NUR 6011 Exam 1 Questions and 100% Correct Answers 2026/27 Update – William
Paterson University
Q U ESTIO N 1
DS has been receiving digoxin, furosemide and lisinopril for several months to treat for CHF. At 3
months it was noted his SrCr went from 1.2 mg/dl to 2.4 mg/dl. What could explain this?
Digoxin lead to pre-renal azotemia
Lisinopril caused an acute decompensation in renal function because the patient had
bilateral renal artery stenosis.
A medication error most likely occurred and the patient was actually given irbesartan.
lisinopril resulted in a high potassium level which then resulted in a decline in renal function.
Q U ESTIO N 2
1. LP is a 56 yo type 1 diabetic with HTN new to your practice. Upon taking a history it is determined he
stopped taking enalapril 8 years ago due to a cough. He has been taking Carvedilol for his BP which is
not very well controlled. Which is correct.
Losartan may be an alternative to the carvedilol, so the carvedilol can be abruptly stopped and
losartan started
Enalapril should not have been used in a diabetic because it is associated with a high risk of
angioedema and cough. Losartan may be an option for this patient since the risk of
cough is much less in the diabetic population
Valsartan may be an alternavie, so it can be started while the carvedilol is slowly weaned off
Neither losratan nor valsartan is an option since the patient has a history of coughing from
enalapril. Another class of medication may be a better choice
Q U ESTIO N 3
1. WR is a 72 yo male with a digoxin level of 1.8 mcg/ml. He has been taking this drug for several
months along with furosemide and spironolactone. Up until today he has had with no complaints but
at today’s visit to the NP he is sluggish. He is noted to have a HR of 42 BPM, BP 120/80 and loss of
appetite. Which is correct?
High serum potassium can lead to digoxin toxicity with normal digoxin levels
Low serum potassium can lead to digoxin toxicity but the digoxin level should not be low in face of
digoxin toxicity
Low serum potassium can lead to digoxin toxicity with normal digoxin levels High serum
potassium levels may be a marker of severe digoxin toxicity
NUR 6011 Exam 1
, Low serum potassium can lead to digoxin toxicity with normal digoxin levels and high serum
potassium levels may be a marker of severe digoxin toxicity
High serum potassium can lead to digoxin toxicity with normal digoxin levels and low serum potassium
levels may be a marker of severe digoxin toxicity
Q U ESTIO N 4
1. KL is a 66 yo female with vasospastic angina? Which of the following medications may be the
best option?
metoprol ol
amlodipi
ne
ranolazin e
atenolol
Q U ESTIO N 5
1. KL is a 57 yo male that is being seen in the ED for suspected food poisoning. He had
had Nausea and some vomiting for 2 days. he has a K of
5.6 mEq/L, SrCr of 1.9 and BUN 23. ECG shows a prolonged PR interval (2 nd
degree AV block). His digoxin level is 4.1 ng/ml taken ~ 3 hrs post ingestion. Based
upon this one can say?
The serum digoxin level was likely taken too soon after ingestion and one should wait at
least 6-8 hrs post ingestion to get a level
With the elevated SrCr the GI complaints are likely from uremia.
It appears this patient is experiencing the signs and symptoms of digoxin toxicity
This patient should immediately be given lipid rescue
Q U ESTIO N 6
1. Beta blockers are the drugs of choice for effort induced angina because they
primarily:
NUR 6011 Exam 1
Paterson University
Q U ESTIO N 1
DS has been receiving digoxin, furosemide and lisinopril for several months to treat for CHF. At 3
months it was noted his SrCr went from 1.2 mg/dl to 2.4 mg/dl. What could explain this?
Digoxin lead to pre-renal azotemia
Lisinopril caused an acute decompensation in renal function because the patient had
bilateral renal artery stenosis.
A medication error most likely occurred and the patient was actually given irbesartan.
lisinopril resulted in a high potassium level which then resulted in a decline in renal function.
Q U ESTIO N 2
1. LP is a 56 yo type 1 diabetic with HTN new to your practice. Upon taking a history it is determined he
stopped taking enalapril 8 years ago due to a cough. He has been taking Carvedilol for his BP which is
not very well controlled. Which is correct.
Losartan may be an alternative to the carvedilol, so the carvedilol can be abruptly stopped and
losartan started
Enalapril should not have been used in a diabetic because it is associated with a high risk of
angioedema and cough. Losartan may be an option for this patient since the risk of
cough is much less in the diabetic population
Valsartan may be an alternavie, so it can be started while the carvedilol is slowly weaned off
Neither losratan nor valsartan is an option since the patient has a history of coughing from
enalapril. Another class of medication may be a better choice
Q U ESTIO N 3
1. WR is a 72 yo male with a digoxin level of 1.8 mcg/ml. He has been taking this drug for several
months along with furosemide and spironolactone. Up until today he has had with no complaints but
at today’s visit to the NP he is sluggish. He is noted to have a HR of 42 BPM, BP 120/80 and loss of
appetite. Which is correct?
High serum potassium can lead to digoxin toxicity with normal digoxin levels
Low serum potassium can lead to digoxin toxicity but the digoxin level should not be low in face of
digoxin toxicity
Low serum potassium can lead to digoxin toxicity with normal digoxin levels High serum
potassium levels may be a marker of severe digoxin toxicity
NUR 6011 Exam 1
, Low serum potassium can lead to digoxin toxicity with normal digoxin levels and high serum
potassium levels may be a marker of severe digoxin toxicity
High serum potassium can lead to digoxin toxicity with normal digoxin levels and low serum potassium
levels may be a marker of severe digoxin toxicity
Q U ESTIO N 4
1. KL is a 66 yo female with vasospastic angina? Which of the following medications may be the
best option?
metoprol ol
amlodipi
ne
ranolazin e
atenolol
Q U ESTIO N 5
1. KL is a 57 yo male that is being seen in the ED for suspected food poisoning. He had
had Nausea and some vomiting for 2 days. he has a K of
5.6 mEq/L, SrCr of 1.9 and BUN 23. ECG shows a prolonged PR interval (2 nd
degree AV block). His digoxin level is 4.1 ng/ml taken ~ 3 hrs post ingestion. Based
upon this one can say?
The serum digoxin level was likely taken too soon after ingestion and one should wait at
least 6-8 hrs post ingestion to get a level
With the elevated SrCr the GI complaints are likely from uremia.
It appears this patient is experiencing the signs and symptoms of digoxin toxicity
This patient should immediately be given lipid rescue
Q U ESTIO N 6
1. Beta blockers are the drugs of choice for effort induced angina because they
primarily:
NUR 6011 Exam 1
