SEVI Test #2 Welsh Actual Exam 2026/2027 –
Complete Exam-Style Questions with Detailed
Rationales | 100% Verified | Pass
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Q1. A patient presents with muscle weakness, fatigue, and an
ECG showing a "tented" T wave. Which electrolyte imbalance is
most likely?
A) Hypokalemia
B) Hyperkalemia
C) Hypocalcemia
D) Hypermagnesemia
✅ Answer: B
Rationale: Hyperkalemia (elevated serum potassium) causes
characteristic ECG changes including peaked or "tented" T
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waves, as well as muscle weakness and fatigue. Hypokalemia
causes flat T waves and U waves. Hypocalcemia prolongs the QT
interval. Hypermagnesemia causes bradycardia and heart block.
Q2. Which IV fluid is most appropriate for a patient with
Diabetic Ketoacidosis (DKA) requiring volume resuscitation?
A) Dextrose 5% in water (D5W)
B) Half-normal saline (0.45% NaCl)
C) Normal saline (0.9% NaCl)
D) Lactated Ringer's solution
✅ Answer: C
Rationale: Normal saline (0.9% NaCl) is the initial fluid of choice
for volume resuscitation in DKA because it is isotonic and
expands intravascular volume without causing rapid shifts in
osmolality. Half-normal saline is used later once glucose begins
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to drop. D5W is added when blood glucose reaches
approximately 250 mg/dL to prevent hypoglycemia while
continuing insulin. Lactated Ringer's is generally avoided due to
the lactate load in an already acidotic patient.
Q3. A patient with DKA is receiving an insulin drip. At what
blood glucose level should D5W be added to the IV fluids?
A) 150 mg/dL
B) 200 mg/dL
C) 250 mg/dL
D) 350 mg/dL
✅ Answer: C
Rationale: The standard of care for DKA management is to add
dextrose-containing fluids (such as D5W or D5 1/2 normal
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saline) when the blood glucose level falls to approximately
250 mg/dL. This prevents hypoglycemia while allowing continued
insulin administration to correct the acidosis and suppress ketone
production.
Q4. The most sensitive indicator of early diabetic nephropathy is:
A) Elevated serum creatinine
B) Elevated BUN
C) Microalbuminuria
D) Proteinuria
✅ Answer: C
Rationale: Microalbuminuria (30–300 mg/24 h) is the earliest
detectable sign of diabetic nephropathy. It is a marker of
glomerular damage that occurs before overt proteinuria or