1. Digoxin (Lanoxin) - positive inotrope (increases force of
contraction); negative chronotrope (decreases heart rate).
How do you assess for this?: - Always take apical pulse for one
full minute
2. Client with a long hx of daily digoxin and fourosemide (Lasix)
use; creates a high risk for dig toxicity because: Lasix can
cause hypokalemia, which can lead to dig toxicity
3. What can happen when Digoxin is taken with Dronedarone
(Multaq), which is another anti-dysrhythmic drug)?: Digoxin
can increase in the blood level and further increase the effects
4. What is the normal digoxin level?: 0.5-2 ng/mL
5. Normal serum potassium level is: 3.5-5.0 mEq/L
6. Low potassium or magnesium levels may increase risk for:
Digoxin toxicity
7. What are the signs and symptoms for digoxin toxcitiy?:
anorexia, brady- cardia, headache, dizziness, confusion, nausea, and
visual disturbances such as blurred, yellow, or halo vision.
8. When should you hold off on giving digoxin drug therapy?:
if apical pulse is less than 60
9. What effects do Calcium channel blockers (-dipine;
amlodipine (Norvasc), nifedipine (Procardia) cause: produce
vasodialation and reflex tachycardia (lowers BP but increases HR)
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, 10.Verapamil and diltiazam produce: vasodialation and
cardiosuppresssion (lowers BP and Lowers heart rate)
11.Beta blockers have the drug ending: -OLOL such as
atenolol, propanolol, esmolol, ect.)
12.Beta 1 blocks receptors in the: heart
13.Beta 2 blocks receptors in the: lungs
14.Beta blockers can be non selective so be aware of any:
respiratory condi- tions such as asthma, emphysema COPD, ect.
15.A nurse should always check and before giving a
beta blocker: AP and BP
do not give if HR is
below 60 never stop
abruptly
16.Why do you never stop taking a Beta BLocker abruptly:
Must taper be- cause angina or MI can orccur
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