NR507 Week 2 Quiz (2 Versions, Latest-2022)/ NR 507 Week 2 Quiz: Advanced Pathophysiology: Chamberlain College of Nursing |100% Correct Answers, Already Graded “A”|

NR507 Week 2 Quiz (2 Versions, Latest-2022)/ NR 507 Week 2 Quiz: Advanced Pathophysiology: Chamberlain College of Nursing |100% Correct Answers, Already Graded “A”| NR507 Week 2 Quiz ( 2 Latest versions): Advanced Pathophysiology: Chamberlain College of Nursing NR 507 Week 2 (Latest): Advanced Pathophysiology: Chamberlain College of Nursing 1. Question : In hyperkalemia, cardiac rhythm changes are a direct result of cardiac cell hypopolarization. cardiac cell hyperexcitability. depression of the sinoatrial (SA) node. cardiac cell repolarization. Question 2. Question : Which are indications of dehydration? Decreased hemoglobin and hematocrit Muscle weakness and decreased deep tendon reflexes Tachycardia and weight loss Polyuria and hyperventilation Question 3. Question : How does the loss of chloride during vomiting cause metabolic alkalosis? Loss of chloride stimulates the release of aldosterone, which causes the retained sodium to bind with the chloride. Loss of chloride causes hydrogen to move into the cell and exchange with potassium to maintain cation balance. Loss of chloride causes retention of bicarbonate to maintain the anion balance. Loss of chloride causes hypoventilation to compensate for the metabolic alkalosis. Question 4. Question : When thirst is experienced, how are osmoreceptors activated? By an increase in the antidiuretic hormone secreted into the plasma By an increase in aldosterone secreted into the plasma By an increase in the hydrostatic pressure of the plasma By an increase in the osmotic pressure of the plasma Question 5. Question : At the arterial end of capillaries, fluid moves from the intravascular space into the interstitial space because the interstitial hydrostatic pressure is higher than the capillary hydrostatic pressure. capillary hydrostatic pressure is higher than the capillary oncotic pressure. interstitial oncotic pressure is higher than the interstitial hydrostatic pressure. capillary oncotic pressure is lower than the interstitial hydrostatic pressure. Question 6. Question : What is a major determinant of the resting membrane potential necessary for transmission of nerve impulses? The ratio between intracellular Na+ and extracellular sodium The ratio between intracellular K+ and extracellular potassium The ratio between intracellular Na+ and extracellular K+ The ratio between intracellular K+ and extracellular Na+ Question 7. Question : Chvostek sign and Trousseau sign indicate hypokalemia. hyperkalemia. hypocalcemia. hypercalcemia. Question 8. Question : Which of the following is a true statement? Hypoventilation causes hypocapnia. Hyperventilation causes hypercapnia. Hyperventilation causes hypocapnia. Hyperventilation results in an increased PaCO2. Question 9. Question : In ARDS, alveoli and respiratory bronchioles fill with fluid as a result of the compression on the pores of Kohn, thus preventing collateral ventilation. increased capillary permeability, which causes alveoli and respiratory bronchioles to fill with fluid. inactivation of surfactant and the impairment of type II alveolar cells. increased capillary hydrostatic pressure that forces fluid into the alveoli and respiratory bronchioles. Question 10. Question : Which inflammatory mediators are produced in asthma? Neutrophil proteases, bradykinin, and histamine Bradykinin, serotonin, and neutrophil proteases Histamine, prostaglandins, and leukotrienes Lymphokines, serotonin, and prostaglandins Question 11. Question : Which pleural abnormality involves a site of pleural rupture that act as a one-way valve, permitting air to enter on inspiration but preventing its escape by closing during expiration? Spontaneous pneumothorax Tension pneumothorax Open pneumothorax Secondary pneumothorax Question 12. Question : _____ is a fulminant form of respiratory failure characterized by acute lung inflammation and diffuse alveolocapillary injury. Acute respiratory distress syndrome (ARDS) Pneumonia Pulmonary emboli Acute pulmonary edema Question 13. Question : A(n) _____ is a circumscribed area of suppuration and destruction of lung parenchyma. consolidation cavitation empyema abscess Question 14. Question : Dyspnea is not a result of decreased pH, increased PaCO2, and decreased PaO2. decreased blood flow to the medulla oblongata. stimulation of stretch or J-receptors. fatigue of the intercostal muscles and diaphragm. Question 15. Question : Clinical manifestations that include unexplained weight loss, dyspnea on exertion, use of accessory muscles, and tachypnea with prolonged expiration are indicative of chronic bronchitis. emphysema. pneumonia. asthma. Question 16. Question : Pulmonary edema usually begins at a pulmonary capillary wedge pressure or left atrial pressure of _____ mm Hg. 10 20 30 40 Question 17. Question : High altitudes may produce hypoxemia through shunting. hypoventilation. decreased inspired oxygen. diffusion abnormalities. Question 18. Question : Kussmaul respirations may be characterized as a respiratory pattern with alternating periods of deep and shallow breathing. commonly observed in pulmonary fibrosis. commonly observed in chronic obstructive pulmonary disease. with a slightly increased ventilatory rate, large tidal volumes, and no expiratory pause. Question 19. Question : Cystic fibrosis (CF) is caused by a(n) autosomal recessive inheritance. autosomal dominant inheritance. infection. malignancy. Question 20. Question : An accurate description of childhood asthma is that it is a(n) obstructive airway disease characterized by reversible airflow obstruction, bronchial hyperreactivity, and inflammation. pulmonary disease characterized by severe hypoxemia, decreased pulmonary compliance, and diffuse densities on chest x-ray. pulmonary disorder involving an abnormal expression of a protein producing viscous mucus that lines the airways, pancreas, sweat ducts, and vas deferens. obstructive airway disease characterized by atelectasis and increased pulmonary resistance as a result of a surfactant deficiency. Question 21. Question : What is the primary cause of RDS of the newborn? An immature immune system Small alveoli A surfactant deficiency Anemia Question 22. Question : Chest wall compliance in infants is _____ in adults. lower than higher than the same as unlike that Question 23. Question : The release of fibroblast growth factors affects ARDS by causing atelectasis and decreased lung compliance. disruption of alveolocapillary membrane. pulmonary hypertension. pulmonary fibrosis. Question 24. Question : Which of the following statements about the advances in the treatment of RDS of the newborn is incorrect? Administering glucocorticoids to women in preterm labor accelerates the maturation of the fetus’s lungs. Administering oxygen to mothers during preterm labor increases their arterial oxygen before birth of the fetus. Treatment includes the instillation of exogenous surfactant down an endotracheal tube of infants weighing less than 1,000 g. Supporting the infant’s respiratory function by using continuous positive airway pressure (CPAP). An infant’s respiratory function is supported by using continuous pressure (CPAP). Question 25. Question : Which immunoglobulin is present in childhood asthma? IgM IgG IgE IgA