Histology
Rapid Revision KROK
Minna Mamik
@ Medic Mukul
, induced immune
Drug
hemolytic anomia
flare
(Cytexicsensitivityi
wheal a
a
S
Hypersensitivity -> Y
dypes
response
I
Type I
->
Anaphylatic, Immediate, Reagine
a
Adopy hemolytic disease in newban
by Rh-incompatibility
Cytotoxic) Type II -
Antibody Mediated, Complement
dependent
Immuno Complex
Type III ->
Hypersensitivity Thymus ->
Produce thymasin
Hasall's Capuscles present all).
Type E ->
Delayed a T-cell mediated
hypersensitively (degenerated
will shinkover deme
↳ & inducation
erythrema >Digeoge Syndrome -
thymosin not produced
>Undergo Age involution
of allengen with Absence Di-Geage Syndrome (Chromosome
Type I interaction
-
IgE, absorbed on -
22 defect)
(is-30 min)
mastocytes and basophils membeanes
↳
e.g bronchial astma, after
acine introduction
Red Bone Marrow ->
Produce RBC
↑
Type II
- Fixadion of alien
antigens fixed on the cells
Ezydheopoeitin
N
actson Redbone.
(B -
cell by prefamed andibodies with the subsequent kidney produce
mediated)
activation of complement
system by the cell-bound Aputation -> Removal of agans
Immune complexes ->
e.g bloodteansfusion, Rh phantom pain Amputadion
Type III -> Formation of cell-bound immune complexes causalgic pain -> Gun shot
fixing ondo tissues &
producing their
damage protopathic pain -> Glossitis, inflammation
->
e.g Systemic lupus eythomedus, Glomoulonephitis
(SLE)
Cellular Ganuloma of
Type
immune Combination bact &
-> response, Delayedin maccophage
-
T-cell=> > CM1
Granuloma
type It
hypes.
->
e.g
Lapzosy "/Sandwich
palisade Azragment
->
Mantcuxtes fatB
-
->
↳ Geanuloma cell's
(at
Poikilocytosis -
Change shape of RBC Tuberculosis -
Langerhans all skinbarrie)
dencritic alls
Anisocytosis ->
change Size of RBC
lepzosy - Vizchow
call/cigarete-like cells (LoVe)
Rhinoschecoma -
Mikuliz cell
Gumma Bodies
Immune
agains
-> Central &
Peripheral Syphilis
-
↓ *
(hymosin) thymus Tonsils -
B-lymp
(RBC&WBC) Red Bane Marrow Lymph noce
splee langs -
3
types of Cell
Antigen Indipendent) Antigen dependent) pneumocytes/ Prumocytes/ pneumocytes/
Alvedlan cell Alseolar Cell Alvedan Maczophage
Tonsils -
Only lymphoid organ
with epithelial lining Type I Type I
single
~ Nasopharynged ↳
Tonsils; ↳ Simple difussion produce Surfactant
surface
engulffonign body
Tonsil -numerous
cryptspresent (Gas exchange) to reduce
↳
ETabralin
Tubal Acts
Tension as
phagocytosis
Waldeyer's
Tonsil >
hymphocytes arranged act as precuse prent
lungs from
paired het / as
lymphatic nodules fa type II
collapsing Emphysema
↳ Prevent
tendency to ↳ macrophage comes
paladine
Tonsil Tonsil
recede
Ccollapse)
~gese
& breaks walls,
baggy lungs,
Hal/gastzomucin alvedi damaged
Lymph noce ->>Bean shaped >Follicles in Contex
nodles - proten,producus
Medullary cads present aka-lymphatic
parital/oxyntic cell
2
It
Trabecal > cards in medulla
Rods
(duilight vision) contains maximum
Spleen -> Cential
artery Spleen consist of parts2 Alveolar
dype II ~ Mitochondria
↳> white pulp-ecentrically
tiven-> Central rein
placed acteride
LoVe ↳ Red pulp-made of
sinusoids & cads
Minna Mamik
, o Surfactant
Composition; o
Eyelens from Surface Ectodum * Redina from Estodem
Dipalmitoylphosphatidycholine (Dppc) chaloid ->
fom Mesodem
Sphingolipids S ↳ have blood veins
phospholipids CECell
Gganelles
lecithin Endoplasmic Redicutum
Ca2t SmoodhER
Rough ER
or
Sant Agranular
Glanclar
A
protein
W
B
Ribosomes(granules) pt.
Function
↓
Ribosome absent
Function:
Protein Synthesis lipid Synthesis
hydrophillic
-
hydrophobic Hydrophilic staage of Cant
#ate
water Detofication of Daugs
BK Be careful
Golgi Apparatus
St
Capillaries 1) post - translational modification
Non-continuous/Sinusoidal 2)
Continuous Fenestzated
sarding proteins
↳ NO Gaps, Nopores ↳ small Gaps ↳ Wider Gaps ↳
having (is &tzans end
kidney Red Bone Marrow
Brain
e.g eg he e.g;
lungs
Sheletal M.
I Liver
Spleen
↓RBC
WBC
-Expating & famation function
Smooth M. absorption platelets
ect,
hysosomes -> Suicide Bag/Residual Bodies
St Articles--Resistance Vessel Function:Destruction of unwanted Subst, bact, etc
Veins- Capacitance ↳ (produce hydedytic enzymes)
act as
phagocytosis
Lymphatic Capillaries (NoBasement or thin
-
Macula Densa ~membrane penoxisomes -> Destruction of te radicals by Catalase
↳ H202
Oxidation of of FA
long & very long
->
chain
St Ammonification. NHy is
dangerous in beain, heart. Cytoskeleton ->
Provides stength &steucture to cell
Muscles
bone of cell)
Brain, heart ↳ Microdubules -
Big filaments
Transport of NH, of NH3
Transport ↳ Intermediate filament -> in
most quanities present
by Glutamate
↓Livert by Alanine
↳ Microfilament -
Small filaments
lots of H20 NHs
↓
both
used fa Ureacycle -> in
cytoplasm
&
excretion of ammonia ↓ mitochondria 6 Epidhelium
wied
↓ simple epith. ->
Single layered
Albumin
most in
kidney Compound epith ->
Multilayered
Blood lange
~In quanidy
et
Collagen -> MostAbundant in
protein body .
Simple epithstium
Bone, Skin, Tendon epith
Type I Simple Squamos 3B- A (mnemonic
-> ->
if absent Osteogenesis imperfecta Bowman's capsule
-
, -
Blood vessels
Cardilage
-
Type II -
>Body Cavity
if absent Achondroplasia
>
> -> Alvedi
Type III - Redicular fibers, Blood Vessels, Aata -Simple Cuboidal epith. -> T-ok
Like Cube II
Ellen Danlos
Syndrome -
Thyzoid Follides
I -> Basement
Membrane ·7777, Germinal Epith. of Ovary
Type
-AlportSynchrome -
kidney Tubules
Minna Mamik poT & DCT (
proximal & Distal
convoluted Tubules
Rapid Revision KROK
Minna Mamik
@ Medic Mukul
, induced immune
Drug
hemolytic anomia
flare
(Cytexicsensitivityi
wheal a
a
S
Hypersensitivity -> Y
dypes
response
I
Type I
->
Anaphylatic, Immediate, Reagine
a
Adopy hemolytic disease in newban
by Rh-incompatibility
Cytotoxic) Type II -
Antibody Mediated, Complement
dependent
Immuno Complex
Type III ->
Hypersensitivity Thymus ->
Produce thymasin
Hasall's Capuscles present all).
Type E ->
Delayed a T-cell mediated
hypersensitively (degenerated
will shinkover deme
↳ & inducation
erythrema >Digeoge Syndrome -
thymosin not produced
>Undergo Age involution
of allengen with Absence Di-Geage Syndrome (Chromosome
Type I interaction
-
IgE, absorbed on -
22 defect)
(is-30 min)
mastocytes and basophils membeanes
↳
e.g bronchial astma, after
acine introduction
Red Bone Marrow ->
Produce RBC
↑
Type II
- Fixadion of alien
antigens fixed on the cells
Ezydheopoeitin
N
actson Redbone.
(B -
cell by prefamed andibodies with the subsequent kidney produce
mediated)
activation of complement
system by the cell-bound Aputation -> Removal of agans
Immune complexes ->
e.g bloodteansfusion, Rh phantom pain Amputadion
Type III -> Formation of cell-bound immune complexes causalgic pain -> Gun shot
fixing ondo tissues &
producing their
damage protopathic pain -> Glossitis, inflammation
->
e.g Systemic lupus eythomedus, Glomoulonephitis
(SLE)
Cellular Ganuloma of
Type
immune Combination bact &
-> response, Delayedin maccophage
-
T-cell=> > CM1
Granuloma
type It
hypes.
->
e.g
Lapzosy "/Sandwich
palisade Azragment
->
Mantcuxtes fatB
-
->
↳ Geanuloma cell's
(at
Poikilocytosis -
Change shape of RBC Tuberculosis -
Langerhans all skinbarrie)
dencritic alls
Anisocytosis ->
change Size of RBC
lepzosy - Vizchow
call/cigarete-like cells (LoVe)
Rhinoschecoma -
Mikuliz cell
Gumma Bodies
Immune
agains
-> Central &
Peripheral Syphilis
-
↓ *
(hymosin) thymus Tonsils -
B-lymp
(RBC&WBC) Red Bane Marrow Lymph noce
splee langs -
3
types of Cell
Antigen Indipendent) Antigen dependent) pneumocytes/ Prumocytes/ pneumocytes/
Alvedlan cell Alseolar Cell Alvedan Maczophage
Tonsils -
Only lymphoid organ
with epithelial lining Type I Type I
single
~ Nasopharynged ↳
Tonsils; ↳ Simple difussion produce Surfactant
surface
engulffonign body
Tonsil -numerous
cryptspresent (Gas exchange) to reduce
↳
ETabralin
Tubal Acts
Tension as
phagocytosis
Waldeyer's
Tonsil >
hymphocytes arranged act as precuse prent
lungs from
paired het / as
lymphatic nodules fa type II
collapsing Emphysema
↳ Prevent
tendency to ↳ macrophage comes
paladine
Tonsil Tonsil
recede
Ccollapse)
~gese
& breaks walls,
baggy lungs,
Hal/gastzomucin alvedi damaged
Lymph noce ->>Bean shaped >Follicles in Contex
nodles - proten,producus
Medullary cads present aka-lymphatic
parital/oxyntic cell
2
It
Trabecal > cards in medulla
Rods
(duilight vision) contains maximum
Spleen -> Cential
artery Spleen consist of parts2 Alveolar
dype II ~ Mitochondria
↳> white pulp-ecentrically
tiven-> Central rein
placed acteride
LoVe ↳ Red pulp-made of
sinusoids & cads
Minna Mamik
, o Surfactant
Composition; o
Eyelens from Surface Ectodum * Redina from Estodem
Dipalmitoylphosphatidycholine (Dppc) chaloid ->
fom Mesodem
Sphingolipids S ↳ have blood veins
phospholipids CECell
Gganelles
lecithin Endoplasmic Redicutum
Ca2t SmoodhER
Rough ER
or
Sant Agranular
Glanclar
A
protein
W
B
Ribosomes(granules) pt.
Function
↓
Ribosome absent
Function:
Protein Synthesis lipid Synthesis
hydrophillic
-
hydrophobic Hydrophilic staage of Cant
#ate
water Detofication of Daugs
BK Be careful
Golgi Apparatus
St
Capillaries 1) post - translational modification
Non-continuous/Sinusoidal 2)
Continuous Fenestzated
sarding proteins
↳ NO Gaps, Nopores ↳ small Gaps ↳ Wider Gaps ↳
having (is &tzans end
kidney Red Bone Marrow
Brain
e.g eg he e.g;
lungs
Sheletal M.
I Liver
Spleen
↓RBC
WBC
-Expating & famation function
Smooth M. absorption platelets
ect,
hysosomes -> Suicide Bag/Residual Bodies
St Articles--Resistance Vessel Function:Destruction of unwanted Subst, bact, etc
Veins- Capacitance ↳ (produce hydedytic enzymes)
act as
phagocytosis
Lymphatic Capillaries (NoBasement or thin
-
Macula Densa ~membrane penoxisomes -> Destruction of te radicals by Catalase
↳ H202
Oxidation of of FA
long & very long
->
chain
St Ammonification. NHy is
dangerous in beain, heart. Cytoskeleton ->
Provides stength &steucture to cell
Muscles
bone of cell)
Brain, heart ↳ Microdubules -
Big filaments
Transport of NH, of NH3
Transport ↳ Intermediate filament -> in
most quanities present
by Glutamate
↓Livert by Alanine
↳ Microfilament -
Small filaments
lots of H20 NHs
↓
both
used fa Ureacycle -> in
cytoplasm
&
excretion of ammonia ↓ mitochondria 6 Epidhelium
wied
↓ simple epith. ->
Single layered
Albumin
most in
kidney Compound epith ->
Multilayered
Blood lange
~In quanidy
et
Collagen -> MostAbundant in
protein body .
Simple epithstium
Bone, Skin, Tendon epith
Type I Simple Squamos 3B- A (mnemonic
-> ->
if absent Osteogenesis imperfecta Bowman's capsule
-
, -
Blood vessels
Cardilage
-
Type II -
>Body Cavity
if absent Achondroplasia
>
> -> Alvedi
Type III - Redicular fibers, Blood Vessels, Aata -Simple Cuboidal epith. -> T-ok
Like Cube II
Ellen Danlos
Syndrome -
Thyzoid Follides
I -> Basement
Membrane ·7777, Germinal Epith. of Ovary
Type
-AlportSynchrome -
kidney Tubules
Minna Mamik poT & DCT (
proximal & Distal
convoluted Tubules
