erythtocytes producing bilirubin is greater
after birth. Answer: Ans: A- excretion of unconjugated bilirubin
through the placenta into the mother's circulation is no longer
possible
◉ How does angiotensin II increase the workload of the heart after a
myocardial infarction (MI)?
