NBME CBSE Actual exam COMPREHENSIVE
QUESTIONS AND VERIFIED ANSWERS
2025(GRADED A+) DETAILED ANSWERS!!
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Type II pneumocytes surfactant (lecithin)
Proliferate after injury
Type I progenitors
Neonatal Respiratory Distress Syndrome
Polio live v killed vaccine Killed = Salk = IgG
Live = Sabin = IgG + IgA
- can be shed in feces
Neonatal Respiratory Distress: Maternal DM (high insulin)
Etiology + Tx or C-section (low cortisol)
TX: dexamethasone before birth
Lung maturity determined with Amniocentesis of Phospholipids (*type II
pneumocytes)
L >> S
Type I pneumocytes Squamous gas diffusion
Elastase in lungs macrophage: lysosomes
PMN: azuronphilic granules
, Elastin stretches and recoils due to Lysine interchain crosslinks
air pressure and Air pressure = 0
intrapleural pressure at FRC Intrapleural pressure = -5
Pulm Vasc Resistance is lowest Exhale of Tidal Volume
during
Lung Compliance is decreased by LHF, pulmonary edema,
pulmonary fibrosis
Lung Compliance is increased by emphysema, age
Obesity affects ERV and FRC DECREASE
ERV & FRC
Blood flow/min (pulmonary v pulmonary = systemic
systemic)
Anatomic pulmonary shunting Bronchial circulation causes
decreased PO2 in LA/LV
than in pulmonary capillaries
More ventilation is at the BASE
O2-Hgb dissociation LEFT shift basic, cold, low 2,3 BPG
low pO2 (compensatory erythrocytosis)
O2-Hgb dissociation RIGHT shift low pH, high 2,3BPG, high T
HOT, ACIDIC
QUESTIONS AND VERIFIED ANSWERS
2025(GRADED A+) DETAILED ANSWERS!!
Edit Save
Type II pneumocytes surfactant (lecithin)
Proliferate after injury
Type I progenitors
Neonatal Respiratory Distress Syndrome
Polio live v killed vaccine Killed = Salk = IgG
Live = Sabin = IgG + IgA
- can be shed in feces
Neonatal Respiratory Distress: Maternal DM (high insulin)
Etiology + Tx or C-section (low cortisol)
TX: dexamethasone before birth
Lung maturity determined with Amniocentesis of Phospholipids (*type II
pneumocytes)
L >> S
Type I pneumocytes Squamous gas diffusion
Elastase in lungs macrophage: lysosomes
PMN: azuronphilic granules
, Elastin stretches and recoils due to Lysine interchain crosslinks
air pressure and Air pressure = 0
intrapleural pressure at FRC Intrapleural pressure = -5
Pulm Vasc Resistance is lowest Exhale of Tidal Volume
during
Lung Compliance is decreased by LHF, pulmonary edema,
pulmonary fibrosis
Lung Compliance is increased by emphysema, age
Obesity affects ERV and FRC DECREASE
ERV & FRC
Blood flow/min (pulmonary v pulmonary = systemic
systemic)
Anatomic pulmonary shunting Bronchial circulation causes
decreased PO2 in LA/LV
than in pulmonary capillaries
More ventilation is at the BASE
O2-Hgb dissociation LEFT shift basic, cold, low 2,3 BPG
low pO2 (compensatory erythrocytosis)
O2-Hgb dissociation RIGHT shift low pH, high 2,3BPG, high T
HOT, ACIDIC
