Diagnosis: Migraine vs. Tension headaches
Physical
Signs and
Pathophysiology Assessment Pharmacologic
Symptoms
Summary Findings Recommendations
(subjective)
(objective)
Diagnosis
#1: The exact Each patient is Upon office After determining the
Migraines pathophysiology of different and may examination patient is experience
migraines is not fully experience assess headache: migraines there are
understood but various level of Onset different treatment
based on symptoms. There approaches that can
researchers it is are four different Duration help alleviate the
linked to abnormal phases that Severity episode and pain. At
changes in the blood patient experience this point, the patient
flow and affects different Frequency may be able to identify
specific brain symptoms: triggers that will help
structures. It is stated Premonitory: 2 Location and avoid having migraine
that the following hours before quality of pain episodes. Avoiding
reaction occur during headache If presence of triggers will help
a migraine attack: onset: aura identify adjustments
Activation of symptoms patient might require in
trigeminal Changes in their lifestyles (diet,
vascular system mood and Inquire any have extra snacks to
which involves activity known triggers avoid starving, make
meningeal Irritability sure they are sleeping
vasodilation, Inquire impact enough, etc.). The next
neurogenic Fatigue on daily life approach would be
, inflammation and
central Food cravings Order lab test to medications that will
sensitization rule out any help alleviate the pain.
Repetitive underlying The following
(Parks-Chapman yawns
and Schub, 2018) conditions: medications are
Stiff neck recommended:
Increases neural CBC
activities spreads Phobia Mild migraines:
BMP 1) NSAID: analgesic/
in the brain
initiating pain Aura: Urine analysis anti-inflammatory
stimuli in experience (Advil/Motrin/etc.)
trigeminal system, neurological Drug test Mechanism of action:
which then deficits, fully Inhibits
reversible Pregnancy test
conducted to the cyclooxygenase
thalamus and (visual, enzymes COX-1 and
pain centers in sensory, COX-2 which makes
the sensory speech/
an anti-inflammatory
cortex (Hubert & language,
and analgesic effect on
VanMeter, 2018) motor brain
stem, retinal the body.
Reduction of 2) Acetaminophen:
serotonin in the Could be analgesic/
brain unilateral antipyretic (Tylenol)
Symptoms Mechanism of action:
Release of “primarily inhibit the
neuropeptides may last 4-72
hours cyclooxygenase (COX)
(substance P,
enzyme, decreasing
vasoactive Headache the synthesis of
intestinal attacks: last
polypeptide (VIP), prostaglandins, and
about 4-72
and calcitonin modulating pain and
hours with at
gene-related temperature”.
least 2 out 4
peptide (CGRP) characteristics (Candidio, Perozo &
in the meninges
Physical
Signs and
Pathophysiology Assessment Pharmacologic
Symptoms
Summary Findings Recommendations
(subjective)
(objective)
Diagnosis
#1: The exact Each patient is Upon office After determining the
Migraines pathophysiology of different and may examination patient is experience
migraines is not fully experience assess headache: migraines there are
understood but various level of Onset different treatment
based on symptoms. There approaches that can
researchers it is are four different Duration help alleviate the
linked to abnormal phases that Severity episode and pain. At
changes in the blood patient experience this point, the patient
flow and affects different Frequency may be able to identify
specific brain symptoms: triggers that will help
structures. It is stated Premonitory: 2 Location and avoid having migraine
that the following hours before quality of pain episodes. Avoiding
reaction occur during headache If presence of triggers will help
a migraine attack: onset: aura identify adjustments
Activation of symptoms patient might require in
trigeminal Changes in their lifestyles (diet,
vascular system mood and Inquire any have extra snacks to
which involves activity known triggers avoid starving, make
meningeal Irritability sure they are sleeping
vasodilation, Inquire impact enough, etc.). The next
neurogenic Fatigue on daily life approach would be
, inflammation and
central Food cravings Order lab test to medications that will
sensitization rule out any help alleviate the pain.
Repetitive underlying The following
(Parks-Chapman yawns
and Schub, 2018) conditions: medications are
Stiff neck recommended:
Increases neural CBC
activities spreads Phobia Mild migraines:
BMP 1) NSAID: analgesic/
in the brain
initiating pain Aura: Urine analysis anti-inflammatory
stimuli in experience (Advil/Motrin/etc.)
trigeminal system, neurological Drug test Mechanism of action:
which then deficits, fully Inhibits
reversible Pregnancy test
conducted to the cyclooxygenase
thalamus and (visual, enzymes COX-1 and
pain centers in sensory, COX-2 which makes
the sensory speech/
an anti-inflammatory
cortex (Hubert & language,
and analgesic effect on
VanMeter, 2018) motor brain
stem, retinal the body.
Reduction of 2) Acetaminophen:
serotonin in the Could be analgesic/
brain unilateral antipyretic (Tylenol)
Symptoms Mechanism of action:
Release of “primarily inhibit the
neuropeptides may last 4-72
hours cyclooxygenase (COX)
(substance P,
enzyme, decreasing
vasoactive Headache the synthesis of
intestinal attacks: last
polypeptide (VIP), prostaglandins, and
about 4-72
and calcitonin modulating pain and
hours with at
gene-related temperature”.
least 2 out 4
peptide (CGRP) characteristics (Candidio, Perozo &
in the meninges
