Cardiac Exam 2 (LEAP) – Questions And Correct
Solutions
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Practice questions for this set
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increased peripheral vasc. resistance, vessels constricted
Choose an answer
Contraindications of ARB's (end in -
1 Parts of the heart 2
sartin)
3 increased afterload 4 Dromotropic Effect
Don't know?
Terms in this set (145)
Parts of the heart
,Coronary Circulation 1. supplies O2 and nutrients
2. heart muscle perfused during diastole (rest)
3. Left Anterior Descending (LAD) is known as widow
maker
4. Right coronary artery feeds right side of heart
Electrophysiology of the heart -SA node (pacemaker): Hearts electrical impulse
-P wave: Depolarization of R/L atria
-PR interval: Tracks the atrial impulse through the AV
node, bundle of his, and R/L bundle branches
-QRS complex: Ventricular depolarization
-ST segment- end of ventricular depolarization;
beginning of ventricular repolarization
-T wave: Ventricular Repolarization
What cardiac test displays the EKG/ECG
electrical conduction of the heart?
Dromotropic Effect causing a change in the speed of electrical
conduction in the heart (+ or -)
chronotropic effect influences heart rate, causing an ↑ or ↓
Heart conduction system (in order) SA Node 60-100 BPM
and BPM AV Node 40-60 BPM
Bundle of His
R & L Bundle Branches
Perkinjie Fibers 20-40 BPM
, Blood Pressure -Systolic (pressure on arteries during V. Contraction;
Diastolic (Rest)
- >140/90 is considered Hypertension
-Systolic - Diastolic = Pulse Pressure
-Factors that affect BP: blood volume, peripheral
vascular resistance, diameter, stress, emotions
-Primary Hypertension has no known cause;
secondary Hypertension is due to illness
RAAS System (initiated by low BV, low -Renin is produced by the kidneys -> converts
BP, and/or low sodium) angiotensinogen -> angiotensin I; ACE converts
angiotensin I -> angiotensin II in the lungs.
Angiotensin II then vasoconstricts and stimulates the
release of aldosterone. Aldosterone then promotes
Na and water retention as well as K excretion.
-Increase in BV and BP
Where do the ACE Inhibitors work in -Blocks release of ACE enzyme
relation to the RAAS? -Angio 1 cannot convert to Angio 2
-decrease in Na+, Water goes out, decrease in BP,
holds onto K
-arbs; block vasoconstriction causing vasodilation
(decrease in BP and Na+, water goes out, hold K+)
-major
What are the effect of ACE Inhibitors BP decreases, water goes out, Na+ decreases, K+ is
on aldosterone? held onto
Aliskiren -direct renin inhibitor (blocks the whole system)
- decrease in Na+, water out, decrease BP, increase in
K+
Contraindications of ARB's (end in - pregnant (BBW), hypotension, hyperkalemia
sartin)
Solutions
Save
Practice questions for this set
Learn 1 /7 Study using Learn
increased peripheral vasc. resistance, vessels constricted
Choose an answer
Contraindications of ARB's (end in -
1 Parts of the heart 2
sartin)
3 increased afterload 4 Dromotropic Effect
Don't know?
Terms in this set (145)
Parts of the heart
,Coronary Circulation 1. supplies O2 and nutrients
2. heart muscle perfused during diastole (rest)
3. Left Anterior Descending (LAD) is known as widow
maker
4. Right coronary artery feeds right side of heart
Electrophysiology of the heart -SA node (pacemaker): Hearts electrical impulse
-P wave: Depolarization of R/L atria
-PR interval: Tracks the atrial impulse through the AV
node, bundle of his, and R/L bundle branches
-QRS complex: Ventricular depolarization
-ST segment- end of ventricular depolarization;
beginning of ventricular repolarization
-T wave: Ventricular Repolarization
What cardiac test displays the EKG/ECG
electrical conduction of the heart?
Dromotropic Effect causing a change in the speed of electrical
conduction in the heart (+ or -)
chronotropic effect influences heart rate, causing an ↑ or ↓
Heart conduction system (in order) SA Node 60-100 BPM
and BPM AV Node 40-60 BPM
Bundle of His
R & L Bundle Branches
Perkinjie Fibers 20-40 BPM
, Blood Pressure -Systolic (pressure on arteries during V. Contraction;
Diastolic (Rest)
- >140/90 is considered Hypertension
-Systolic - Diastolic = Pulse Pressure
-Factors that affect BP: blood volume, peripheral
vascular resistance, diameter, stress, emotions
-Primary Hypertension has no known cause;
secondary Hypertension is due to illness
RAAS System (initiated by low BV, low -Renin is produced by the kidneys -> converts
BP, and/or low sodium) angiotensinogen -> angiotensin I; ACE converts
angiotensin I -> angiotensin II in the lungs.
Angiotensin II then vasoconstricts and stimulates the
release of aldosterone. Aldosterone then promotes
Na and water retention as well as K excretion.
-Increase in BV and BP
Where do the ACE Inhibitors work in -Blocks release of ACE enzyme
relation to the RAAS? -Angio 1 cannot convert to Angio 2
-decrease in Na+, Water goes out, decrease in BP,
holds onto K
-arbs; block vasoconstriction causing vasodilation
(decrease in BP and Na+, water goes out, hold K+)
-major
What are the effect of ACE Inhibitors BP decreases, water goes out, Na+ decreases, K+ is
on aldosterone? held onto
Aliskiren -direct renin inhibitor (blocks the whole system)
- decrease in Na+, water out, decrease BP, increase in
K+
Contraindications of ARB's (end in - pregnant (BBW), hypotension, hyperkalemia
sartin)
