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NSG 3280(Patho) Nclex Reviews For Exam 3 | QUESTIONS AND ANSWERS

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NSG 3280(Patho) Nclex Reviews For Exam 3

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NSG 3280(Patho) Nclex Reviews For Exam 3

The physiologic change most likely to lead a. cerebral vasodilation
to an increase in intracranial pressure is


a. cerebral vasodilation.
b.hypernatremia.
c. respiratory hyperventilation.
d. REM sleep.

Manifestations of acute brain b. sympathetic nervous system activation.
ischemia (Cushing reflex) are due
primarily to


a.parasympathetic nervous system
activation.
b.sympathetic nervous system activation.
c. autoregulation of body systems.
d.loss of brainstem reflexes.
Which group of clinical findings indicates c. Assumes decorticate posture with light touch, no verbal response.
the poorest neurologic functioning?


a.Spontaneous eye opening, movement
to command, oriented to self only.
b.Eyes open to light touch on shoulder,
pupils briskly reactive to light
bilaterally.
c.Assumes decorticate posture with
light touch, no verbal response.
d.No eye opening, responds to painful
stimulus by withdrawing.

,Acceleration-deceleration movements of d. focal injuries occur in two places at opposite poles.
the head often result in polar injuries in
which:


a.injury is localized to the site of
initial impact.
b.widespread neuronal damage is incurred.
c. bleeding from venules fills the subdural
space.
d.focal injuries occur in two places at
opposite poles.


Secondary injury after head trauma refers c. brain injury resulting from the body's response to tissue damage.
to:


a.brain injury resulting from the initial
trauma.
b.focal areas of bleeding.
c. brain injury resulting from the
body's response to tissue damage.
d.injury as a result of medical therapy.

An example of inappropriate treatment for C. Hypoventilation
head trauma would be:


a.head elevation.
b.free water restriction.
c. hypoventilation.
d. bed rest.

Risk factors for hemorrhagic stroke include: c. acute hypertension.


a.atherosclerosis.
b.dysrhythmias.
c. acute hypertension.
d.sedentary lifestyle.

The stroke etiology with the a. intracranial hemorrhage
highest morbidity and mortality
is:


a.intracranial hemorrhage.
b.intracranial thrombosis.
c. intracranial embolization.
d.cardiac arrest.
Clinical manifestations of a stroke within d. left-sided muscle weakness and neglect.
the right cerebral hemisphere include:


a.cortical blindness.
b.right visual field blindness.
c. expressive and receptive aphasia.
d.left-sided muscle weakness and neglect.

, The most important preventive measure for B. blood pressure control.
hemorrhagic stroke is:


a.anticoagulation.
b. blood pressure control.
c. thrombolytics.
d.management of dysrhythmias.

In the acute phase of stroke, treatment is a. stabilization of respiratory and cardiovascular function.
focused on:


a.stabilization of respiratory
and cardiovascular function.
b.risk factor modification.
c. prevention of and contractures spasticity.
d. neurologic rehabilitation.

Cerebral aneurysm is most frequently the B. Subarachnoid hemorrhage
result of:


a.embolic stroke.
b.subarachnoid hemorrhage.
c. subdural hemorrhage.
d.meningitis.

Leakage of CS from the nose or ears is c. basilar skull fracture.
commonly associated with:


a.epidural hematoma.
b.temporal skull fracture.
c. basilar skull fracture.
d. cerebral aneurysm.

Rupture of a cerebral aneurysm should be D. sudden, severe headache
suspected if the patient reports:


a.ringing in the ears.
b.transient episodes of numbness.
c. transient episodes of vertigo.
d.sudden, severe headache.

Subarachnoid hemorrhage is usually a. cerebral vasospasm.
managed with volume expansion and
blood pressure support to
enhance cerebral perfusion. This is
necessary because subarachnoid
hemorrhage predisposes to:


a. cerebral vasospasm.
b. hypotension.
c. excessive volume loss.
d.decreased intracranial pressure.

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