Exam 5 QUESTIONS AND
VERIFIED CORRECT
ANSWERS LATEST 2026-2027
GRADED A+
Keep catheter site clean and dry
Maintain clean technique for exchanges
Notify HCP about purulent drainage
Which OTC medication would you be concerned about when assessing your patient who has
jaundice? - CORRECT ANSWER-Ibuprofen
It is important to monitor albumin for a patient who has liver cirrhosis and ascites - CORRECT
ANSWER-True
It is ok for a patient with liver cirrhosis to drink alcohol - CORRECT ANSWER-False
The patient has a K+ of 3.1. There are orders for sprionolactalone and furosemide. Give the
furosemide first. - CORRECT ANSWER-False
Lactulose is effective for the patient with liver cirrohisis and encephalopathy and decreased LOC
when: - CORRECT ANSWER-The patient is A&O x 4
,What is the purpose of a TIPS procedure? - CORRECT ANSWER-To decrease incidence of
bleeding varices
What should you have the patient do prior to having a paracentesis? - CORRECT ANSWER-Have
them urinate
Which lab value is the nurse most concerned with for a patient with pancreatitis? - CORRECT
ANSWER-Amylase
SATA: risk factors for acute pancreatitis - CORRECT ANSWER-Trauma
Excessive alcohol consumption
SATA: Pt. instructions for pancreatic enzymes(pancrealipase) should include: - CORRECT
ANSWER-Take with each meal
Wipe Lips and rinse mouth after taking
Drink a full glass of water after taking
Pantoprazole is a proton pump inhibitor - CORRECT ANSWER-True
Occult blood means blood in urine - CORRECT ANSWER-False
Trousseau's sign is due to hypocalcemia - CORRECT ANSWER-True
An NG tube may be used rest the GI system and decrease abdominal pain. - CORRECT ANSWER-
True
When might you see asterixis? - CORRECT ANSWER-Acute Liver Failure
,SATA: Which hepatitis' are fecal-oral - CORRECT ANSWER-Hep A & E
Risk of contracting Hep E is minimal when traveling to different countries and drinking an
unknown water supply - CORRECT ANSWER-False
Hepatitis B, C, and D are often caused by IV drug use - CORRECT ANSWER-True
Acute Kidney Injury (AKI) - CORRECT ANSWER-In most cases, the findings of AKI are related to
waste
buildup and decreased urine output. However, almost
every body system can be affected.
●● CARDIOVASCULAR: fluid overload (dependent and
generalized edema), dysrhythmia (hyperkalemia)
●● RESPIRATORY: crackles, decreased oxygenation,
shortness of breath
●● RENAL: scant to normal or excessive urine output,
depending on the phase; possible hematuria
●● NEUROLOGICAL: lethargy, muscle twitching, seizures
●● INTEGUMENTARY: dry skin and mucous membranes
Types of AKI - CORRECT ANSWER-Prerenal: Occurs as a result of volume depletion and
prolonged reduction of blood flow to the kidneys,
which leads to ischemia of the nephrons. Occurs before
damage to the kidney. Early intervention restoring fluid
volume deficit can reverse AKI and prevent chronic
kidney disease (CKD).
●● Intrarenal: Occurs as a result of direct damage to the
, kidney from lack of oxygen (acute tubular necrosis).
●● Postrenal: Occurs as a result of bilateral obstruction of
structures leaving the kidney.
Pre-renal AKI - CORRECT ANSWER-reduced perfusion; . Is the result of impaired blood flow that
leads to hypoperfusion of the kidney and a decrease in the GFR. (blood loss, hypovolemic shock,
hypotension from BP medications, heart attack/heart failure causing low cardiac output,
infection, liver failure, NSAID use, anaphylaxis, burns, dehydration, renal artery stenosis,
bleeding/clotting, atherosclerosis)
Intra-renal AKI - CORRECT ANSWER-damage to kidney tissue; injury to the glomeruli, nephrons,
and tubules ( bldding in kidney, emobli in kidney blood vessels, hemolytic uremic syndrome,
sepsis, pyelonephritis (infection of kidney), lupus, nephrotoxic drugs (contrast, antibiotics,
chemo), cancer, toxins, vasculitis, ischemia (due to respiratory/cardiac arrest)
Post-Renal AKI - CORRECT ANSWER-obstruction of urine flow; . Pressure rises in the kidney
tubules and eventually, the GFR decreases- bladder cancer, cervical, colon, prostate cancers,
enlarged prostate, kidney stones, neurogenic bladder, blood clots in urinary tract)
Phases of AKI - CORRECT ANSWER-Onset (Initiation phase)
Oliguric phase
Diuretic phase
Recovery phase
Non-Oliguric phase
Onset/Initiation phase - CORRECT ANSWER-decrease in urine output and increase in creat;
Begins with the initial insult and ends when oliguria develops.
increase in BUN and Creatinine that can last hours to days.
Urine output is 30 ml or less per hour- 50% of the pts. Are noted to be oliguric