Care of Critically Ill Patients with Neurologic Problems / Chapter 47 (25
questions)
Traumatic Brain Injury
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1. Contrecoup injury
Coup is an impact to the frontal lobe; countrecoup is the impact to the
occipital area from the brain bouncing backwards.
2. Contusion
Bruising of the brain tissue; structural damage
3. Concussion
Shaky movement of the brain; may lose consciousness; retrograde amnesia;
NO structural damage.
Client teaching: have pt. woken every 3-4hrs to assess LOC; EXPECT
headache, nausea and dizziness for 24hrs, if gets worse or does not subside,
go back to hospital; no alcohol, sedatives or sleeping pills, give Tylenol prn
every 4hrs; GO BACK TO HOSPITAL IF THESE SYMPTOMS OCCUR: blurred
vision, rhinorrhea or ottorrhea, weakness, slurred speech, progressive
sleepiness, vomiting, unequal pupil size, and worsening headache.
4. Basilar skull fracture (unique fracture)*
Fracture at base of skull; causes CSF leakage of the nose (rhinorrhea) and
the ears (ottorrhea);
Risk for INFECTION r/t direct access to subarachnoid space.
Potential for hemorrhage: Raccoon eyes (bleeding around orbits of eyes) and
Battle Sign (bruising behind ears)
Hemorrhage (Brain)
*All hematomas are potentially life-threatening because they act as space-occupying lesions
and are surrounded by edema, thus increasing ICP.
1. Epidural hematoma
Located above dura; primarily an arterial bleed.
2. Subdural hematoma
Located below dura and above arachnoid; primarily a venous bleed.
Slow bleed; acute stage happens within 48hrs. after impact; high mortality rate;
usually goes unrecognized.
Earliest sign is a change in personality; Ask pt. if they fell or hit their
headhowcheck headput on gloves and palpate.
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, 3. Intracerebral hemorrhage
Can be both venous and an arterial bleed; usually results from a blown
aneurysm; causes increased ICP.
Increased Intracranial Pressure
**Normal ICP: 10-15 mmHg
1. Earliest change:
Change in LOC r/t pressure on frontal lobe, best indicator!!!!
Other early s/s: headache and projectile N/V
2. Cushing’s triad-:
classic, late sign: severe hypertension with widened pulse pressure and
bradycardia
3. Pupillary changes :
ovoid pupil: midstage between a normal pupil and a dilated pupil
pupils that are dilated and fixed (‘blown’) r/t pressure exerted onto III cranial
nerve. Cranial nerve III directly affects pupils.
4. EOMs diminished- CN III, IV, and VI
EOM: Extra Ocular Eye Movement
These nerves regulate eye muscle movement
Diminished cranial nerves r/t increased pressure on brain stem (where these
nerves are located); since pressure moves downward from frontal lobe to
brainstem, once pressure gets to this point, it means increased ICP is severe.
Pinpoint and nonresponsive pupils are indicative of brainstem dysfunction at
the level of the pons.
5. Papilledema
Choked disk (edema and hyperemia; increased blood flow of optic disk; only
seen with an ophthalmoscope)
6. Decorticate and decerebrate posturing
Decorticate: Upper extremeties are flexed inwards at core
Decerebrate: upper extremeties/wrists are tensed outwards at sides; this is
more severe than decorticate, indicates more damage to brain.
7. CSF leak- “halo” sign
CSF contains glucose and protein
To assess for CSF leak, obtain gauze to absorb fluid, fluid will scatter outwards
forming a “yellowish halo”.
8. Glasgow Coma Scale
Three major areas assessed on scale: Eye opening, motor response and verbal
response
Graded from 3-15; 3 being the worst, 15 the best.
9. Brain Herniation – death
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