Nephrology + toxicology 2023
Anki:
https://docs.google.com/document/d/e/2PACX-1vSz5QhLaVIOm0zRgR
zPEqKsNoGz8ZDEqlsyemf7sIHz1JDMCbr4xWq6zkWNJ_d_NiKlC00RO
oJWHaDt/pub
(double-check)
● Acute glomerulonephritis is: #1 (colloqu) #14 100% #10
● А. Bilateral kidney disease
● B. Unilateral kidney disease
● C. Degenerative kidney disease
● D. Large vessel vasculitis
● E. Cystic Kidney Disease
○ А. Bilateral kidney disease
● Acute Glomerulonephritis Affects:#1 (colloqu) 14# 100% #10
● А. The tubular part of the nephron
● B. The Glomerulus
● C. The tubules in one of the kidneys
● D. The collective tubules
● E. Only the upper parts of the kidneys
○ B. The Glomerulus
● Acute Glomerulonephritis Is:#1 (colloqu) #14 100% #10
● А. Immune complex disease
● B. Secondary Autoimmune Glomerulonephritis
● C. Toxic alteration of the kidney
● D. Granulomatous inflammation
● E. Autoimmune disease
○ А. Immune complex disease
● Pathogenesis of acute glomerulonephritis includes: #1 (colloqu) 100% #10
● А. Alternative Complement pathway activation
● B. Participation of fibrinolytic system
● C. Kallikrein kinin system activation
● D. Angiotensin II blockage
● E. Classic complement pathway activation
○ А. Alternative Complement pathway activation
● In Acute Glomerulonephritis Is Pathogenesis Takes Part: #1 (colloqu) 100% #10
,● А. C3 complement fraction
● B. AntiGBM antibodies
● C. рАNСА
● D. сАNСА
● D. Cryoglobulins
○ А. C3 complement fraction
● Which bacteria has the mainline the etiology of acute glomerulonephritis? #1
(colloqu) 100% #10
● А. Group A beta-haemolytic streptococcus
● B. Klebsiella pneumoniae
● C. Staphylococcus epidermidis
● D. Morganella Morganii
● E. Acinetobacterbaumanii
○ А. Group A beta-haemolytic streptococcus
● In Acute Glomerulonephritis There Is Proliferation Of: #1 (colloqu) 100% #10
● А. Mesangial cells
● B. Podocytes
● C. The Henle’s loop cells
● D. Tubular cells
● E. Interstitial cells
○ А. Mesangial cells
● Subepithelial “humps” in acute glomerulonephritis are”: #1(colloqu) 100% #10
● А. Immune complex depositions
● B. Hemosiderin depositions
● C. Melanin depositions
● D. Uric acid depositions
● E. Cholesterol depositions
○ А. Immune complex depositions
● The Glomerular Capillary narrowing in acute glomerulonephritis due to: #1
(colloqu) 100% #10
● А. Crescent formation
● B. Mesangial Cell Proliferation And Swelling of the endothelial cells
● C. Destruction and fusion of the podocytes’ foot processes
● D. Interstitial Cells Proliferation
● E. Epithelial cells proliferatioN
○ B. Mesangial Cell Proliferation And Swelling of the endothelial cells
● The Main Symptoms Of Acute Glomerulonephritis Are: #1 (colloqu) 100% #10
● А. Caught and dyspnea
● B. Oedema and chest pain
,● C. Pruritus, oedema, joint pain
● D. Hematuria, hypertension, oedema
● E. Meteorism, constipation,hypertension
○ D. Hematuria, hypertension, oedema
● A percutaneous kidney biopsy is performed in patients with acute
glomerulonephritis in case of: #1 (colloqu) 100% #10
● A. Increasing of serum creatinine to exclude rapidly progressive glomerulonephritis
● B. Improvement in the clinical status
● C. Reduction of serum creatinine and persistence of hypertension
● D. Joints swelling and headache for excluding hypertensive nephropathy
● E. Increasing of serum glucose to diagnose diabetic nephropathy
○ A. Increasing of serum creatinine to exclude rapidly progressive
glomerulonephritis
● The name of typical dysmorphic Erythrocytes In Urine Of patients with
glomerulonephritis is: #1 (colloqu) 100% #10
● А. schistocytes
● B. Acanthocytes
● C. Мicrocytes
● D. Punctiform erythrocytes
● E. Маcrocytes
○ B. Acanthocytes
● The plasma volume in patients with acute glomerulonephritis is?#1 (colloqu) 100%
#10
● А. Reduced
● B. Normal
● C. Reduced in the beginning and increased after that
● D. Increased
● E. The plasma volume doesn’t take part in the pathogenesis of the nephritic syndrome
○ А. Reduced
● Rapidly progressive glomerulonephritis is: #1 (colloqu) 100% #10
● А. Acute disease with immune /autoimmune pathogenesis
● B. Acute infectious nephritis
● C. Acute drug-induced interstitial nephritis
● D. IgA glomerulonephritis
● E. C3 glomerulonephritis
○ А. Acute disease with immune /autoimmune pathogenesis
● The other names of rapidly progressive glomerulonephritis are: #1 (colloqu) 100%
#10
● А. Acute, С3 и IgМ glomerulonephritis
, ● B. Crescentic and subacute glomerulonephritis
● C. Hypertensive, diabetic and idiopathic nephrosclerosis
● D. Interstitial, gouty and toxic nephritis
● E.Multiple myeloma nephropathy, gouty and satellite nephropathy
○ B. Crescentic and subacute glomerulonephritis
● AntiGBM antibodies take part in pathogenesis of: #1(colloqu) 100% #10
● А. Balkan endemic nephropathy
● B. IgA glomerulonephritis
● C. IgM glomerulonephritis
● D. Acute pyelonephritis
● E. Rapidly progressive glomerulonephritis
○ E. Rapidly progressive glomerulonephritis
● Wegener's granulomatosis may lead to development of: #1 (colloqu) 100% #10
● А. Balkan endemic nephropathy
● B. IgA glomerulonephritis
● C. IgM glomerulonephritis
● D. Acute pyelonephritis
● E. Rapidly progressive glomerulonephritis
○ E. Rapidly progressive glomerulonephritis
● There is rapidly progressive glomerulonephritis with pulmonary-renal syndrome in
patients with: #1 (colloqu) 100% #10
● А. Gitelman‘s syndrome
● B. Barter‘s syndrome
● C. Goodpasture‘s syndrome
● D. Sjogren's syndrome
● E. Sicca syndrome
○ C. Goodpasture‘s syndrome
● Microscopic polyangiitis may lead to development of: #9 (colloqu) 100% #10
● a. Balkan endemic nephropathy
● b. lgA glomerulonephritis
● c. lgM glomerulonephritis
● d. Acute pyelonephritis
● e. Rapidly progressive glomerulonephritis
○ e. Rapidly progressive glomerulonephritis
● Rapidly progressive glomerulonephritis has been complicated with end stage
CKDfor: #1 (colloqu) 100% #10
● А. 5 years
● B. 10 years
● C. couple of weeks to months
Anki:
https://docs.google.com/document/d/e/2PACX-1vSz5QhLaVIOm0zRgR
zPEqKsNoGz8ZDEqlsyemf7sIHz1JDMCbr4xWq6zkWNJ_d_NiKlC00RO
oJWHaDt/pub
(double-check)
● Acute glomerulonephritis is: #1 (colloqu) #14 100% #10
● А. Bilateral kidney disease
● B. Unilateral kidney disease
● C. Degenerative kidney disease
● D. Large vessel vasculitis
● E. Cystic Kidney Disease
○ А. Bilateral kidney disease
● Acute Glomerulonephritis Affects:#1 (colloqu) 14# 100% #10
● А. The tubular part of the nephron
● B. The Glomerulus
● C. The tubules in one of the kidneys
● D. The collective tubules
● E. Only the upper parts of the kidneys
○ B. The Glomerulus
● Acute Glomerulonephritis Is:#1 (colloqu) #14 100% #10
● А. Immune complex disease
● B. Secondary Autoimmune Glomerulonephritis
● C. Toxic alteration of the kidney
● D. Granulomatous inflammation
● E. Autoimmune disease
○ А. Immune complex disease
● Pathogenesis of acute glomerulonephritis includes: #1 (colloqu) 100% #10
● А. Alternative Complement pathway activation
● B. Participation of fibrinolytic system
● C. Kallikrein kinin system activation
● D. Angiotensin II blockage
● E. Classic complement pathway activation
○ А. Alternative Complement pathway activation
● In Acute Glomerulonephritis Is Pathogenesis Takes Part: #1 (colloqu) 100% #10
,● А. C3 complement fraction
● B. AntiGBM antibodies
● C. рАNСА
● D. сАNСА
● D. Cryoglobulins
○ А. C3 complement fraction
● Which bacteria has the mainline the etiology of acute glomerulonephritis? #1
(colloqu) 100% #10
● А. Group A beta-haemolytic streptococcus
● B. Klebsiella pneumoniae
● C. Staphylococcus epidermidis
● D. Morganella Morganii
● E. Acinetobacterbaumanii
○ А. Group A beta-haemolytic streptococcus
● In Acute Glomerulonephritis There Is Proliferation Of: #1 (colloqu) 100% #10
● А. Mesangial cells
● B. Podocytes
● C. The Henle’s loop cells
● D. Tubular cells
● E. Interstitial cells
○ А. Mesangial cells
● Subepithelial “humps” in acute glomerulonephritis are”: #1(colloqu) 100% #10
● А. Immune complex depositions
● B. Hemosiderin depositions
● C. Melanin depositions
● D. Uric acid depositions
● E. Cholesterol depositions
○ А. Immune complex depositions
● The Glomerular Capillary narrowing in acute glomerulonephritis due to: #1
(colloqu) 100% #10
● А. Crescent formation
● B. Mesangial Cell Proliferation And Swelling of the endothelial cells
● C. Destruction and fusion of the podocytes’ foot processes
● D. Interstitial Cells Proliferation
● E. Epithelial cells proliferatioN
○ B. Mesangial Cell Proliferation And Swelling of the endothelial cells
● The Main Symptoms Of Acute Glomerulonephritis Are: #1 (colloqu) 100% #10
● А. Caught and dyspnea
● B. Oedema and chest pain
,● C. Pruritus, oedema, joint pain
● D. Hematuria, hypertension, oedema
● E. Meteorism, constipation,hypertension
○ D. Hematuria, hypertension, oedema
● A percutaneous kidney biopsy is performed in patients with acute
glomerulonephritis in case of: #1 (colloqu) 100% #10
● A. Increasing of serum creatinine to exclude rapidly progressive glomerulonephritis
● B. Improvement in the clinical status
● C. Reduction of serum creatinine and persistence of hypertension
● D. Joints swelling and headache for excluding hypertensive nephropathy
● E. Increasing of serum glucose to diagnose diabetic nephropathy
○ A. Increasing of serum creatinine to exclude rapidly progressive
glomerulonephritis
● The name of typical dysmorphic Erythrocytes In Urine Of patients with
glomerulonephritis is: #1 (colloqu) 100% #10
● А. schistocytes
● B. Acanthocytes
● C. Мicrocytes
● D. Punctiform erythrocytes
● E. Маcrocytes
○ B. Acanthocytes
● The plasma volume in patients with acute glomerulonephritis is?#1 (colloqu) 100%
#10
● А. Reduced
● B. Normal
● C. Reduced in the beginning and increased after that
● D. Increased
● E. The plasma volume doesn’t take part in the pathogenesis of the nephritic syndrome
○ А. Reduced
● Rapidly progressive glomerulonephritis is: #1 (colloqu) 100% #10
● А. Acute disease with immune /autoimmune pathogenesis
● B. Acute infectious nephritis
● C. Acute drug-induced interstitial nephritis
● D. IgA glomerulonephritis
● E. C3 glomerulonephritis
○ А. Acute disease with immune /autoimmune pathogenesis
● The other names of rapidly progressive glomerulonephritis are: #1 (colloqu) 100%
#10
● А. Acute, С3 и IgМ glomerulonephritis
, ● B. Crescentic and subacute glomerulonephritis
● C. Hypertensive, diabetic and idiopathic nephrosclerosis
● D. Interstitial, gouty and toxic nephritis
● E.Multiple myeloma nephropathy, gouty and satellite nephropathy
○ B. Crescentic and subacute glomerulonephritis
● AntiGBM antibodies take part in pathogenesis of: #1(colloqu) 100% #10
● А. Balkan endemic nephropathy
● B. IgA glomerulonephritis
● C. IgM glomerulonephritis
● D. Acute pyelonephritis
● E. Rapidly progressive glomerulonephritis
○ E. Rapidly progressive glomerulonephritis
● Wegener's granulomatosis may lead to development of: #1 (colloqu) 100% #10
● А. Balkan endemic nephropathy
● B. IgA glomerulonephritis
● C. IgM glomerulonephritis
● D. Acute pyelonephritis
● E. Rapidly progressive glomerulonephritis
○ E. Rapidly progressive glomerulonephritis
● There is rapidly progressive glomerulonephritis with pulmonary-renal syndrome in
patients with: #1 (colloqu) 100% #10
● А. Gitelman‘s syndrome
● B. Barter‘s syndrome
● C. Goodpasture‘s syndrome
● D. Sjogren's syndrome
● E. Sicca syndrome
○ C. Goodpasture‘s syndrome
● Microscopic polyangiitis may lead to development of: #9 (colloqu) 100% #10
● a. Balkan endemic nephropathy
● b. lgA glomerulonephritis
● c. lgM glomerulonephritis
● d. Acute pyelonephritis
● e. Rapidly progressive glomerulonephritis
○ e. Rapidly progressive glomerulonephritis
● Rapidly progressive glomerulonephritis has been complicated with end stage
CKDfor: #1 (colloqu) 100% #10
● А. 5 years
● B. 10 years
● C. couple of weeks to months
