DO NOT delegate what you can EAT!
Evaluate
Assess
Teach
Don’t delegate Unstable patients
Initial Assessment, Teaching, IV drips, Evaluations only RN
AIRBORNE TRANSMISSION-BASED PRECAUTIONS: MTV
Measl
esTB
Varicella-Chicken Pox/Herpes Zoster-Shingles
Private Room: Negative pressure with 6-12 air
exchanges/hrMask: N95 for TB
DROPLET TRANSMISSION-BASED PRECAUTIONS: Think of SPIDERMAN!
Sepsis
Scarlet
fever
Streptococcal Pharyngitis (Streptococcus group A/ Strep Throat): Can Lead to
Glomerulonephritis & Rheumatic
Parvovirus B19 Fever.
Pneumon
ia
Pertussis
Influenza/ Haemophilus influenza type B
Diphtheria (Pharyngeal): Serious bacterial
infection. Epiglottitis: Medial Emergency! No
Throat Inspection.Rubella/ German measles
Mumps
Meningitis/ Neisseria
Meningitidis Mycoplasma/
Meningeal Pneumonia An -
Adenovirus
Private Room or Cohort Surgical mask PRN for
Procedures Mask 3ft Distance
CONTACT PRECAUTION TRANSMISSION-BASED PRECAUTIONS: MRS.WEE
1
Multidrug resistant organism/ MRSA/ VRE
Respiratory
infectionSkin
,infections Wound
infection
Enteric infection - Clostridium Difficile
Eye infection – Conjunctivitis
*MRSA - Contact precaution ONLY. Use Chlorhexidine Wipe!
*VRSA - Contact & Airborne precaution (Private room, door closed, negative pressure)
*SARS (Severe Acute Resp Syndrome) Airborne & Contact (just like Varicella)
SKIN INFECTIONS- VCHIPS- CONTACT
Varicella Zoster
Cutaneous Diphtheria (Bacteria Infection in the Wound)
Herpes Simplex
Impetigo (Bacterial Skin Infection)
Pediculosis (Lice)
2
,Scabies (Itchy Skin condition. Burrowing Trail of the Scabies Mite)
Middle East Respiratory Syndrome (MERS): Viral respiratory illness caused by Coronavirus
(MERS-CoV).S/S: Fever, Cough, SOB, and Death. The Incubation Period is 5-6 days but can range
from 2-14 days.
CDC: Standard (Gloves), Contact (Gown), Eye Protection (Goggles), Airborne Precautions (N95)
Negative room: Negative disease (TB, Disseminated Herpes Zoster)
Positive room: Protect the Patient (HIV, Cancer)
Addison’s= hyponatremia, hypotension, decreased blood vol, hypoglycemia, hyperKalemia,
HyperCalcemia.
Cushing’s= HyperNatremia, HyperTension, Incr. Blood Vol, HyperGlycemia, hypokalemia,
hypocalcemia.
Managing Stress in a patient with Adrenal Insufficiency (Addison’s) is paramount,
because if the Adrenal glands are stressed further it could result in Addisonian Crisis.
Addison’s: Remember BP is the most Important assessment parameter, as it causes Severe
Hypotension. Addison’s: (need to "add" hormone): Hypoglycemia, Dark pigmentation, Decr.
Resistance to Stress, fractures,Alopecia, Weight Loss, GI distress. Vitiligo. Mood swings (Normal)
Need to Report S/S of Infection/ Fever
(Addisonian Crisis) Tx: Mineral Corticoids.
Addisonian Crisis: Hypoglycemia, Confusion, n/v, Abd Pain, Extreme Weakness, Dehydration,
Decr. BP.
Cushings: (have extra "Cushion" of Hormones): Hyperglycemia, prone to Infection,
Muscle Wasting,Weakness, Edema, HTN, Hirsutism, Moonfaced/Buffalo Hump
Cause: Excessive production of Corticotropin (Hyperplasia of the Adrenal Cortex) &
Cortisol-secretingAdrenal Tumor.
Prednisone Toxicity: Cushing’s syndrome- Buffalo Hump, Moon face, Hyperglycemia,
Hypertension.
Acetaminophen: 10-20. Max 4000mg per day.
Acetaminophen Poisoning: Possible Liver Failure for about 4 days. Close
observation required. Tx: (Antidote) n-AcetylCysteine/Mucomyst
3
, AcetylSalicyclic Acid (ASA): Metabolic Acidosis.
S/S: Tinnitus, Coffee Ground Emesis (Old Blood), Black tarry stools (Melena), Bruising,
Tachycardia,Hypotension, GI Ulcers.
Tx: Activated Charcoal, then IV Na+ Carbonate.
Acromegaly: Coarse Facial feature. Assess Cardiac Problems (eg. S3, S4).
Acute Respiratory Distress Syndrome (ARDS):
The 1st Sign is Incr. Respirations. Later comes Dyspnea, Retractions, Air Hunger,
Cyanosis.Cardinal sign is Hypoxemia (Low O2 level in tissues).
Refractory Hypoxemia is the hallmark of ARDS, a progressive form of acute respiratory failure
that has a highMortality rate. It can develop following a Pulmonary Insult (eg, aspiration,
pneumonia, toxic inhalation) or nonpulmonary insult (eg, sepsis, multiple blood transfusions,
trauma) to the Lung.
The Inability to improve Oxygenation With Incr. in O2 concentration.
The insult triggers a Massive Inflammatory response that causes the lung tissue to release
inflammatory mediators (leukotrienes, proteases) that cause damage to the alveolar-capillary
(A-C) membrane. As a result of
4