1|Page
NUR 334 FINAL EXAM 2025 | ALL QUESTIONS AND
CORRECT ANSWERS | ALREADY GRADED A+ |
VERIFIED ANSWERS | LATEST EXAM
What is the primary mechanism of action for Famotidine?
......ANSWER......It selectively blocks histamine-2 receptor sites, leading
to a reduction in gastric acid secretion and pepsin production.
What is a potential risk of long-term H2 antagonist use due to
decreased gastric acidity? ......ANSWER......Bacterial colonization in the
gut.
What are the common CNS side effects associated with Famotidine,
particularly in older adults? ......ANSWER......Lethargy, hallucinations,
confusion, and restlessness.
Which adverse effects of H2 antagonists are related to the blocking of
androgen receptors? ......ANSWER......Decreased libido, gynecomastia,
and impotence.
What patient education should be provided regarding the
administration of antacids and H2 blockers? ......ANSWER......Antacids
decrease the absorption and effectiveness of H2 blockers; they should
be taken at least 1 hour before or 2 hours after other medications.
pg. 1
,2|Page
What are the primary nursing interventions for a patient experiencing
constipation while taking H2 antagonists or aluminum-based antacids?
......ANSWER......Monitor bowel function, increase fiber and fluid
intake, increase physical activity, and consider stool softeners.
Which patient populations are contraindicated for the use of antacids
containing potassium, magnesium, or aluminum?
......ANSWER......Clients with renal failure.
What is the primary mechanism of action for Proton Pump Inhibitors
(PPIs) like Pantoprazole? ......ANSWER......They act at specific secretory
surface receptors to prevent the final step of acid production,
decreasing overall stomach acid levels.
What are the long-term musculoskeletal risks associated with PPI
therapy? ......ANSWER......Bone loss (osteoporosis) due to impaired
calcium absorption.
What nursing interventions are recommended for patients on long-term
PPI therapy to mitigate bone loss? ......ANSWER......Limit therapy to the
lowest effective dose, monitor bone density, encourage weight-
bearing exercises, and ensure adequate calcium and vitamin D intake.
Why are patients on PPIs at an increased risk for C. difficile infections?
......ANSWER......PPIs lower the stomach's natural acid barrier, which
alters the gut microbiome and allows for bacterial overgrowth.
pg. 2
,3|Page
What are the clinical manifestations of hypomagnesemia that a patient
on PPIs should report? ......ANSWER......Tremors, muscle cramps, and
seizures.
What is 'rebound acid secretion' in the context of PPI discontinuation?
......ANSWER......When a PPI is stopped, elevated gastrin levels
stimulate the stomach to produce more acid than before; it is
managed by tapering the dose slowly.
What respiratory risk is associated with the chronic use of PPIs?
......ANSWER......Community-acquired pneumonia, due to bacterial
colonization in the gut from decreased acidity.
What is the mechanism of action of Sucralfate? ......ANSWER......It forms
an ulcer-adherent complex at duodenal ulcer sites, protecting the area
against acid, pepsin, and bile salts.
What is the correct administration schedule for Sucralfate?
......ANSWER......Four times a day, 1 hour before meals and at bedtime
on an empty stomach.
What is a critical administration precaution for Sucralfate tablets?
......ANSWER......Do not chew the tablets; they can be dissolved in
water to form a slurry, but they pose a choking hazard if swallowed
whole.
pg. 3
, 4|Page
How does the administration of antacids interact with Sucralfate?
......ANSWER......Antacids should not be given within 30 minutes of
Sucralfate administration.
What symptoms of GI bleeding should a patient on anti-ulcer
medications be instructed to report? ......ANSWER......Black stools and
coffee-ground emesis.
Which antacid component is most commonly associated with diarrhea?
......ANSWER......Magnesium salts (e.g., Magnesium hydroxide).
Which antacid components are most commonly associated with
constipation? ......ANSWER......Aluminum and calcium salts.
What is the primary goal of nursing assessment for a patient taking
antacids? ......ANSWER......Monitoring bowel function and watching for
electrolyte abnormalities.
How should a patient be instructed to manage persistent heartburn
while taking OTC H2 blockers? ......ANSWER......Seek medical attention
if symptoms persist or recur, as it may indicate a more serious
underlying disease.
pg. 4
NUR 334 FINAL EXAM 2025 | ALL QUESTIONS AND
CORRECT ANSWERS | ALREADY GRADED A+ |
VERIFIED ANSWERS | LATEST EXAM
What is the primary mechanism of action for Famotidine?
......ANSWER......It selectively blocks histamine-2 receptor sites, leading
to a reduction in gastric acid secretion and pepsin production.
What is a potential risk of long-term H2 antagonist use due to
decreased gastric acidity? ......ANSWER......Bacterial colonization in the
gut.
What are the common CNS side effects associated with Famotidine,
particularly in older adults? ......ANSWER......Lethargy, hallucinations,
confusion, and restlessness.
Which adverse effects of H2 antagonists are related to the blocking of
androgen receptors? ......ANSWER......Decreased libido, gynecomastia,
and impotence.
What patient education should be provided regarding the
administration of antacids and H2 blockers? ......ANSWER......Antacids
decrease the absorption and effectiveness of H2 blockers; they should
be taken at least 1 hour before or 2 hours after other medications.
pg. 1
,2|Page
What are the primary nursing interventions for a patient experiencing
constipation while taking H2 antagonists or aluminum-based antacids?
......ANSWER......Monitor bowel function, increase fiber and fluid
intake, increase physical activity, and consider stool softeners.
Which patient populations are contraindicated for the use of antacids
containing potassium, magnesium, or aluminum?
......ANSWER......Clients with renal failure.
What is the primary mechanism of action for Proton Pump Inhibitors
(PPIs) like Pantoprazole? ......ANSWER......They act at specific secretory
surface receptors to prevent the final step of acid production,
decreasing overall stomach acid levels.
What are the long-term musculoskeletal risks associated with PPI
therapy? ......ANSWER......Bone loss (osteoporosis) due to impaired
calcium absorption.
What nursing interventions are recommended for patients on long-term
PPI therapy to mitigate bone loss? ......ANSWER......Limit therapy to the
lowest effective dose, monitor bone density, encourage weight-
bearing exercises, and ensure adequate calcium and vitamin D intake.
Why are patients on PPIs at an increased risk for C. difficile infections?
......ANSWER......PPIs lower the stomach's natural acid barrier, which
alters the gut microbiome and allows for bacterial overgrowth.
pg. 2
,3|Page
What are the clinical manifestations of hypomagnesemia that a patient
on PPIs should report? ......ANSWER......Tremors, muscle cramps, and
seizures.
What is 'rebound acid secretion' in the context of PPI discontinuation?
......ANSWER......When a PPI is stopped, elevated gastrin levels
stimulate the stomach to produce more acid than before; it is
managed by tapering the dose slowly.
What respiratory risk is associated with the chronic use of PPIs?
......ANSWER......Community-acquired pneumonia, due to bacterial
colonization in the gut from decreased acidity.
What is the mechanism of action of Sucralfate? ......ANSWER......It forms
an ulcer-adherent complex at duodenal ulcer sites, protecting the area
against acid, pepsin, and bile salts.
What is the correct administration schedule for Sucralfate?
......ANSWER......Four times a day, 1 hour before meals and at bedtime
on an empty stomach.
What is a critical administration precaution for Sucralfate tablets?
......ANSWER......Do not chew the tablets; they can be dissolved in
water to form a slurry, but they pose a choking hazard if swallowed
whole.
pg. 3
, 4|Page
How does the administration of antacids interact with Sucralfate?
......ANSWER......Antacids should not be given within 30 minutes of
Sucralfate administration.
What symptoms of GI bleeding should a patient on anti-ulcer
medications be instructed to report? ......ANSWER......Black stools and
coffee-ground emesis.
Which antacid component is most commonly associated with diarrhea?
......ANSWER......Magnesium salts (e.g., Magnesium hydroxide).
Which antacid components are most commonly associated with
constipation? ......ANSWER......Aluminum and calcium salts.
What is the primary goal of nursing assessment for a patient taking
antacids? ......ANSWER......Monitoring bowel function and watching for
electrolyte abnormalities.
How should a patient be instructed to manage persistent heartburn
while taking OTC H2 blockers? ......ANSWER......Seek medical attention
if symptoms persist or recur, as it may indicate a more serious
underlying disease.
pg. 4
