NSG 533 Exam 3 (NEW 2025/ 2026 Update) Advanced
Pharmacology Guide| Questions & Answers| Grade A| 100%
Correct (Verified Solutions)- Wilkes
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NSG 533 Opioid Quiz NSG 533 Exam 3 NSG 533 Exam 3 Op
24 terms 43 terms 45 terms 10
blazierose Preview ksanner13 Preview the_maincharacter Preview
Practice questions for this set
Learn 1 /7 Study with Learn
obesity, diabetes, increased age, black males, family hx
Choose an answer
1 risk factors for HTN 2 risk factors for Pad
risk factors/causes of coronary artery
3 cad risk factors 4
disease
Don't know?
Terms in this set (144)
Universal stages of Heart Failure (HF) - At-risk for HF
- Pre HF
- HF
- Advanced HF
, according to the universal definition, what is "at-risk for - do not have HF, but at risk
HF"? - no current or prior s/s
- no structural, functional, or biomarker evidence supporting HF
according to the universal definition, what is "HF" - clinical syndrome
- current or prior s/s of HF
- caused by structural of functional cardiac abnormality
- elevated natriuretic peptide levels
- evidence of pulmonary or systemic congestion
who is most at risk for HF black males and females; older women
two biggest causes of HF 1. ischemic heart disease (CAD)
2. HTN
calculate EF SV/LVEDV
synonym for inotropy contraction (HFrEF)
synonym for lusitropy relaxation (HFpEF)
two organizations for classification of HF 1. American Heart Assocation (A,B,C,D) - based on structure
2. New York Heart Association (None, I, I, II, III, IV, V) - based on physical
activity limitations
how to distinguish cardiac dysfunction versus cardiac cardiac dysfunction has structural abnormalities w/o symptoms
HF
once a patient develops symptoms along with structural abnormalities, they
have HF
compensatory mechanisms for HF 1. Hemodynamics (Frank Starling Law) - can give inotropes as short term use to
increase contractility (long-term can cause mortality)
2. SNS activation (increase contractility, tachycardia, vasoconstriction)
3. RAAS activation (angiotensin II binds to AT 1 Receptor - vasoconstriction)
two types of angiotensin II 1. circulating
2. tissue-derived
effects of circulating angiotensin II on HF - causes vasoconstriction
- increases secretion of ADH
- increase aldosterone secretion
- increases sodium reabsorption
are aldosterone levels high or low in HF? 20x higher than normal
Pharmacology Guide| Questions & Answers| Grade A| 100%
Correct (Verified Solutions)- Wilkes
Leave the first rating
Save
Students also studied
Flashcard sets Study guides
NSG 533 Opioid Quiz NSG 533 Exam 3 NSG 533 Exam 3 Op
24 terms 43 terms 45 terms 10
blazierose Preview ksanner13 Preview the_maincharacter Preview
Practice questions for this set
Learn 1 /7 Study with Learn
obesity, diabetes, increased age, black males, family hx
Choose an answer
1 risk factors for HTN 2 risk factors for Pad
risk factors/causes of coronary artery
3 cad risk factors 4
disease
Don't know?
Terms in this set (144)
Universal stages of Heart Failure (HF) - At-risk for HF
- Pre HF
- HF
- Advanced HF
, according to the universal definition, what is "at-risk for - do not have HF, but at risk
HF"? - no current or prior s/s
- no structural, functional, or biomarker evidence supporting HF
according to the universal definition, what is "HF" - clinical syndrome
- current or prior s/s of HF
- caused by structural of functional cardiac abnormality
- elevated natriuretic peptide levels
- evidence of pulmonary or systemic congestion
who is most at risk for HF black males and females; older women
two biggest causes of HF 1. ischemic heart disease (CAD)
2. HTN
calculate EF SV/LVEDV
synonym for inotropy contraction (HFrEF)
synonym for lusitropy relaxation (HFpEF)
two organizations for classification of HF 1. American Heart Assocation (A,B,C,D) - based on structure
2. New York Heart Association (None, I, I, II, III, IV, V) - based on physical
activity limitations
how to distinguish cardiac dysfunction versus cardiac cardiac dysfunction has structural abnormalities w/o symptoms
HF
once a patient develops symptoms along with structural abnormalities, they
have HF
compensatory mechanisms for HF 1. Hemodynamics (Frank Starling Law) - can give inotropes as short term use to
increase contractility (long-term can cause mortality)
2. SNS activation (increase contractility, tachycardia, vasoconstriction)
3. RAAS activation (angiotensin II binds to AT 1 Receptor - vasoconstriction)
two types of angiotensin II 1. circulating
2. tissue-derived
effects of circulating angiotensin II on HF - causes vasoconstriction
- increases secretion of ADH
- increase aldosterone secretion
- increases sodium reabsorption
are aldosterone levels high or low in HF? 20x higher than normal
