NUR 631 Advanced Physiology and
Pathophysiology Midterm
NUR 631 Advanced Physiology and
Pathophysiology Midterm and Final
Exam Prep Test Bank 1 with 350 Exam
Questions and Correct Answers/ GCU
NUR 631 Latest Exam Prep Test Bank
(New!)
Antidiuretic hormone acts to cause vasoconstriction when?
A+ TEST BANK 1
, NUR 631 Advanced Physiology and
Pathophysiology Midterm
a. Urine output is less than 20ml/hr.
b. Serum osmolality is increased.
c. Osmotic and oncotic pressures are increased.
d. Vasopressin is pharmacologically administered. –
Correct Answer :d. Vasopressin in pharmacologically administered.
Exp: ADH was originally named vasopressin
because, in extremely high doses, it causes vasoconstriction and a resulting increase in
arterial blood pressure. However, significant vasoconstriction may only be achieved
pharmacologically. Antidiuretic hormone induced vasoconstriction is not a result of the other
options. page 698-699
Where is antidiuretic hormone (ADH) synthesized, and where does it act?
a. Hypothalamus; renal tubular cells
b. Renal tubules; renal collecting ducts
c. Anterior pituitary; posterior pituitary
d. Posterior pituitary; loop of Henle –
Correct Answer :b. Renal tubules ; Renal collecting ducts
Exp: Once synthesized in the hypothalamus, ADH acts on the vasopressin 2 (V2) receptors of
the renal duct cells to increase their permeability. This information helps eliminate the other
options. page 696-698
A+ TEST BANK 2
, NUR 631 Advanced Physiology and
Pathophysiology Midterm
Diabetes insipidus is a result of:
a. Antidiuretic hormone hyposecretion
c. Insulin hyposecretion
b. Antidiuretic hormone hypersecretion
d. Insulin hypersecretion –
Correct Answer :a. Antidiuretic hormone hyposecretion
Exp: Of the available options, diabetes insipidus is a result of insufficient antidiuretic hormone.
page 719
The cause of neurogenic diabetes insipidus (DI) is related to an organic lesion of the:
a. Anterior pituitary
b. Thalamus
c. Posterior pituitary
d. Renal tubules –
Correct Answer :c. Posterior pituitary
Exp: Neurogenic DI is a result of dysfunctional antidiuretic hormone synthesis, caused by a
lesion of the posterior pituitary, hypothalamus, or pituitary stalk. page 729
A+ TEST BANK 3
, NUR 631 Advanced Physiology and
Pathophysiology Midterm
Which form of diabetic insipidus (DI) will result if the target cells for antidiuretic hormone
(ADH) in the renal collecting tubules demonstrate insensitivity?
a. Neurogenic
b. Nephrogenic
c. Psychogenic
d. Ischemic –
Correct Answer :b. Nephrogenic
Exp: Only nephrogenic DI is associated with an insensitivity of the renal collecting tubules to
ADH. page 720
Which form of diabetes insipidus (DI) is treatable with exoxenous antidiuretic hormone
(ADH)?
a. Neurogenic
c. Nephrogenic
b. Psychogenic
d. Ischemic –
Correct Answer :a. Neurogenic
Exp: Neurogenic DI is treated with ADH replacement therapy. The other options are incorrect.
page 720
A+ TEST BANK 4
Pathophysiology Midterm
NUR 631 Advanced Physiology and
Pathophysiology Midterm and Final
Exam Prep Test Bank 1 with 350 Exam
Questions and Correct Answers/ GCU
NUR 631 Latest Exam Prep Test Bank
(New!)
Antidiuretic hormone acts to cause vasoconstriction when?
A+ TEST BANK 1
, NUR 631 Advanced Physiology and
Pathophysiology Midterm
a. Urine output is less than 20ml/hr.
b. Serum osmolality is increased.
c. Osmotic and oncotic pressures are increased.
d. Vasopressin is pharmacologically administered. –
Correct Answer :d. Vasopressin in pharmacologically administered.
Exp: ADH was originally named vasopressin
because, in extremely high doses, it causes vasoconstriction and a resulting increase in
arterial blood pressure. However, significant vasoconstriction may only be achieved
pharmacologically. Antidiuretic hormone induced vasoconstriction is not a result of the other
options. page 698-699
Where is antidiuretic hormone (ADH) synthesized, and where does it act?
a. Hypothalamus; renal tubular cells
b. Renal tubules; renal collecting ducts
c. Anterior pituitary; posterior pituitary
d. Posterior pituitary; loop of Henle –
Correct Answer :b. Renal tubules ; Renal collecting ducts
Exp: Once synthesized in the hypothalamus, ADH acts on the vasopressin 2 (V2) receptors of
the renal duct cells to increase their permeability. This information helps eliminate the other
options. page 696-698
A+ TEST BANK 2
, NUR 631 Advanced Physiology and
Pathophysiology Midterm
Diabetes insipidus is a result of:
a. Antidiuretic hormone hyposecretion
c. Insulin hyposecretion
b. Antidiuretic hormone hypersecretion
d. Insulin hypersecretion –
Correct Answer :a. Antidiuretic hormone hyposecretion
Exp: Of the available options, diabetes insipidus is a result of insufficient antidiuretic hormone.
page 719
The cause of neurogenic diabetes insipidus (DI) is related to an organic lesion of the:
a. Anterior pituitary
b. Thalamus
c. Posterior pituitary
d. Renal tubules –
Correct Answer :c. Posterior pituitary
Exp: Neurogenic DI is a result of dysfunctional antidiuretic hormone synthesis, caused by a
lesion of the posterior pituitary, hypothalamus, or pituitary stalk. page 729
A+ TEST BANK 3
, NUR 631 Advanced Physiology and
Pathophysiology Midterm
Which form of diabetic insipidus (DI) will result if the target cells for antidiuretic hormone
(ADH) in the renal collecting tubules demonstrate insensitivity?
a. Neurogenic
b. Nephrogenic
c. Psychogenic
d. Ischemic –
Correct Answer :b. Nephrogenic
Exp: Only nephrogenic DI is associated with an insensitivity of the renal collecting tubules to
ADH. page 720
Which form of diabetes insipidus (DI) is treatable with exoxenous antidiuretic hormone
(ADH)?
a. Neurogenic
c. Nephrogenic
b. Psychogenic
d. Ischemic –
Correct Answer :a. Neurogenic
Exp: Neurogenic DI is treated with ADH replacement therapy. The other options are incorrect.
page 720
A+ TEST BANK 4
