DAVIS 5dADVANTAGE 5dFOR 5dPATHOPHYSIOLOGY3rd EDITION 5dCAPRIOTTI 5dTEST
5dBANK
, DAVIS 5dADVANTAGE 5dFOR 5dPATHOPHYSIOLOGY3rd EDITION 5dCAPRIOTTI 5dTEST
5dBANK
Chapter 5d1, 5dThe 5dCell 5din 5dHealth 5dand 5dIllnes
TEST 5dBANK 5dFOR 5dDAVIS 5dADVANTAGE 5dFOR 5dPATHOPHYSIOLOGY 5d3r 5ddEDITION 5dBY
5dCAPRIOTTI
Multiple 5dChoice
Identify 5dthe 5dchoice 5dthat 5dbest 5dcompletes 5dthe 5dstatement 5dor 5danswers 5dthe 5dquestion.
5 d 1. 5 d Which 5dstatement 5dregarding 5dthe 5dsodium–potassium 5dpump 5dis 5dcorrect?
1. The 5dcell’s 5dplasma 5dmembrane 5dis 5dmore 5dsoluble 5dto 5dsodium 5dions 5dthan 5dpotassium 5dions.
2. The 5dconcentration 5dof 5dsodium 5dions 5dshould 5dbe 5dhigher 5dinside 5dthe 5dcell 5dcompartment.
3. The 5dconcentration 5dof 5dpotassium 5dions 5dshould 5dbe 5dhigher 5doutside
the 5dcell 5dcompartment.
5d
4. The 5dactive 5dtransport 5dinvolves 5dpumping 5dout 5dthree 5dsodium 5dions 5dand
pumping 5din 5dtwo 5dpotassium 5dions.
5d
5 d 2. 5dIn 5dthe 5dabsence 5dof 5doxygen, 5dwhich 5dcellular 5dfunction 5dcreates 5dthe 5dsame 5damount 5dof 5denergy
as 5dis 5dcreated 5din 5dthe 5dpresence 5dof 5doxygen?
5d
1. Dissipation 5dof 5dpyruvic 5dacid
2. Initiation 5dof 5dthe 5dcitric 5dacid 5dcycle
3. Activation 5dof 5dacetyl-coenzyme 5dA
4. Creation 5dof 5dacidosis 5dvia 5dlactic 5dacid
5 d 3. 5 d How 5dmany 5dadenosine 5dtriphosphates 5d(ATPs) 5dare 5dproduced 5din 5daerobic 5denergy 5dmetabolism?
1. 5 d 5 d 2
2. 5 d 5 d 3
3. 5d 5d 34
4. 5d 5d 53
4. Which 5dcell 5dorganelles 5ddiffer 5din 5dtheir 5dnumber 5daccording 5dto 5dthe 5dcell’s
5d energy 5dneeds?
1. Ribosomes
2. Mitochondria
3. Ribonucleic 5dacids
4. Deoxyribonucleic 5dacids
, DAVIS 5dADVANTAGE 5dFOR 5dPATHOPHYSIOLOGY3rd EDITION 5dCAPRIOTTI 5dTEST
5dBANK
5. Which 5doption 5dbest 5dsupports 5dthe 5dreason 5dmore 5denergy 5dis 5dproduced 5dwhen 5da 5dperson 5dis
exercising?
5d
1.Exercise 5dcauses 5dan 5dincrease 5din 5dthe 5dsynthesis 5dof 5dprotein.
2.There 5dis 5dan 5dincrease 5din 5dthe 5dproduction 5dof 5dpyruvic 5dacid 5din 5dthe 5dcells.
3.The 5dconversion 5dof 5dpyruvic 5dacid 5dto 5dlactic 5dacid 5dis 5dincreased 5dby 5dexercise.
4.Muscle 5dcells 5dhave 5dmore 5dmitochondria 5dto 5dmeet 5denergy 5ddemands.
6. When 5ddoes 5dribosomal 5dprotein 5dsynthesis 5dcease?
1. During 5dendoplasmic 5dreticulum 5dstress
2. During 5dthe 5dsynthesis 5dof 5dadenosine 5dtriphosphate 5d(ATP)
3. During 5da 5dsevere 5dhypoxic 5dstate
4. During 5dthe 5dprocessing 5dof 5d prohormone
7. Which 5dcellular 5dorganelles 5dare 5dresponsible 5dfor 5dpropelling 5dmucus 5dand 5dinhaled 5ddebris 5dout 5dof
the 5dlungs?
5d
1. Cilia
2. Microfilaments
3. Secretory 5dvesicles
, DAVIS 5dADVANTAGE 5dFOR 5dPATHOPHYSIOLOGY3rd EDITION 5dCAPRIOTTI 5dTEST
5dBANK
4. 5 d Endoplasmic 5dreticula
5d 8. 5 d Which 5dare 5dthe 5dkey 5dproteins 5din 5dthe 5dcontractile 5dunits 5dof 5dthe 5dmuscle 5dcells?
1. Actin 5dand 5dmyosin
2. Prohormone 5dand 5dtubulin
3. Tubulin 5dand 5dactin
4. Myosin 5dand 5dprohormone
5d 9. 5 d Which 5ddeficiency 5dcauses 5dTay–Sachs 5ddisease?
1. Proteasome
2. Peroxisome
3. Macrophage
4. Lysosomal 5denzymes
10. 5dWhich 5dis 5da 5dcharacteristic 5dof 5dadrenoleukodystrophy?
1. Accumulation 5dof 5dganglioside
2. Cessation 5dof 5dribosomal 5dprotein 5dsynthesis
3. Acceleration 5dof 5dcellular 5dproteasome 5dactivity
4. Accumulation 5dof 5dlong-chain 5dfatty 5dacids 5din 5dthe 5dnervous 5dsystem
11. 5dWhich 5dstatement 5dregarding 5dendoplasmic 5dreticulum 5d(ER) 5dstress 5dis 5dcorrect?
1. During 5dER 5dstress, 5dproteins 5dare 5drapidly 5ddegraded.
2. During 5dER 5dstress, 5dlipids 5dcannot 5dtravel 5dto 5dtheir 5dproper 5dintracellular 5dlocations.
3. During 5dER 5dstress, 5dlong-chain 5dfatty 5dacids 5daccumulate 5din 5dthe 5dnervous 5dsystem.
4. During5dER 5dstress, 5dnondeNgU
raRdeSdIsN
ubGsT
taB
nc.eC
s 5dO
acMcumulate 5 d in 5dthe5dcells.
12. 5dA 5dclient 5dis 5ddiagnosed 5dwith 5dtype 5d1 5ddiabetes 5dmellitus. 5dAt 5da 5dcellular 5dlevel, 5dwhich 5dfunction
5d is 5dlikely 5dto 5dbe 5dinvolved?
1. Inability5dof 5dribosomes 5dto 5dproduce 5da 5dspecific 5dtype 5dof 5dprotein
2. Incorrect 5dprocessing 5dof 5da 5dprotein 5dby5dthe 5dGolgi 5dapparatus
3. Stagnation 5dof 5da 5dpreviously 5ddynamic 5daction 5din 5dmicrotubules
4. Obstruction 5dof 5dthe 5dsmooth 5dendoplasmic 5dreticulum
13. 5dA 5dnewborn 5dpatient 5dexhibits 5dcharacteristics 5dof 5dsevere 5dphysical 5ddeformities. 5dWhich 5dcellular
component 5dis 5dexamined 5dto 5ddetermine 5dthe 5dcause 5dand 5dprobability 5dof 5dthe 5ddisease 5dbeing
5d
genetically 5dtransferred?
5d
1. Transfer 5dRNA
2. Ribosomal 5dRNA
3. Double 5dhelix 5dof 5dDNA
4. Mitochondrial 5dDNA
14. 5dA 5dhiker 5dexperiences 5dmuscle 5dpain 5dand 5dacidosis 5dwhile 5dascending 5da 5dmountain 5dduring 5da
long, 5dsteep 5dclimb. 5dWhich 5dis 5dthe 5dreason 5dfor 5dthese 5dmanifestations?
5d
1. Cellular 5dhypoxia
2. Autolysis
3. Heterolysis
4. Cellular 5dedema
15. 5dWhich 5dfactor 5dprovides 5dDNA 5dthe 5dunique 5dmolecular 5dability 5dto 5dreplicate?
1. The 5dpairing 5dof 5dnitrogenous 5dbases
5dBANK
, DAVIS 5dADVANTAGE 5dFOR 5dPATHOPHYSIOLOGY3rd EDITION 5dCAPRIOTTI 5dTEST
5dBANK
Chapter 5d1, 5dThe 5dCell 5din 5dHealth 5dand 5dIllnes
TEST 5dBANK 5dFOR 5dDAVIS 5dADVANTAGE 5dFOR 5dPATHOPHYSIOLOGY 5d3r 5ddEDITION 5dBY
5dCAPRIOTTI
Multiple 5dChoice
Identify 5dthe 5dchoice 5dthat 5dbest 5dcompletes 5dthe 5dstatement 5dor 5danswers 5dthe 5dquestion.
5 d 1. 5 d Which 5dstatement 5dregarding 5dthe 5dsodium–potassium 5dpump 5dis 5dcorrect?
1. The 5dcell’s 5dplasma 5dmembrane 5dis 5dmore 5dsoluble 5dto 5dsodium 5dions 5dthan 5dpotassium 5dions.
2. The 5dconcentration 5dof 5dsodium 5dions 5dshould 5dbe 5dhigher 5dinside 5dthe 5dcell 5dcompartment.
3. The 5dconcentration 5dof 5dpotassium 5dions 5dshould 5dbe 5dhigher 5doutside
the 5dcell 5dcompartment.
5d
4. The 5dactive 5dtransport 5dinvolves 5dpumping 5dout 5dthree 5dsodium 5dions 5dand
pumping 5din 5dtwo 5dpotassium 5dions.
5d
5 d 2. 5dIn 5dthe 5dabsence 5dof 5doxygen, 5dwhich 5dcellular 5dfunction 5dcreates 5dthe 5dsame 5damount 5dof 5denergy
as 5dis 5dcreated 5din 5dthe 5dpresence 5dof 5doxygen?
5d
1. Dissipation 5dof 5dpyruvic 5dacid
2. Initiation 5dof 5dthe 5dcitric 5dacid 5dcycle
3. Activation 5dof 5dacetyl-coenzyme 5dA
4. Creation 5dof 5dacidosis 5dvia 5dlactic 5dacid
5 d 3. 5 d How 5dmany 5dadenosine 5dtriphosphates 5d(ATPs) 5dare 5dproduced 5din 5daerobic 5denergy 5dmetabolism?
1. 5 d 5 d 2
2. 5 d 5 d 3
3. 5d 5d 34
4. 5d 5d 53
4. Which 5dcell 5dorganelles 5ddiffer 5din 5dtheir 5dnumber 5daccording 5dto 5dthe 5dcell’s
5d energy 5dneeds?
1. Ribosomes
2. Mitochondria
3. Ribonucleic 5dacids
4. Deoxyribonucleic 5dacids
, DAVIS 5dADVANTAGE 5dFOR 5dPATHOPHYSIOLOGY3rd EDITION 5dCAPRIOTTI 5dTEST
5dBANK
5. Which 5doption 5dbest 5dsupports 5dthe 5dreason 5dmore 5denergy 5dis 5dproduced 5dwhen 5da 5dperson 5dis
exercising?
5d
1.Exercise 5dcauses 5dan 5dincrease 5din 5dthe 5dsynthesis 5dof 5dprotein.
2.There 5dis 5dan 5dincrease 5din 5dthe 5dproduction 5dof 5dpyruvic 5dacid 5din 5dthe 5dcells.
3.The 5dconversion 5dof 5dpyruvic 5dacid 5dto 5dlactic 5dacid 5dis 5dincreased 5dby 5dexercise.
4.Muscle 5dcells 5dhave 5dmore 5dmitochondria 5dto 5dmeet 5denergy 5ddemands.
6. When 5ddoes 5dribosomal 5dprotein 5dsynthesis 5dcease?
1. During 5dendoplasmic 5dreticulum 5dstress
2. During 5dthe 5dsynthesis 5dof 5dadenosine 5dtriphosphate 5d(ATP)
3. During 5da 5dsevere 5dhypoxic 5dstate
4. During 5dthe 5dprocessing 5dof 5d prohormone
7. Which 5dcellular 5dorganelles 5dare 5dresponsible 5dfor 5dpropelling 5dmucus 5dand 5dinhaled 5ddebris 5dout 5dof
the 5dlungs?
5d
1. Cilia
2. Microfilaments
3. Secretory 5dvesicles
, DAVIS 5dADVANTAGE 5dFOR 5dPATHOPHYSIOLOGY3rd EDITION 5dCAPRIOTTI 5dTEST
5dBANK
4. 5 d Endoplasmic 5dreticula
5d 8. 5 d Which 5dare 5dthe 5dkey 5dproteins 5din 5dthe 5dcontractile 5dunits 5dof 5dthe 5dmuscle 5dcells?
1. Actin 5dand 5dmyosin
2. Prohormone 5dand 5dtubulin
3. Tubulin 5dand 5dactin
4. Myosin 5dand 5dprohormone
5d 9. 5 d Which 5ddeficiency 5dcauses 5dTay–Sachs 5ddisease?
1. Proteasome
2. Peroxisome
3. Macrophage
4. Lysosomal 5denzymes
10. 5dWhich 5dis 5da 5dcharacteristic 5dof 5dadrenoleukodystrophy?
1. Accumulation 5dof 5dganglioside
2. Cessation 5dof 5dribosomal 5dprotein 5dsynthesis
3. Acceleration 5dof 5dcellular 5dproteasome 5dactivity
4. Accumulation 5dof 5dlong-chain 5dfatty 5dacids 5din 5dthe 5dnervous 5dsystem
11. 5dWhich 5dstatement 5dregarding 5dendoplasmic 5dreticulum 5d(ER) 5dstress 5dis 5dcorrect?
1. During 5dER 5dstress, 5dproteins 5dare 5drapidly 5ddegraded.
2. During 5dER 5dstress, 5dlipids 5dcannot 5dtravel 5dto 5dtheir 5dproper 5dintracellular 5dlocations.
3. During 5dER 5dstress, 5dlong-chain 5dfatty 5dacids 5daccumulate 5din 5dthe 5dnervous 5dsystem.
4. During5dER 5dstress, 5dnondeNgU
raRdeSdIsN
ubGsT
taB
nc.eC
s 5dO
acMcumulate 5 d in 5dthe5dcells.
12. 5dA 5dclient 5dis 5ddiagnosed 5dwith 5dtype 5d1 5ddiabetes 5dmellitus. 5dAt 5da 5dcellular 5dlevel, 5dwhich 5dfunction
5d is 5dlikely 5dto 5dbe 5dinvolved?
1. Inability5dof 5dribosomes 5dto 5dproduce 5da 5dspecific 5dtype 5dof 5dprotein
2. Incorrect 5dprocessing 5dof 5da 5dprotein 5dby5dthe 5dGolgi 5dapparatus
3. Stagnation 5dof 5da 5dpreviously 5ddynamic 5daction 5din 5dmicrotubules
4. Obstruction 5dof 5dthe 5dsmooth 5dendoplasmic 5dreticulum
13. 5dA 5dnewborn 5dpatient 5dexhibits 5dcharacteristics 5dof 5dsevere 5dphysical 5ddeformities. 5dWhich 5dcellular
component 5dis 5dexamined 5dto 5ddetermine 5dthe 5dcause 5dand 5dprobability 5dof 5dthe 5ddisease 5dbeing
5d
genetically 5dtransferred?
5d
1. Transfer 5dRNA
2. Ribosomal 5dRNA
3. Double 5dhelix 5dof 5dDNA
4. Mitochondrial 5dDNA
14. 5dA 5dhiker 5dexperiences 5dmuscle 5dpain 5dand 5dacidosis 5dwhile 5dascending 5da 5dmountain 5dduring 5da
long, 5dsteep 5dclimb. 5dWhich 5dis 5dthe 5dreason 5dfor 5dthese 5dmanifestations?
5d
1. Cellular 5dhypoxia
2. Autolysis
3. Heterolysis
4. Cellular 5dedema
15. 5dWhich 5dfactor 5dprovides 5dDNA 5dthe 5dunique 5dmolecular 5dability 5dto 5dreplicate?
1. The 5dpairing 5dof 5dnitrogenous 5dbases
