Top NBME Concepts - Cardiology (USMLE
Step 1)
Rahul Damania, MD
Hi Guru is a webinar series tackling all of the organ systems in
the next few weeks. Hi Guru aims to really put in evidence-
based strategies for us MLA preparation. I'm so excited that
you all are here this session is going to be about an hour and
15 minutes. Just to let you know, and I hope you can tune in
until the end. I want to give you the fundamentals on how you
can make a damn good study schedule and think like the test
maker. At the end of the webinar I will answer live Q&A with
you guys, but please first just pay attention, make sure you're
taking notes, make sure that you are active and engaged in
typing in answers to the chat box. We're going to talk a little
bit about thermoregulation- what is our body's response to cold
as well as heat. We're going to do a brief review on S3 and S4
talk about various types of murmurs, what makes them louder,
and what is the different murmurs that you hear. Next, we are
going to be talking about vasculitis specifically- what shows up
are the hemoptysis and hematuria syndromes. Then we'll end
with some post-MI complications.
Pharyngeal pouches are ectodermally derived and exam exam
makers want you to know that that is going to be ectodermal
and neural crest derived. The first pouch gives rise to the
eustachian tube and the middle ear cavity. The third pouch is
the two pouch that are abnormal in dijord syndrome. This is an
integrative session, so remember that patients who have
digeorge syndrome are going to have recurrent infections in
your exam questions related to viral and fungal diseases.
Statins are hmg coa reductase inhibitors and they are going to
up regulate ldl receptors onto pathocytes. That in turn
decreases ldl levels with statins. Azidomide is going to be
inhibiting cholesterol absorption in the gut. Cholestyramine
specifically inhibits bile acid reuptake in the terminal ileum.
Nicotinamide (niacin) increases good cholesterol nicely, but
niacin has a bad side effect and that is it can cause you to
become hyperuricemic and exacerbate gout. Fibrates activate
the protein ppar alpha, which is a gene transcription factor,
and that 's going to actually increase fat metabolism and
fibrates will reduce triacylglyceride levels but, like statins, they
Step 1)
Rahul Damania, MD
Hi Guru is a webinar series tackling all of the organ systems in
the next few weeks. Hi Guru aims to really put in evidence-
based strategies for us MLA preparation. I'm so excited that
you all are here this session is going to be about an hour and
15 minutes. Just to let you know, and I hope you can tune in
until the end. I want to give you the fundamentals on how you
can make a damn good study schedule and think like the test
maker. At the end of the webinar I will answer live Q&A with
you guys, but please first just pay attention, make sure you're
taking notes, make sure that you are active and engaged in
typing in answers to the chat box. We're going to talk a little
bit about thermoregulation- what is our body's response to cold
as well as heat. We're going to do a brief review on S3 and S4
talk about various types of murmurs, what makes them louder,
and what is the different murmurs that you hear. Next, we are
going to be talking about vasculitis specifically- what shows up
are the hemoptysis and hematuria syndromes. Then we'll end
with some post-MI complications.
Pharyngeal pouches are ectodermally derived and exam exam
makers want you to know that that is going to be ectodermal
and neural crest derived. The first pouch gives rise to the
eustachian tube and the middle ear cavity. The third pouch is
the two pouch that are abnormal in dijord syndrome. This is an
integrative session, so remember that patients who have
digeorge syndrome are going to have recurrent infections in
your exam questions related to viral and fungal diseases.
Statins are hmg coa reductase inhibitors and they are going to
up regulate ldl receptors onto pathocytes. That in turn
decreases ldl levels with statins. Azidomide is going to be
inhibiting cholesterol absorption in the gut. Cholestyramine
specifically inhibits bile acid reuptake in the terminal ileum.
Nicotinamide (niacin) increases good cholesterol nicely, but
niacin has a bad side effect and that is it can cause you to
become hyperuricemic and exacerbate gout. Fibrates activate
the protein ppar alpha, which is a gene transcription factor,
and that 's going to actually increase fat metabolism and
fibrates will reduce triacylglyceride levels but, like statins, they
