Venous Thromboembolic
Disorders
Tammy L. Gravel MS RN EdD (c)
NUR 226 Pathophysiology/Pharmacology
MCPHS University
School of Nursing
Fall 2017
, Class Objectives
Describe venous thromboembolic disorders to include risk
stratification, diagnosis and treatment of;
DVT
PE
Discuss pharmacological strategies for the prophylaxis
and treatment of VTE
Identify appropriate nursing considerations appropriate
for the pharmacological strategies
Highlight key information necessary for patient education
for both disease process and pharmacological
interventions
, Hemostasis
Hemostasis is the normal mechanism to
prevent both unnecessary clotting or
bleeding.
Dependent on role of blood vessels, platelets
and coagulation. (Virchow’s Triad)
Coagulation begins when blood or platelets
contact something they should not,
extravascular tissue or a foreign surface
Clotting factors most are manufactured by the
liver and are denoted by names and Roman
Numerals
Intrinsic and Extrinsic coagulation pathways
, Homeostasis
Disorders
Tammy L. Gravel MS RN EdD (c)
NUR 226 Pathophysiology/Pharmacology
MCPHS University
School of Nursing
Fall 2017
, Class Objectives
Describe venous thromboembolic disorders to include risk
stratification, diagnosis and treatment of;
DVT
PE
Discuss pharmacological strategies for the prophylaxis
and treatment of VTE
Identify appropriate nursing considerations appropriate
for the pharmacological strategies
Highlight key information necessary for patient education
for both disease process and pharmacological
interventions
, Hemostasis
Hemostasis is the normal mechanism to
prevent both unnecessary clotting or
bleeding.
Dependent on role of blood vessels, platelets
and coagulation. (Virchow’s Triad)
Coagulation begins when blood or platelets
contact something they should not,
extravascular tissue or a foreign surface
Clotting factors most are manufactured by the
liver and are denoted by names and Roman
Numerals
Intrinsic and Extrinsic coagulation pathways
, Homeostasis
