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Rasmussen Pathophysiology Exam 2 200 Questions and Answers (2026) | Latest Test Bank with Verified Solutions | Grade A+

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Rasmussen Pathophysiology Exam 2 200 Questions and Answers (2026) | Latest Test Bank with Verified Solutions | Grade A+

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Rasmussen Pathophysiology Exam 2 200
Questions and Answers (2026) | Latest
Test Bank with Verified Solutions |
Grade A+
Inflammation of the stomach's mucosal lining (may involve the entire stomach or a
region) -✓✓Gastritis

_________Can be a mild, transient irritation, or it cab be a severe ulceration with
hemorrhage -✓✓Acute Gastritis

_________ Usually develops suddenly and is likely to be accompanied by nausea and
epigastric pain -✓✓Acute Gastritis

_________ Gastritis develops gradually. -✓✓Chronic Gastritis

Gastritis can be further categorized as erosive or nonerosive -✓✓Chronic Gastritis

Symptoms of: Anorexia, nausea & vomiting, postprandial discomfort, and hematemesis.
-✓✓Acute Gastritis

Symptoms of: May be asymptomatic, but usually accompanied by a dull epigastric pain
and a sensation of fullness after minimal intake. -✓✓Chronic Gastritis

Inflammation of the stomach and intestines, usually because of an infection or allergic
reaction -✓✓Gastroenteritis

Usually due to primary inflammatory disease such as crohns disease -✓✓Chronic
Gastroenteritis

Commonly due to direct infection such as salmonella from raw or undercooked chicken
or eggs -✓✓Acute Gastroenteritis

Signs & Symptoms: Diarrhea, abdominal discomfort, pain, nausea, and vomiting -
✓✓Gastroenteritis

Most common cause of chronic gastritis -✓✓Helicobacter pylori

Embeds itself in the mucous layer, activating toxins and enzymes that cause
inflammation. Genetic vulnerability and lifestyle behaviors (smoking and stress) may
increase the susceptibility -✓✓Helicobacter pylori

,Other causes of?: Organisms transmitted though food and water contamination, long-
term use of nonsteroidal anti-inflammatory drugs, excessive alcohol use, severe stress,
autoimmune conditions, and other chronic disease -✓✓Gastritis

Complications of?: Peptic ulcers, gastric cancer, and hemorrhage -✓✓Chronic Gastritis

Manifestations of?: Include indigestion, heartburn, epigastric pain, abdominal cramping,
nausea, vomiting, anorexia, fever, and malaise. Hematemesis and dark, tarry stools can
indicate ulceration and bleeding. -✓✓Gastritis

Chyme periodically backs up from the stomach into the esophagus. Bile can also back
up into the esophagus. -✓✓GERD (Gastroesophageal Reflux Disease)

These gastric secretions irritate the esophageal mucosa -✓✓GERD (Gastroesophageal
Reflux Disease)

Causes of?: certain foods (e.g., chocolate, caffeine, carbonated beverages, citrus fruit,
tomatoes, spicy or fatty foods, and peppermint), alcohol consumption, nicotine, hiatal
hernia, obesity, pregnancy, certain medications (e.g., corticosteroids, beta blockers,
calcium-channel blockers, and anticholinergics), nasogastric intubation, and delayed
gastric emptying -✓✓GERD (Gastroesophageal Reflux Disease)

Manifestations of?: heartburn, epigastric pain (usually after a meal or when
recombinant), dysphagia, dry cough, laryngitis, pharyngitis, regurgitation of food, and
sensation of a lump in the throat. -✓✓GERD (Gastroesophageal Reflux Disease)

Complications of?: esophagitis, strictures, ulcerations, esophageal cancer, and chronic
pulmonary disease -✓✓GERD (Gastroesophageal Reflux Disease)

Often confused with angina and may warrant ruling out cardiac disease -✓✓GERD
(Gastroesophageal Reflux Disease)

Lesions affecting the lining of the stomach or duodenum -✓✓Peptic Ulcer Disease
(PUD)

Risk factors of?: being male, advancing age, nonsteroidal anti-inflammatory drug use
(NSAIDs), H. pylori infections, certain gastric tumors, and those for GERD. -✓✓Peptic
Ulcer Disease (PUD)

Vary in severity from superficial erosions to complete penetration through the GI tract
wall. Develops because of an imbalance between destructive forces and protective
mechanisms -✓✓Peptic Ulcer Disease (PUD)

, Types of Peptic Ulcer Disease (PUD) -✓✓Duodenal Ulcers
Gastric Ulcers
Stress Ulcers

Most commonly associated with excessive acid or H. pylori infections. Typically present
with epigastric pain that is relieved in the presence of food -✓✓Duodenal ulcers

Less frequent but more deadly. Typically associated with malignancy and nonsteroidal
anti-inflammatory drugs. Pain typically worsens with eating. -✓✓Gastric Ulcers

Develop because of a major physiological stressor on the body due to local tissue
ischemia, tissue acidosis, bile salts entering the stomach, and decreased GI motility. -
✓✓Stress ulcers

Stress ulcers associated with burns -✓✓Curling's ulcers

Stress ulcers associated with head injuries -✓✓Cushing's ulcers

Most frequently develop in the stomach; multiple ulcers can form within hours of the
precipitating event. -✓✓Stress ulcers

Often hemorrhage is the first indicator because the ulcer develops rapidly and tends to
be masked by the primary problem -✓✓Stress ulcer

Complications of?: GI hemorrhage, obstruction, perforation, and peritonitis -✓✓Peptic
Ulcer Disease (PUD)

Manifestations of?: epigastric or abdominal pain, abdominal cramping, heartburn,
indigestion, nausea, and vomiting -✓✓Peptic Ulcer Disease (PUD)

Acute inflammation and necrosis of large intestine; it affects the mucosa and sometimes
other layers -✓✓Pseudomembranous Colitis (C. Diff)

Causes of?: Exposure to antibiotics, patients with cancer, or post abdominal surgery
susceptible, mediated by bacterial toxins -✓✓Pseudomembranous Colitis (C. Diff)

Manifestations of?: Diarrhea (often bloody), abdominal pain, fever, and leukocytosis -
✓✓Pseudomembranous Colitis (C. Diff)

Inflammation of the vermiform appendix. Most often caused by an infection. Triggers
local tissue edema, which obstructs the small structure. As fluid builds inside the
appendix, microorganisms proliferate -✓✓Appendicitis

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