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NUR 2063 Essentials of Pathophysiology
(NUR2063) / Pathophysiology Final Rasmussen
University Winter 2022
1. •Neither recessive nor dominant—co-dominant.
• Hemoglobin S causes erythrocytes to be abnormally shaped.
• Abnormal erythrocytes carry less oxygen and clog vessels, causing hypoxia and tissue ischemia.
Exercise-induced asthma
Chronic Bronchitis
Sickle Cell Anemia
Occupational asthma
2. blood flow reestablished to quickly
Distributive shock
Anaphylactic shock
Reprofusion injury
spinal cord injury
3. •Life-threatening complication of many conditions
• Results from an inappropriate immune response
• Widespread coagulation followed by massive bleeding because of the depletion of clotting factors
• Complications: shock and multisystem organ failure
Disseminated intravascular coagulation - DIC
Chronic Bronchitis Manifestations and Treatment
Disseminated Intravascular Coagulation DIC
Traumatic Brain Injury Complications
4. thyroid gland - T3, T4, calcitonin
parathyroid gland - parathyroid hormone
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, during acute respiratory failure
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stress incontinence
secondary brain injury
Glands that regulate calcium
5. altered level of consciousness
BP drops
HR goes up
hypotensive
Glands that regulate calcium
spinal cord injury
shock sighs and symptoms
Reprofusion injury
6. chest pain that occurs while a person is at rest and not exerting
himself does not get better
hemorrhagic stroke
Bone electrolytes
sickle cell crisis
unstable angina
7. Abnormally low white blood cell count
• Normal range = 5,000 to 10,000 cells/mL3 blood
beta cells
Leukopenia
Pneumonia
CT scan
8. damage to the alveoli
air can get in, but not out
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emphysema lecture
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rasmussen-univer
NUR 2063 Essentials of Pathophysiology
(NUR2063) / Pathophysiology Final Rasmussen
University Winter 2022
1. •Neither recessive nor dominant—co-dominant.
• Hemoglobin S causes erythrocytes to be abnormally shaped.
• Abnormal erythrocytes carry less oxygen and clog vessels, causing hypoxia and tissue ischemia.
Exercise-induced asthma
Chronic Bronchitis
Sickle Cell Anemia
Occupational asthma
2. blood flow reestablished to quickly
Distributive shock
Anaphylactic shock
Reprofusion injury
spinal cord injury
3. •Life-threatening complication of many conditions
• Results from an inappropriate immune response
• Widespread coagulation followed by massive bleeding because of the depletion of clotting factors
• Complications: shock and multisystem organ failure
Disseminated intravascular coagulation - DIC
Chronic Bronchitis Manifestations and Treatment
Disseminated Intravascular Coagulation DIC
Traumatic Brain Injury Complications
4. thyroid gland - T3, T4, calcitonin
parathyroid gland - parathyroid hormone
1 of 52 3/14/2021, 4:58
Downloaded by: Richarestqueen |
Distribution of this document is illegal
, during acute respiratory failure
2 of 52 3/14/2021, 4:58
Downloaded by: Richarestqueen |
Distribution of this document is illegal
, Stuvia.com - The Marketplace to Buy and Sell your Study
Material
stress incontinence
secondary brain injury
Glands that regulate calcium
5. altered level of consciousness
BP drops
HR goes up
hypotensive
Glands that regulate calcium
spinal cord injury
shock sighs and symptoms
Reprofusion injury
6. chest pain that occurs while a person is at rest and not exerting
himself does not get better
hemorrhagic stroke
Bone electrolytes
sickle cell crisis
unstable angina
7. Abnormally low white blood cell count
• Normal range = 5,000 to 10,000 cells/mL3 blood
beta cells
Leukopenia
Pneumonia
CT scan
8. damage to the alveoli
air can get in, but not out
3 of 52 3/14/2021, 4:58
Downloaded by: Richarestqueen |
Distribution of this document is illegal
, Stuvia.com - The Marketplace to Buy and Sell your Study
Material
emphysema lecture
4 of 52 3/14/2021, 4:58
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