🔹 1. Traumatic causes:
a. Mechanical
b. Thermal
c. Chemical
🔹 There are 2 forms of the disease :
d. Tobacco
1. Pemphigus valgaris: severe form. 🔹
e. Radiotherapy
2. Infectious causes:
🔹
2. Pemphigus vegetans: mild form.
Diagnosis:
Biopsy and microscopic examination shows:
I. Bacterial:
•Acute:
1. Pharyngitis and tonsillitis
🔹
Intra epithelial lesion with a canthosis.
Treatment:
🔹
2. Vincent’angina
🔹
{foot-hand-mouth disease}
Steroid and immunosuppressive drugs.
🔹Pemphigus:
Causes:
1.blood disease:
a. Agranulocytosis
3. Faucial diphtheria
•Chronic: 🔹 Organism: coxsakie A virus.
Clinical pictures:
🔹
It is a disease of skin and mucosa of unknown b. Acute leukemia 1. T.B. Self limited vesicular eruption that may occur in epidemics
aetiology 2.vitamine 2. Syphilis Vesicles appear in the feet, hands,buttocks and oral cavity.
A. Local causes: II. Viral: Herpangina Vesicles rupture in to form shallow ulcer with punchedout
🔹
It may be autoimmune. deficiencies:
Clinical picture:
The oral cavity show irregularly scattered large
a. Vit. A def.
b. Vit.c def.{scurvy} Stomatitis and
1. Herpangina
2. Herpes simplex 🔺
edges, grey ceater and surrounded by a deeply red areola.
To distinguished the oral vesicles from herpes simplex:
🔹
c. Riboflavin def.{vit, B2] 3. Herpes zoster The erythema and vesicles lie posteriorly effecting the
Bullous pemphegoid:
A less severe form of the disease.
bulk that rupture to form irregular painful ulcer.
3.Metabolic disorders: oropharyngeal 4. AIDS
5. Infectious mononucleosis
tonsils,pillars and soft palate while in H.simplix erythema and
vesicles are mainly anteriorly.
a. D.M.
Affects mainly children below 5 yrs and old b. Uraemia ulceration 6. Exanthemata:
🔹
above 60yrs. c. Addisons’s disease a. Vesicles of chicken pox.
Biopsy and microscopic examination show: 4.skin disease: B. Miscellaneous causes b. Koplik’s spots in measles. Organism:
Sub epithelial lesion without a canthosis. a. Pemphigus III. Fungal: The humen immunodeficiency virus.
Stomatitis with skin disease b. Erythema multiforme
c. Lichen planus 🔹
Monoliasis {candidiasis}
🔹 3. Allergic stomatitis. 🔹
Oropharyngeal manifestation:
1.kaposi sarcoma:
Clinical picture: d. Systemic lupus
🔹
🔹
4. Malignant ulcer. The most common condition.
🔹
Vesicles and bullae in the oral cavity that erythematosis 5. Dyspeptic ulcer. Raised or flat red mucosal patches which may
rupture quickly to form ulcers that are friable, 5. Recurrent 6. Precancerous lesions. ulcerate .
AIDS
🔹
bleed easily and are covered with a white aphthous stomatitis It affects buccal,gingiva, palatal and
🔹 🔹
pseudomembrane. 6. Behcet’sdisease. pharyngeal
Erythema multiform
🔹
Treatment: 7. Cancrum oris. 2.Candidiasis {oral, pharyngeal and
Topical steroid in severe cases Causes
8. Stomatitis due to oesophageal}
It is a self limited disease of the skin and
metals and drugs. Forms a painful thick white membrane
mucosa of unknown aetiology.
It may be due to delayed hypersensitivity It is prolonged and recurrent.
reaction to:
Drugs e.g pencillin
Or infection e.g mycoplasma.
It is due to chemical or contact allergy {lip
stick, mouth wash, tooth paste, ….}
Allergic stomatitis It is characterized by the formation of vesicles
that rupture to form shallow ulcer
Behcet’s disease:
It is a condition of unknown aetiology which is Precancerous lesion:{Leukoplakia}
characterized by: Causes:
1. Oral ulers. It may be due to:
2. Genital ulcers. Behcet’s disease 1. ill filling denture.
3. Iridocyclitis, conjunctivitis. 2. Dental sepsis.
It pass into remission and exacerbation. 3. Smoking.
Treatment: It appear as a raised white area usually on :
Consist of corticosteroid topical and systemic. Precancerous lesion 1. Dorsum of the tongue.
2. Inner side of the check.
3. Lower lip.
It has ill defined border and no surrounding
inflammation
M.P:
This is a rapidly spreading gangrenous
Hyperkeratosis and dysplasia.
condition usually affects young children.
The condition usually follows a
deblitating illness or is associated with
severe malnutrition e.g: following T.B.,
leukaemias or agranulocytosis.
Cancrum oris {gangrenous stomatitis}
Clinical pictures: by fatema okoff
The condition starts with ulceration of gum, tongue and
buccal mucosa.
The tissues become gangrenous and within a few days the
slough separates and teeth and the bones of the jaw are
exposed.
The condition may spread to involve the whole side of the
face reaching the eyes and ears.
It is invariably fatal.
Nowaday in the antibiotic vera, it has nearly disappeared.
Metals: mercury, lead and gold.
Drugs: epanutin, antidepressants and cytotoxic
drugs.
They cause ulcers which are large, deep,
necrotic with irregular edges and lack of
surrounding zone of inflammation.
Stomatitis due to metals and drugs
a. Mechanical
b. Thermal
c. Chemical
🔹 There are 2 forms of the disease :
d. Tobacco
1. Pemphigus valgaris: severe form. 🔹
e. Radiotherapy
2. Infectious causes:
🔹
2. Pemphigus vegetans: mild form.
Diagnosis:
Biopsy and microscopic examination shows:
I. Bacterial:
•Acute:
1. Pharyngitis and tonsillitis
🔹
Intra epithelial lesion with a canthosis.
Treatment:
🔹
2. Vincent’angina
🔹
{foot-hand-mouth disease}
Steroid and immunosuppressive drugs.
🔹Pemphigus:
Causes:
1.blood disease:
a. Agranulocytosis
3. Faucial diphtheria
•Chronic: 🔹 Organism: coxsakie A virus.
Clinical pictures:
🔹
It is a disease of skin and mucosa of unknown b. Acute leukemia 1. T.B. Self limited vesicular eruption that may occur in epidemics
aetiology 2.vitamine 2. Syphilis Vesicles appear in the feet, hands,buttocks and oral cavity.
A. Local causes: II. Viral: Herpangina Vesicles rupture in to form shallow ulcer with punchedout
🔹
It may be autoimmune. deficiencies:
Clinical picture:
The oral cavity show irregularly scattered large
a. Vit. A def.
b. Vit.c def.{scurvy} Stomatitis and
1. Herpangina
2. Herpes simplex 🔺
edges, grey ceater and surrounded by a deeply red areola.
To distinguished the oral vesicles from herpes simplex:
🔹
c. Riboflavin def.{vit, B2] 3. Herpes zoster The erythema and vesicles lie posteriorly effecting the
Bullous pemphegoid:
A less severe form of the disease.
bulk that rupture to form irregular painful ulcer.
3.Metabolic disorders: oropharyngeal 4. AIDS
5. Infectious mononucleosis
tonsils,pillars and soft palate while in H.simplix erythema and
vesicles are mainly anteriorly.
a. D.M.
Affects mainly children below 5 yrs and old b. Uraemia ulceration 6. Exanthemata:
🔹
above 60yrs. c. Addisons’s disease a. Vesicles of chicken pox.
Biopsy and microscopic examination show: 4.skin disease: B. Miscellaneous causes b. Koplik’s spots in measles. Organism:
Sub epithelial lesion without a canthosis. a. Pemphigus III. Fungal: The humen immunodeficiency virus.
Stomatitis with skin disease b. Erythema multiforme
c. Lichen planus 🔹
Monoliasis {candidiasis}
🔹 3. Allergic stomatitis. 🔹
Oropharyngeal manifestation:
1.kaposi sarcoma:
Clinical picture: d. Systemic lupus
🔹
🔹
4. Malignant ulcer. The most common condition.
🔹
Vesicles and bullae in the oral cavity that erythematosis 5. Dyspeptic ulcer. Raised or flat red mucosal patches which may
rupture quickly to form ulcers that are friable, 5. Recurrent 6. Precancerous lesions. ulcerate .
AIDS
🔹
bleed easily and are covered with a white aphthous stomatitis It affects buccal,gingiva, palatal and
🔹 🔹
pseudomembrane. 6. Behcet’sdisease. pharyngeal
Erythema multiform
🔹
Treatment: 7. Cancrum oris. 2.Candidiasis {oral, pharyngeal and
Topical steroid in severe cases Causes
8. Stomatitis due to oesophageal}
It is a self limited disease of the skin and
metals and drugs. Forms a painful thick white membrane
mucosa of unknown aetiology.
It may be due to delayed hypersensitivity It is prolonged and recurrent.
reaction to:
Drugs e.g pencillin
Or infection e.g mycoplasma.
It is due to chemical or contact allergy {lip
stick, mouth wash, tooth paste, ….}
Allergic stomatitis It is characterized by the formation of vesicles
that rupture to form shallow ulcer
Behcet’s disease:
It is a condition of unknown aetiology which is Precancerous lesion:{Leukoplakia}
characterized by: Causes:
1. Oral ulers. It may be due to:
2. Genital ulcers. Behcet’s disease 1. ill filling denture.
3. Iridocyclitis, conjunctivitis. 2. Dental sepsis.
It pass into remission and exacerbation. 3. Smoking.
Treatment: It appear as a raised white area usually on :
Consist of corticosteroid topical and systemic. Precancerous lesion 1. Dorsum of the tongue.
2. Inner side of the check.
3. Lower lip.
It has ill defined border and no surrounding
inflammation
M.P:
This is a rapidly spreading gangrenous
Hyperkeratosis and dysplasia.
condition usually affects young children.
The condition usually follows a
deblitating illness or is associated with
severe malnutrition e.g: following T.B.,
leukaemias or agranulocytosis.
Cancrum oris {gangrenous stomatitis}
Clinical pictures: by fatema okoff
The condition starts with ulceration of gum, tongue and
buccal mucosa.
The tissues become gangrenous and within a few days the
slough separates and teeth and the bones of the jaw are
exposed.
The condition may spread to involve the whole side of the
face reaching the eyes and ears.
It is invariably fatal.
Nowaday in the antibiotic vera, it has nearly disappeared.
Metals: mercury, lead and gold.
Drugs: epanutin, antidepressants and cytotoxic
drugs.
They cause ulcers which are large, deep,
necrotic with irregular edges and lack of
surrounding zone of inflammation.
Stomatitis due to metals and drugs
