NUR2407 CERTIFICATION EVALUATION
TEST PAPER 2026 COMPLETE STUDY
QUESTIONS WITH CORRECT ANSWERS
◉ Prerenal AKI cause. Answer: factors that reduce systemic
circulation causing reduction in renal blood flow which leads to
oliguria
- severe dehydration, HF, decreased CO
◉ intrarenal AKI cause. Answer: problems that cause direct damage
to kidney tissue
- prolonged ischemia, nephrotoxins, hemoglobin released from
hemolyzed rbc, myoglobin released from necrotic muscle cells,
kidney diseases
◉ acute tubular nectosis. Answer: results from ischemia,
nephrotoxins or sepsis
severe ischemia causes disruption in basement membrane and
patchy destruction of tubular epithelium
nephrotoxic agents cause necrosis of tubular epitherlial cells -
cloged tubules
, potentially reversible
◉ postrenal AKI cause. Answer: mechanical obstruction of outflow
which results into renal pelvis, impairing renal function
- benign prostatic hyperplasia, prostate cancer, calculi, trauma,
extrarenal tumors
◉ what are the three phases of aki. Answer: Oliguric, diuretic,
recovery
◉ AKI diagnostic studies contraindications. Answer: contrast
medium is a contraindication in aki
◉ AKI interprofessional care. Answer: Ensure adequate
intravascular volume and cardiac output
- Loop diuretics (e.g., furosemide [Lasix])
- Osmotic diuretics (e.g., mannitol)
Closely monitor fluid intake during oliguric phase
Fluid restriction calculation: All fluid losses for previous 24 hours +
600 mL
◉ gerontologic considerations for AKI. Answer: decreased GFR with
aging
More susceptible to AKI
TEST PAPER 2026 COMPLETE STUDY
QUESTIONS WITH CORRECT ANSWERS
◉ Prerenal AKI cause. Answer: factors that reduce systemic
circulation causing reduction in renal blood flow which leads to
oliguria
- severe dehydration, HF, decreased CO
◉ intrarenal AKI cause. Answer: problems that cause direct damage
to kidney tissue
- prolonged ischemia, nephrotoxins, hemoglobin released from
hemolyzed rbc, myoglobin released from necrotic muscle cells,
kidney diseases
◉ acute tubular nectosis. Answer: results from ischemia,
nephrotoxins or sepsis
severe ischemia causes disruption in basement membrane and
patchy destruction of tubular epithelium
nephrotoxic agents cause necrosis of tubular epitherlial cells -
cloged tubules
, potentially reversible
◉ postrenal AKI cause. Answer: mechanical obstruction of outflow
which results into renal pelvis, impairing renal function
- benign prostatic hyperplasia, prostate cancer, calculi, trauma,
extrarenal tumors
◉ what are the three phases of aki. Answer: Oliguric, diuretic,
recovery
◉ AKI diagnostic studies contraindications. Answer: contrast
medium is a contraindication in aki
◉ AKI interprofessional care. Answer: Ensure adequate
intravascular volume and cardiac output
- Loop diuretics (e.g., furosemide [Lasix])
- Osmotic diuretics (e.g., mannitol)
Closely monitor fluid intake during oliguric phase
Fluid restriction calculation: All fluid losses for previous 24 hours +
600 mL
◉ gerontologic considerations for AKI. Answer: decreased GFR with
aging
More susceptible to AKI
