Case
Study: Mrs.
J. Brooke
Lopez
NRS 410V - 0501 Pathophysiology
and Nursing Management of
Clients’ Health ALL ANSWERS
100% CORRECT SPRING FALL-
2021/2022 GUARANTEED GRADE
A+
, CASE STUDY: MRS. J. 2
Clinical Manifestations of Mrs. J.
Upon admission, Mrs. J is presenting with respiratory complications as evidence by
stating feeling anxious, exhausted, and that she feels like she cannot get enough air. This is most
likely due to a collection of fluid in the lungs causing the patient to become hypoxic related to
her extensive history of tobacco use, hypertension, chronic heart failure (CHF), and chronic
obstructive pulmonary disease (COPD). Respiratory distress is confirmed on collection of
objective data such as an elevated respiratory rate at 34, pulse oximetry of 82%, faint point of
maximal impulse (PMI), and crackles in the lungs. At this time, it’s vital that Mrs. J maintain a
patent airway and adequate oxygenation. The purpose of this paper is to determine and meet the
needs for Mrs. J and provide the education required in order to prevent future COPD
exacerbations, acute decompensated heart failure, and hospital re-admissions.
Nursing interventions Upon Admission
Nursing interventions for Mrs. J should begin with a thorough nursing assessment by
collecting objective and subjective data. Mrs. J should be placed in high fowlers position with
two liters of supplemental oxygen to increase oxygenation and decrease the workload of the
heart. Interventions noted at the time of admission also seem appropriate for Mrs. J.
Intravenous furosemide (Lasix) is a diuretic used to offload fluid accumulation in the body to
reduce the workload of the heart. Daily weights, strict intake and output measurements, fluid
restrictions, and assessment of peripheral edema are crucial elements associated with
hypervolemia. Enalapril (Vasotec) is an antihypertensive, angiotensin converting enzyme (ACE)
inhibitor which helps to control blood pressure resulting in more effective pumping of the heart.
Metoprolol (Lopressor) is a selective beta1-adenoreceptor blocking agent. It reduces the force of
contractions on the myocardium which decreases the heart’s need for more oxygen.