BSMCON PHR 3101 Exam 5 - FINAL non-
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Terms in this set (66)
What is the purpose of cholesterol in For energy utilization, for cell membrane
the body? stabilization, for steroid and hormones production,
for bile acid formation but also lipid deposition
What are lipoproteins and how are Consisted of various amounts of cholesterol,
they classified? triglycerides, and phospholipids along with a
protein carrier. They are classified by their size,
weight or density.
What are the general goals for lipids -Total cholesterol >200
in the blood? - TG > 150
-LDL (lousy) - transports cholesterol from liver to
tissues >100
-HDL - transport surpluses cholesterol from tissues
back to the liver for destruction <40 >60
What is atherosclerosis and why is it -Hardening of the arteries- fat build up.
dangerous? · Large plaque can reduce blood flow.
· Plaque ruptures causing a blood clot.
· Plaque breaks off (embolism) and travels.
, What are the three major injurious HTN, Hyperglycemia, Hyperlipidemia increases the
agents to the blood vessel chance of atherosclerosis
endothelium (trifecta)?
If atherosclerosis is diagnosed, how - Stabilize and prevent plaque formation with
do we treat it? Why is it important to statins
treat as soon as possible? - Decrease risk of thrombus formation with aspirin
- Decrease cardiovascular risk: BP control, BG
control, fish oil, diet, and exercise
Factors that decrease heart oxygen - Coronary plaque presence
supply: - Decrease perfusion pressure
- Decreased arterial oxygen content (Ex: COPD)
Factors that increase heart oxygen -Increaser HR
demand: - Increase Preload
- Increase afterload.
-Increase Contractility
Why is a nitroglycerin-free period - Do not discontinue abruptly - lead to rebound
required for the patch and ointment? issues
- 10-12 nitrate-free to prevent tolerance
Beta blockers -Decrease O2 demand by decreasing HR and
contractility
How do these medications work for -Because is not related to exertion (the 3 triad) it is
CAD? not predictable
Why do we not use it for variant
angina treatment?
cumulative Ultimate Exam | Questions and
Answers | Verified Solutions | 2026 Edition | Pass
Guaranteed
Save
Terms in this set (66)
What is the purpose of cholesterol in For energy utilization, for cell membrane
the body? stabilization, for steroid and hormones production,
for bile acid formation but also lipid deposition
What are lipoproteins and how are Consisted of various amounts of cholesterol,
they classified? triglycerides, and phospholipids along with a
protein carrier. They are classified by their size,
weight or density.
What are the general goals for lipids -Total cholesterol >200
in the blood? - TG > 150
-LDL (lousy) - transports cholesterol from liver to
tissues >100
-HDL - transport surpluses cholesterol from tissues
back to the liver for destruction <40 >60
What is atherosclerosis and why is it -Hardening of the arteries- fat build up.
dangerous? · Large plaque can reduce blood flow.
· Plaque ruptures causing a blood clot.
· Plaque breaks off (embolism) and travels.
, What are the three major injurious HTN, Hyperglycemia, Hyperlipidemia increases the
agents to the blood vessel chance of atherosclerosis
endothelium (trifecta)?
If atherosclerosis is diagnosed, how - Stabilize and prevent plaque formation with
do we treat it? Why is it important to statins
treat as soon as possible? - Decrease risk of thrombus formation with aspirin
- Decrease cardiovascular risk: BP control, BG
control, fish oil, diet, and exercise
Factors that decrease heart oxygen - Coronary plaque presence
supply: - Decrease perfusion pressure
- Decreased arterial oxygen content (Ex: COPD)
Factors that increase heart oxygen -Increaser HR
demand: - Increase Preload
- Increase afterload.
-Increase Contractility
Why is a nitroglycerin-free period - Do not discontinue abruptly - lead to rebound
required for the patch and ointment? issues
- 10-12 nitrate-free to prevent tolerance
Beta blockers -Decrease O2 demand by decreasing HR and
contractility
How do these medications work for -Because is not related to exertion (the 3 triad) it is
CAD? not predictable
Why do we not use it for variant
angina treatment?
