Robbins
Chapters
Basic1–3
Pathology
Study
Robbins
Chapters
Guide
Basic
Cell
1–3
Pathology
Injury
Study
Inflammation
Chapters
Guide Cell
1–3
andInjury
Hemodynamic
Study
Inflammation
Guide Cell
Disorders.pdf
andInjury
Hemodynamic
Inflammation
Disorders.pdf
and Hemodynamic Disorders.pdf
Robbins Basic
Pathology
Chapters 1–3
Study Guide Cell
Injury Inflammation
and Hemodynamic
Disorders
Robbins Basic Pathology
Robbins
Chapters
Basic1–3
Pathology
Study
Robbins
Chapters
Guide
Basic
Cell
1–3
Pathology
Injury
Study
Inflammation
Chapters
Guide Cell
1–3
andInjury
Hemodynamic
Study
Inflammation
Guide Cell
Disorders.pdf
andInjury
Hemodynamic
Inflammation
Disorders.pdf
and Hemodynamic Disorders.pdf
,Robbins Basic Pathology chap 1,2,3.pdf Robbins Basic Pathology chap 1,2,3.pdf Robbins Basic Pathology chap 1,2,3.pdf
Terms in this set (113)
Stages in cellular response to stress and injurious
stimulus
The relationship among normal, adapted, reversibly
injured and dead myocardial cells
Hypertrophy Increase in cell size often in response to increase in workload induced by
growth factors in response to mechanical stress or other stimuli; occurs in
tissues incapable of cell division
Physiologic hypertrophy of uterus during pregnancy
Robbins Basic Pathology chap 1,2,3.pdf Robbins Basic Pathology chap 1,2,3.pdf Robbins Basic Pathology chap 1,2,3.pdf
,Robbins Basic Pathology chap 1,2,3.pdf Robbins Basic Pathology chap 1,2,3.pdf Robbins Basic Pathology chap 1,2,3.pdf
Hyperplasia Increase in cell number in response to hormones and other growth factors;
occurs in tissues that are capable of dividing or that contain abundant stem
cells
Atrophy decrease in cell and organ size as a result of decreased nutrient supply or
disuse; associated with decreased synthesis of cellular building blocks and
increased breakdown of cellular organelles
Atrophy of atherosclerotic brain
Metaplasia Change in phenotype of differentiated cells; often in response to chronic
irritation that makes cells better able to withstand the stress; usually
induced by altered differentiation pathway of tissue stem cells; may result in
reduced functions or increased propensity for malignant transformation
Metaplasia of normal columnar epithelium to
squamous epithelium in bronchus
Robbins Basic Pathology chap 1,2,3.pdf Robbins Basic Pathology chap 1,2,3.pdf Robbins Basic Pathology chap 1,2,3.pdf
, Robbins Basic Pathology chap 1,2,3.pdf Robbins Basic Pathology chap 1,2,3.pdf Robbins Basic Pathology chap 1,2,3.pdf
Cellular adaptations to stress Hypertrophy: increased cell and organ size, often in response to increased
workload; induced by growth factors produced in response to mechanical
stress or other stimuli; occurs in tissues incapable of cell division
Hyperplasia: increased cell numbers in response to hormones and other
growth factors; occurs in tissues whose cells are able to divide or contain
abundant tissue stem cells
Atrophy: decreased cell and organ size, as a result of decreased nutrient
supply or disuse; associated with decreased synthesis of cellular building
blocks and increased breakdown of cellular organelles
Metaplasia: change in phenotype of differentiated cells, often in response
to chronic irritation, that makes cells better able to withstand the stress;
usually induced by altered differentiation pathway of tissue stem cells; may
result in reduced functions or increased propensity for malignant
transformation
Reversible cell injury cell swelling, fatty change, plasma membrane blebbing, loss of microvilli,
mitochondrial swelling, dilation of the ER, eosinophilia
Robbins Basic Pathology chap 1,2,3.pdf Robbins Basic Pathology chap 1,2,3.pdf Robbins Basic Pathology chap 1,2,3.pdf