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NUR 209 Exam 3 Medical Surgical Nursing II | LATEST 2026/2027 | Complete Questions with Verified Answers and Detailed Rationales | LPN Medical-Surgical Exam

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INSTANT PDF DOWNLOAD — This official test bank for NUR 209 Exam 3 Medical Surgical Nursing II covers the essential content for the third examination, focusing on critical medical-surgical nursing concepts across multiple body systems . This comprehensive resource includes verified questions and answers with detailed rationales in multiple-choice, select-all-that-apply (SATA), and clinical scenario formats aligned with LPN/PN standards . CARDIOVASCULAR DISORDERS Endocarditis – Inflammation of the inner layer of the heart (endocardium) and heart valves . Common causative organism: Streptococcus viridans (dental procedures) and Staphylococcus aureus (IV drug use) . Risk factors: prosthetic heart valves, IV drug use, structural heart defects, history of rheumatic fever , recent dental or surgical procedures . Assessment findings: fever, chills, malaise, new or changing heart murmur, Osler nodes (tender, pea-sized nodules on fingers/toes), Janeway lesions (non-tender macules on palms/soles), splinter hemorrhages (linear red-brown streaks under nails), Roth spots (retinal hemorrhages with white centers), petechiae, weight loss, night sweats . Diagnosis: blood cultures (3 sets from different sites), echocardiography (transthoracic TTE vs transesophageal TEE) . Treatment: IV antibiotics (penicillin G, ceftriaxone, vancomycin) for 4-6 weeks . Surgical valve replacement for severe regurgitation, heart failure, abscess, or failed antibiotic therapy . Nursing interventions: administer IV antibiotics on time (peak/trough levels for vancomycin), monitor for embolization (neurologic changes, extremity pain, abdominal pain, hematuria), teach prophylactic antibiotics before dental procedures (AHA guidelines for high-risk patients) . Heart Failure (Left-Sided vs Right-Sided) – Left-sided heart failure: blood backs up into pulmonary circulation . Manifestations: dyspnea (initially on exertion), orthopnea (difficulty breathing lying flat), paroxysmal nocturnal dyspnea (PND), crackles (rales) in lung bases, dry hacking cough, S3 gallop (early sign), fatigue, restlessness, confusion (cerebral hypoxia) . Right-sided heart failure: blood backs up into systemic circulation . Manifestations: peripheral edema (dependent edema – feet/ankles, sacrum in bedridden), jugular venous distention (JVD), hepatomegaly (enlarged liver), ascites (abdominal fluid), weight gain (fluid retention), anorexia, nausea, weakness . Nursing interventions: daily weight (same scale, same time, same clothing), strict I&O (intake and output), low sodium diet (2,000 mg/day), fluid restriction (1.5-2 L/day if ordered), elevate head of bed (semi-Fowler's to high-Fowler's), monitor lung sounds (crackles), administer medications (diuretics, ACE inhibitors, beta-blockers, digoxin), monitor for digoxin toxicity (nausea, vomiting, visual changes yellow-green halos, bradycardia, cardiac dysrhythmias – hold for HR 60) . Angina Pectoris – Chest pain due to myocardial ischemia without cell death (reversible) . Types: stable angina (predictable with exertion/stress, relieved by rest or nitroglycerin), unstable angina (increasing frequency, occurs at rest, unpredictable – medical emergency), variant angina (Prinzmetal's – due to coronary artery spasm, often at rest) . Precipitating factors: the 4 E's – Exertion (physical activity, exercise), Eating (large meal), Emotional distress (stress, anger, fear), Extreme temperatures (cold weather, hot weather) . Additional triggers: smoking, sexual activity, caffeine, cocaine . Assessment findings: crushing, squeezing, substernal chest pain radiating to left arm, jaw, neck, back, or epigastrium; associated symptoms: diaphoresis (sweating), pallor, dyspnea, nausea, vomiting, indigestion . Women and diabetics more likely to have atypical symptoms (fatigue, weakness, indigestion, back/jaw pain, no chest pain) . Treatment (MONA): Morphine (pain unrelieved by nitroglycerin), Oxygen (maintain SpO2 90%), Nitroglycerin (vasodilator – 0.4 mg SL q5 min x3 doses, contraindicated with phosphodiesterase inhibitors Viagra/Cialis/Levitra within 24-48 hours), Aspirin (324 mg chewed) . Nursing interventions: administer nitroglycerin, monitor BP (hypotension is side effect), 12-lead ECG (compare to previous, look for ST changes), cardiac enzymes (troponin, CK-MB) to rule out MI, educate about precipitating factors, teach PRN nitroglycerin use (take at first sign of chest pain, call 911 if no relief after 5 minutes and 2 additional doses) . Myocardial Infarction (MI) – Tissue death (necrosis) of heart muscle due to prolonged ischemia . Assessment findings (same as angina but more severe and prolonged 20 minutes, not relieved by rest or nitroglycerin): crushing substernal chest pain radiating to left arm/jaw/back, diaphoresis, pallor, dyspnea, nausea, vomiting, anxiety, feeling of impending doom, women/diabetics may have atypical symptoms (fatigue, weakness, indigestion, back/jaw pain) . Diagnosis: ECG (ST elevation STEMI vs ST depression or T wave inversion NSTEMI), cardiac enzymes (troponin – specific and sensitive, rises 2-4 hours, remains elevated 7-10 days; CK-MB rises 4-6 hours, returns to normal 48-72 hours), elevated WBC, elevated ESR . Treatment (STEMI): emergency revascularization – percutaneous coronary intervention (PCI) with stent within 90 minutes of arrival, or thrombolytic therapy (e.g., alteplase tPA) if PCI not available within 120 minutes . Medications: MONA (Morphine, Oxygen, Nitroglycerin, Aspirin), beta-blockers (decrease O2 demand, given within 24 hours, contraindicated in heart failure or bradycardia), ACE inhibitors (prevent remodeling), statins (aggressive lipid lowering) . Nursing interventions: immediate assessment and treatment, cardiac monitoring (dysrhythmias common – PVCs, VT, VF), frequent vital signs (q15 min initially), oxygen (maintain SpO2 90%), pain management (morphine), bedrest (first 12-24 hours), monitor for complications (heart failure, cardiogenic shock, dysrhythmias, pericarditis), patient education (lifestyle modifications, medications, cardiac

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Instelling
NUR 209/ NUR209
Vak
NUR 209/ NUR209

Voorbeeld van de inhoud

1|Page




NUR 209 Exam 3 Medical Surgical

Nursing II Test Bank with Verified

Answers and Detailed Rationales Grade A


1. What does the endocrine system do?

Correct Answer: Control hormones that affect: Growth, Metabolism,

Stress, Fluid balance

Rationale:

1. The endocrine system regulates body functions through hormone secretion.

2. Growth hormones control physical development and cell reproduction.

3. Metabolic hormones (thyroid) regulate energy production and utilization.

4. Stress hormones (cortisol, epinephrine) mediate the fight-or-flight

response.

5. Fluid balance hormones (ADH, aldosterone) control water and electrolyte

homeostasis.



2. How do endocrine glands release hormones?

Correct Answer: Directly into the bloodstream

,2|Page


Rationale:

1. Endocrine glands are ductless glands.

2. Hormones are secreted directly into the interstitial fluid and then into the

bloodstream.

3. Blood carries hormones to target organs throughout the body.

4. This differs from exocrine glands which secrete through ducts.



3. Why is the pancreas unique?

Correct Answer: It is BOTH endocrine and exocrine

Rationale:

1. The pancreas has both endocrine functions (hormone secretion) and exocrine

functions (digestive enzyme secretion).

2. Endocrine pancreas: islets of Langerhans secrete insulin and glucagon into

blood.

3. Exocrine pancreas: acinar cells secrete digestive enzymes into the pancreatic

duct.

4. No other organ has such dual function.



4. What does insulin do?

Correct Answer: Lowers blood sugar

,3|Page


Rationale:

1. Insulin is secreted by beta cells of the pancreas.

2. It facilitates glucose uptake into cells (muscle, liver, fat).

3. Insulin promotes glycogenesis (storage of glucose as glycogen).

4. It lowers blood glucose levels after meals.



5. What does glucagon do?

Correct Answer: Raises blood sugar

Rationale:

1. Glucagon is secreted by alpha cells of the pancreas.

2. It stimulates glycogenolysis (breakdown of glycogen to glucose).

3. It promotes gluconeogenesis (glucose production from amino acids).

4. Glucagon raises blood glucose levels during fasting or hypoglycemia.



6. What is the exocrine pancreas function?

Correct Answer: Digestion

Rationale:

1. Exocrine pancreas secretes digestive enzymes.

2. These enzymes break down carbohydrates, fats, and proteins.

, 4|Page


3. Enzymes are delivered to the duodenum via the pancreatic duct.

4. Exocrine dysfunction causes malabsorption and malnutrition.



7. Where do digestive enzymes go?

Correct Answer: Common bile duct → duodenum

Rationale:

1. Pancreatic enzymes travel through the pancreatic duct.

2. The pancreatic duct joins the common bile duct.

3. Together they empty into the duodenum (first part of the small intestine).

4. Bile from the liver and enzymes from the pancreas mix at this junction.



8. What does amylase break down?

Correct Answer: Carbohydrates

Rationale:

1. Amylase is a digestive enzyme produced by the pancreas and salivary glands.

2. It breaks down starches (complex carbohydrates) into simpler sugars.

3. Elevated serum amylase indicates pancreatitis.

4. Carbohydrate digestion begins in the mouth and continues in the small

intestine.

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Vak
NUR 209/ NUR209

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