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Relias - Fetal Heart Monitoring Exam 2026 ACTUAL
COMPREHENSIVE REAL VERIFIED EXAM QUESTIONS
AND CORRECT ANSWERS (VERIFIED ANSWERS)
ALREADY GRADED A+|| NEWEST EXAM!!!
Ms. Goldberg is in labor. Her fetal tracing is showing a
prolonged deceleration that has lasted 4 minutes in spite
of repositioning the patient and discontinuing the oxytocin
infusion. Her current Sa02 is 90%. Which additional
interventions could be considered at this time?
Select all that apply.
A. Vaginal examination for prolapsed cord
b. Application of oxygen via tight face mask for 15-30
minutes
c. Palpate uterus
d. Digital scalp stimulation to provoke FHR acceleration -
Answer-a, b
Although an evoked FHR acceleration can provide
reassurance regarding the absence of current fetal
acidemia, this is true only when scalp stimulation is
performed while the FHR is at baseline. Scalp stimulation
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during a deceleration can cause a vagal response, further
decreasing the FHR.
Appropriate interventions for a prolonged deceleration
include repositioning the patient, performing a vaginal
exam to rule out a cord prolapse, discontinuation of
uterine stimulants, and application of supplemental
maternal oxygen for 15-30 minutes.
Repositioning the patient may increase uterine perfusion
by alleviating the pressure of the aorta and vena cava. It
may also reposition the fetus alleviating cord compression
by the fetus.
For a sudden prolonged deceleration, it is important to
perform a vaginal exam to ensure that a cord did not
prolapse, requiring an immediate intervention to relieve
compression and cesarean delivery.
Discontinuation of uterine stimulants, such as oxytocin,
may lessen uterine contractions allowing for increased
oxygenation to the fetus.
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Short-term application of oxygen to the mother may
improve fetal oxygenation, but long-term use may be
detrimental to the fetus and should be discouraged. More
data is needed on the long-term use of oxygen on the
mother and fetus (Lyndon, Wiser 2021).
Fetal heart rate bradycardia associated with umbilical cord
prolapse, when pH would result in which of the following?
Select an answer.
a. Normal acid-base balance
b. Metabolic acidosis
c. Respiratory acidosis
d. Mixed acidosis - Answer-d. Mixed acidosis
Mixed acidosis is concurrent metabolic and respiratory
acidosis that may develop when respiratory acidosis
persists for a prolonged period of time, resulting in a large
amount of buffers being used in the physiological
compensatory effort to reduce the acidic environment.
Mixed acidosis is most often seen in prolonged
bradycardia at the time of birth.
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When fetal oxygen reserves are limited, uterine
contractions are excessive, or uteroplacental blood flow is
reduced, what negative consequences may result?
Select 2
a. Moderate FHR variability
b. Hypoxemia
c. Abnormal FHR pattern
d. Variable decelerations - Answer-b and c
Fetal hypoxemia refers to reduced oxygen in the fetal
blood, which can result from reduced fetal oxygen
reserves, excessive uterine activity, or reduced
uteroplacental blood flow. Prolonged periods of fetal
hypoxemia can lead to fetal hypoxia. Worsening fetal
hypoxemia may lead to abnormal FHR patterns--most
likely minimal or absent variability from acidemia.
Hypoxemia refers to the condition of reduced oxygen
levels in the blood.
Relias - Fetal Heart Monitoring Exam 2026 ACTUAL
COMPREHENSIVE REAL VERIFIED EXAM QUESTIONS
AND CORRECT ANSWERS (VERIFIED ANSWERS)
ALREADY GRADED A+|| NEWEST EXAM!!!
Ms. Goldberg is in labor. Her fetal tracing is showing a
prolonged deceleration that has lasted 4 minutes in spite
of repositioning the patient and discontinuing the oxytocin
infusion. Her current Sa02 is 90%. Which additional
interventions could be considered at this time?
Select all that apply.
A. Vaginal examination for prolapsed cord
b. Application of oxygen via tight face mask for 15-30
minutes
c. Palpate uterus
d. Digital scalp stimulation to provoke FHR acceleration -
Answer-a, b
Although an evoked FHR acceleration can provide
reassurance regarding the absence of current fetal
acidemia, this is true only when scalp stimulation is
performed while the FHR is at baseline. Scalp stimulation
,2|Page
during a deceleration can cause a vagal response, further
decreasing the FHR.
Appropriate interventions for a prolonged deceleration
include repositioning the patient, performing a vaginal
exam to rule out a cord prolapse, discontinuation of
uterine stimulants, and application of supplemental
maternal oxygen for 15-30 minutes.
Repositioning the patient may increase uterine perfusion
by alleviating the pressure of the aorta and vena cava. It
may also reposition the fetus alleviating cord compression
by the fetus.
For a sudden prolonged deceleration, it is important to
perform a vaginal exam to ensure that a cord did not
prolapse, requiring an immediate intervention to relieve
compression and cesarean delivery.
Discontinuation of uterine stimulants, such as oxytocin,
may lessen uterine contractions allowing for increased
oxygenation to the fetus.
,3|Page
Short-term application of oxygen to the mother may
improve fetal oxygenation, but long-term use may be
detrimental to the fetus and should be discouraged. More
data is needed on the long-term use of oxygen on the
mother and fetus (Lyndon, Wiser 2021).
Fetal heart rate bradycardia associated with umbilical cord
prolapse, when pH would result in which of the following?
Select an answer.
a. Normal acid-base balance
b. Metabolic acidosis
c. Respiratory acidosis
d. Mixed acidosis - Answer-d. Mixed acidosis
Mixed acidosis is concurrent metabolic and respiratory
acidosis that may develop when respiratory acidosis
persists for a prolonged period of time, resulting in a large
amount of buffers being used in the physiological
compensatory effort to reduce the acidic environment.
Mixed acidosis is most often seen in prolonged
bradycardia at the time of birth.
, 4|Page
When fetal oxygen reserves are limited, uterine
contractions are excessive, or uteroplacental blood flow is
reduced, what negative consequences may result?
Select 2
a. Moderate FHR variability
b. Hypoxemia
c. Abnormal FHR pattern
d. Variable decelerations - Answer-b and c
Fetal hypoxemia refers to reduced oxygen in the fetal
blood, which can result from reduced fetal oxygen
reserves, excessive uterine activity, or reduced
uteroplacental blood flow. Prolonged periods of fetal
hypoxemia can lead to fetal hypoxia. Worsening fetal
hypoxemia may lead to abnormal FHR patterns--most
likely minimal or absent variability from acidemia.
Hypoxemia refers to the condition of reduced oxygen
levels in the blood.
