NUR 2513
PediatricAssessment PDA RSV.
Pediatric Assessment PDA with RSV
xxx
Rasmussen University
NUR2513: Maternal-Child Nursing
Professor xxx
August 22, 2021
This study source was downloaded by 100000810282105 from CourseHero.com on 11-22-2022 08:31:54 GMT -06:00
https://www.coursehero.com/file/104768387/PediatricAssessment-PDA-RSVdocx/
, NUR 2513
PediatricAssessment PDA RSV.
Pediatric Assessment PDA with RSV
The pathophysiology of bronchiolitis begins with an acute infection of the epithelial cells lining
the small airways within the lungs. These infections result in edema, increased mucus production, and
eventually necrosis and regeneration of the cells (Wagner, 2009). Bronchiolitis presents with rhinitis,
cough, tachypnea, use of accessory muscles, hypoxia, and variable wheezing and crackles upon
auscultation. The most identified infectious agent is respiratory syncytial virus (RSV), though others
include adenovirus, human metapneumovirus, influenza virus, and parainfluenza virus (Wagner, 2009).
Diagnosis may include viral testing, with collection of a mucus sample for the virus causing bronchiolitis.
A chest X-ray may be done to assess for signs of pneumonia, blood tests for an increase in white blood
cells, and determining the level of oxygen in the bloodstream (Bronchiolitis, 2020)
The ductus arteriosus is a normal connection between the pulmonary artery and aorta and is
necessary for fetal circulation. At birth, the rise in PaO2 and decrease in prostaglandin concentration
cause the closure of the ductus arteriosus normally in the first 10 to 15 hours of life (Merck Manual,
2020). When this does not occur, the ductus arteriosus will remain patent. Infants with a small PDA are
typically asymptomatic, but those with a large PDA will have signs of heart failure, failure to thrive, poor
feeding, tachypnea, dyspnea with feeding, and tachycardia (Merck Manual, 2020). This is significant
with this patient because this Vivi was born at 36 weeks; and premature infants with PDA may have
respiratory distress, apnea, worse mechanical ventilation requirements, and other serious
complications (Merck Manual, 2020).
Vivi is at risk of developing bronchiolitis because she has a positive RSV result. She has a history
of PDA which can impact her cardiac and respiratory system putting her at increased risk for further
complications. She was born prematurely and is very small for her age. Although Vivi doesn’t attend
daycare, her siblings do, and they will also be exposing Vivi to other viruses and germs.
This study source was downloaded by 100000810282105 from CourseHero.com on 11-22-2022 08:31:54 GMT -06:00
https://www.coursehero.com/file/104768387/PediatricAssessment-PDA-RSVdocx/
PediatricAssessment PDA RSV.
Pediatric Assessment PDA with RSV
xxx
Rasmussen University
NUR2513: Maternal-Child Nursing
Professor xxx
August 22, 2021
This study source was downloaded by 100000810282105 from CourseHero.com on 11-22-2022 08:31:54 GMT -06:00
https://www.coursehero.com/file/104768387/PediatricAssessment-PDA-RSVdocx/
, NUR 2513
PediatricAssessment PDA RSV.
Pediatric Assessment PDA with RSV
The pathophysiology of bronchiolitis begins with an acute infection of the epithelial cells lining
the small airways within the lungs. These infections result in edema, increased mucus production, and
eventually necrosis and regeneration of the cells (Wagner, 2009). Bronchiolitis presents with rhinitis,
cough, tachypnea, use of accessory muscles, hypoxia, and variable wheezing and crackles upon
auscultation. The most identified infectious agent is respiratory syncytial virus (RSV), though others
include adenovirus, human metapneumovirus, influenza virus, and parainfluenza virus (Wagner, 2009).
Diagnosis may include viral testing, with collection of a mucus sample for the virus causing bronchiolitis.
A chest X-ray may be done to assess for signs of pneumonia, blood tests for an increase in white blood
cells, and determining the level of oxygen in the bloodstream (Bronchiolitis, 2020)
The ductus arteriosus is a normal connection between the pulmonary artery and aorta and is
necessary for fetal circulation. At birth, the rise in PaO2 and decrease in prostaglandin concentration
cause the closure of the ductus arteriosus normally in the first 10 to 15 hours of life (Merck Manual,
2020). When this does not occur, the ductus arteriosus will remain patent. Infants with a small PDA are
typically asymptomatic, but those with a large PDA will have signs of heart failure, failure to thrive, poor
feeding, tachypnea, dyspnea with feeding, and tachycardia (Merck Manual, 2020). This is significant
with this patient because this Vivi was born at 36 weeks; and premature infants with PDA may have
respiratory distress, apnea, worse mechanical ventilation requirements, and other serious
complications (Merck Manual, 2020).
Vivi is at risk of developing bronchiolitis because she has a positive RSV result. She has a history
of PDA which can impact her cardiac and respiratory system putting her at increased risk for further
complications. She was born prematurely and is very small for her age. Although Vivi doesn’t attend
daycare, her siblings do, and they will also be exposing Vivi to other viruses and germs.
This study source was downloaded by 100000810282105 from CourseHero.com on 11-22-2022 08:31:54 GMT -06:00
https://www.coursehero.com/file/104768387/PediatricAssessment-PDA-RSVdocx/
