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Pathophysiology Final Rasmussen University Winter 2026
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1. •Neither recessive nor dominant—co-dominant.
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• Hemoglobin S causes erythrocytes to be abnormally shaped.
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• Abnormal erythrocytes carry less oxygen and clog vessels, causing hypoxia and tissue ischemia.
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k Exercise-induced asthma k
k Chronic Bronchitis k
k k Sickle Cell Anemia k k
k Occupational asthma k
2. blood flow reestablished to quickly
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k Distributive shock k
k Anaphylactic shock k
k k Reprofusion injury k
k spinal cord injury k k
3. •Life-threatening complication of many conditions k k k k
• Results from an inappropriate immune response
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• Widespread coagulation followed by massive bleeding because of the depletion of clotting factors
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• Complications: shock and multisystem organ failure k k k k k
k k Disseminated intravascular coagulation - DIC k k k k
k k Chronic Bronchitis Manifestations and Treatment
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k k Disseminated Intravascular Coagulation DIC k k k
k Traumatic Brain Injury Complications k k k
4. thyroidgland -T3,T4,calcitonin
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parathyroid gland - parathyroid hormone
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k k during acute respiratory failure k k k
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, k stress incontinence k
k secondary brain injury k k
k k Glands that regulate calcium k k k
5. altered level of consciousnessk k k
k BPdrops
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HRgoesup k k
k hypotensive
k k Glands that regulate calcium k k k
k spinal cord injury k k
k k shock sighs and symptoms k k k
k Reprofusion injury k
6. chest pain that occurs while a person is at rest and not exerting himself does
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k not get betterk k
k hemorrhagic stroke k
k Bone electrolytes k
k sickle cell crisis k k
k k unstable angina k
7. Abnormallylowwhite blood cellcount k k k k k
• Normal range = 5,000 to 10,000 cells/mL3 blood
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k beta cells k
k k Leukopenia
k Pneumonia
k CT scan k
8. damage to the alveoli k k k
air can get in, but not out
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k k emphysema lecture k
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, k Normal CO2 levels k k
k anemia symptoms k
k Stabilize the patient k k
9. Manifestations
• May be vague and develop slowly, or may be sudden and severe.
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• Symptoms may improve and then suddenly worsen. k k k k k k
• The outward appearance of the head is not an indication of the injury severity.
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• Not being able to recall event details
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• Indications of a concussion k k k
• Changes in or unequal pupil size k k k k k
• Seizures
• Asymmetricalfacial features k k
• Fluid draining from the nose, mouth, or ears
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• Fracture of skullor face k k k k
• Bruising of face k k
• Swelling at site of injury k k k k
• Scalp wound k
• Impaired hearing, smell, taste, speech, or vision
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• Inability to move one or more limbs k k k k k k
• Irritability (especially in children) k k k
• Personality changes k
• Unusual behavior k
• Loss of consciousness
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• Bradypnea
• Hypotension
• Restlessness
• Lack of coordination
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• Lethargy
• Stiff neckk
• Vomiting
k k Acute Respiratory Distress Syndrome (ARDS) Manifestations and Treatment
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k Acute Bronchitis Manifestations k k
k k Tuberculosis Manifestations and Treatment k k k
k k Traumatic Brain Injury Manifestations k k k
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, 10. not enough ADH
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k Alkalosis
k Stable Angina k
k spinal cord injury k k
k k diabetes insipidus k
11. -Homozygous.
-Most severe. k
-Almost all erythrocytes are sickled.
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k Cushing's syndrome k
k sickle cell crisis k k
k k Sickle cell disease k k
k Normal CO2 levels k k
12. •Frequently caused by aspirin—prevents the conversion of prostaglandins, which stimulate leukotriene release, a
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k powerfulbronchoconstrictor. k
• Can befatal. k k
• Reactions can be delayed up to 12 hours afterdrug ingestion.
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k Acute Bronchitis k
k k Drug-induced asthma k
k stress incontinence k
k Tuberculosis TB k
13. pulmonaryedema, k
wet coughing,
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k shortness of breath, k k
and dyspnea
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k k Acute lymphoblastic leukemiak k
k k Left sided heart failure symptoms
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k k Right sided heart failure symptoms
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k CerebralVascularAccident (CVA) k k k
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