NSG 3600 Peds Exam 2 – Questions & Accurate
Solutions
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Terms in this set (133)
Congestive Heart Failure Etiology Inability of the heart to perform is function of moving
blood forward. Caused by CHD, dysrhythmias,
cardiomyopathy, Kawasaki's.
Congestive Heart Failure Signs and Poor feed, poor growth, irritability, shortness of air,
Symptoms for Baby exercise intolerance, peripheral edema (puffy legs
and feet). The child uses a lot of energy in feeding so
they can get tired which causes poor feeding.
Congestive Heart Failure Monitor Vital signs, keep the child comfortable,
Treatment/Interventions oxygenation when needed. Give medications
(Angiotensin Converting Enzyme -ACE- Inhibitor
(Captopril) Vasodilation, Cardiac Glycoside (Digoxin),
Loop Diuretic (Furosemide)).
What is important information to know Makes the heart pump harder so we need to make
about Digoxin? sure we DO NOT give this medication if their heart
rate is less than 90 in infant OR 70 in child. Toxicity of
this drug can cause bradycardia and NV.
What is important information we We need to watch the potassium levels when it
need to know about Furosemide when comes to this drug. If the levels begin to decrease
it comes to Congestive Heart Failure? the digoxin levels will increase causing toxicity.
Atrial Septal Defect (ASD) Simple defect of the atria results when the two
septae fail to overlap properly. Will occur in the
second intercostal space and is on the LEFT side.
Sound will either be blowing or very harsh. These
kids will be surgery due to their defect not closing on
its own.
,Ventricular Septal Defect (VSD) MOST COMMON! Defect of the ventricles when the
two septae fail to overlap properly. Is in the lower
intercostal space. You can feel it with your hand. The
spleen and liver will be enlarged due to extra blood
flow. The sound will be very harsh sounding.
Treatment for Atrial Septal Defect Watch for spontaneous closure, surgical repair,
(ASD) and Ventricular Septal Defect monitor vitals signs, watch for bleeding tamponade
(VSD) or migration of the device (chest pain, sudden
decrease in BP, palpation) Watch oxygenation and
do congestive heart failure management.
Patent Ductus Arteriosus (PDA) In utero, the shunt serves the purpose to bypass the
lungs. The ducts starts to close usually within 48
hours but can take as long as 1 year. If the whole is
too large we would need to patch it. This causes
blood to go into the lungs that is not oxygenated.
Patent Ductus Arteriosus (PDA) Harsh murmur-Left subconfiural margin sound
Clinical Manifestations machine like and depends on the side of the defect.
The smaller it is the louder it is. Systolic is higher and
the Diastolic gets lower. Frequent colds, susceptible
to RSV, poor feeding/poor growth pattern.
Patent Ductus Arteriosus (PDA) Medical care is usually closure by surgery or a
Therapeutic Management transcatheter device. Postop measures are wound
care, monitoring vital signs, hydration/nutrition.
Pulmonic Stenosis: Pathophysiology Malformation of the pulmonary artery or pulmonic
valve. Increase workload on right ventricle.
Pulmonic Stenosis: Clinical These kids could have their valve replace. If any of
Manifestations these kids have oxygen issues while on wall oxygen
there are more issues and other problems.
Pulmonic Stenosis: Therapeutic Reducing stressful situations that may cause high
Management blood pressure. Angioplasty or valvulopasty.
, Aortic Stenosis: Pathophysiology Malformation and narrowing in the aorta or around
the aortic valve. Kids can pass out from overexertion
and can cause heart failure.
Coarctation of the Aorta: A narrowing or constriction of the descending aorta
Pathophysiology distal to the carotid arteries. Decrease blood
pressure and decrease pulses in lower part of the
lower body. Which makes the upper body have
bounding and high pressure.
Coarctation of the Aorta: Clinical Murmur, Signs of Congestive Heart Failure, pain in
Manifestations the legs or cyanotic lower extremities, increase
pressure to head and upper extremities, decrease
pressure to lower extremities (cold and hard to
perfused)
Coarctation of the Aorta: Therapeutic Surgery-Nursing care consist of post op
Management management.
Cardiac Cath- angioplasty and valvuloplasty.
Captopril (Capoten) or enalapril (Vasotec). Can also
give Digoxin and Lasix.
Tricuspid Atresia: Pathophysiology Caused by an error in the formation if the tricuspid
valve. As a single defect this condition is
incompatible with life. Most children born with TA
also have ASD or VSD. The deoxygenated blood
must reach the pulmonary bed to sustain life. SINGLE
DEFECT where the deoxygenated blood never
reaches the lungs which means its impossible for
these kids to survive.
When is a tricuspid atresia found? It is found in prenatal visit at 20 weeks (child will have
surgery right after birth) If not found or caught the
baby will turn blue and the baby will need surgery
right away to see where the issue is.
Tricuspid Atresia: Clinical Cyanosis-a bluish discoloration of the skin resulting
Manifestations from poor circulation
Tachycardia-a rapid heartbeat
Dyspnea-difficult or labored breathing.
Solutions
Save Groups
Terms in this set (133)
Congestive Heart Failure Etiology Inability of the heart to perform is function of moving
blood forward. Caused by CHD, dysrhythmias,
cardiomyopathy, Kawasaki's.
Congestive Heart Failure Signs and Poor feed, poor growth, irritability, shortness of air,
Symptoms for Baby exercise intolerance, peripheral edema (puffy legs
and feet). The child uses a lot of energy in feeding so
they can get tired which causes poor feeding.
Congestive Heart Failure Monitor Vital signs, keep the child comfortable,
Treatment/Interventions oxygenation when needed. Give medications
(Angiotensin Converting Enzyme -ACE- Inhibitor
(Captopril) Vasodilation, Cardiac Glycoside (Digoxin),
Loop Diuretic (Furosemide)).
What is important information to know Makes the heart pump harder so we need to make
about Digoxin? sure we DO NOT give this medication if their heart
rate is less than 90 in infant OR 70 in child. Toxicity of
this drug can cause bradycardia and NV.
What is important information we We need to watch the potassium levels when it
need to know about Furosemide when comes to this drug. If the levels begin to decrease
it comes to Congestive Heart Failure? the digoxin levels will increase causing toxicity.
Atrial Septal Defect (ASD) Simple defect of the atria results when the two
septae fail to overlap properly. Will occur in the
second intercostal space and is on the LEFT side.
Sound will either be blowing or very harsh. These
kids will be surgery due to their defect not closing on
its own.
,Ventricular Septal Defect (VSD) MOST COMMON! Defect of the ventricles when the
two septae fail to overlap properly. Is in the lower
intercostal space. You can feel it with your hand. The
spleen and liver will be enlarged due to extra blood
flow. The sound will be very harsh sounding.
Treatment for Atrial Septal Defect Watch for spontaneous closure, surgical repair,
(ASD) and Ventricular Septal Defect monitor vitals signs, watch for bleeding tamponade
(VSD) or migration of the device (chest pain, sudden
decrease in BP, palpation) Watch oxygenation and
do congestive heart failure management.
Patent Ductus Arteriosus (PDA) In utero, the shunt serves the purpose to bypass the
lungs. The ducts starts to close usually within 48
hours but can take as long as 1 year. If the whole is
too large we would need to patch it. This causes
blood to go into the lungs that is not oxygenated.
Patent Ductus Arteriosus (PDA) Harsh murmur-Left subconfiural margin sound
Clinical Manifestations machine like and depends on the side of the defect.
The smaller it is the louder it is. Systolic is higher and
the Diastolic gets lower. Frequent colds, susceptible
to RSV, poor feeding/poor growth pattern.
Patent Ductus Arteriosus (PDA) Medical care is usually closure by surgery or a
Therapeutic Management transcatheter device. Postop measures are wound
care, monitoring vital signs, hydration/nutrition.
Pulmonic Stenosis: Pathophysiology Malformation of the pulmonary artery or pulmonic
valve. Increase workload on right ventricle.
Pulmonic Stenosis: Clinical These kids could have their valve replace. If any of
Manifestations these kids have oxygen issues while on wall oxygen
there are more issues and other problems.
Pulmonic Stenosis: Therapeutic Reducing stressful situations that may cause high
Management blood pressure. Angioplasty or valvulopasty.
, Aortic Stenosis: Pathophysiology Malformation and narrowing in the aorta or around
the aortic valve. Kids can pass out from overexertion
and can cause heart failure.
Coarctation of the Aorta: A narrowing or constriction of the descending aorta
Pathophysiology distal to the carotid arteries. Decrease blood
pressure and decrease pulses in lower part of the
lower body. Which makes the upper body have
bounding and high pressure.
Coarctation of the Aorta: Clinical Murmur, Signs of Congestive Heart Failure, pain in
Manifestations the legs or cyanotic lower extremities, increase
pressure to head and upper extremities, decrease
pressure to lower extremities (cold and hard to
perfused)
Coarctation of the Aorta: Therapeutic Surgery-Nursing care consist of post op
Management management.
Cardiac Cath- angioplasty and valvuloplasty.
Captopril (Capoten) or enalapril (Vasotec). Can also
give Digoxin and Lasix.
Tricuspid Atresia: Pathophysiology Caused by an error in the formation if the tricuspid
valve. As a single defect this condition is
incompatible with life. Most children born with TA
also have ASD or VSD. The deoxygenated blood
must reach the pulmonary bed to sustain life. SINGLE
DEFECT where the deoxygenated blood never
reaches the lungs which means its impossible for
these kids to survive.
When is a tricuspid atresia found? It is found in prenatal visit at 20 weeks (child will have
surgery right after birth) If not found or caught the
baby will turn blue and the baby will need surgery
right away to see where the issue is.
Tricuspid Atresia: Clinical Cyanosis-a bluish discoloration of the skin resulting
Manifestations from poor circulation
Tachycardia-a rapid heartbeat
Dyspnea-difficult or labored breathing.
