Amyotrophic Lateral Sclerosis
Student’s Name:
Institutional Affiliation:
Course:
Professor’s Name:
Date:
, Amyotrophic Lateral Sclerosis
Introduction
Amyotrophic Lateral Sclerosis (ALS), is the neurodegenerative disease of the motor
neurons. The condition is also referred to as Lou Gehrig's disease. The inability to trace the
critical etiology of the disease, ALS presents multiple pathways that depict similar disease
existence. The variable paths are either sporadic or hereditary. ALS extends its effects to the
lower and upper neurons with different onset patterns where often the initial degeneration occurs
to the lower motor neuron usually located within proximal limbs. The disease is progressive;
thus, it ruins the neurons' normal functioning, leading to paralysis, bringing forth untimely and
inevitable death. Currently, there is no designed cure for the disease only through the
collaboration of multiple medications and other interventions to manage the symptoms to
prolong the individual's life. The life extension hack can result in a duration of 10 or even more
years. (Zarei et al., 2015)
Symptoms
Amyotrophic lateral sclerosis defines a distinctive feature that depicts the lower and
upper motor neuron clinical manifestations' coexistence. Under the lower motor neuron (LMN)
findings, the disease shows muscle atrophy and fasciculations. Muscle atrophy is the loss of
muscle mass. On the other hand, fasciculations are spontaneous involuntary muscle contraction
and relaxation of the delicate muscle fibers. The findings of the upper motor neuron (UMN)
entail hyperreflexia and stratification. Hyperreflexia indicates the existence of the upper motor
neuron lesion that depicts the absence of the inhibitory mechanism to modulate the motor
pathways. Therefore, the general body weakness about the aftermath of the disease on the upper
Student’s Name:
Institutional Affiliation:
Course:
Professor’s Name:
Date:
, Amyotrophic Lateral Sclerosis
Introduction
Amyotrophic Lateral Sclerosis (ALS), is the neurodegenerative disease of the motor
neurons. The condition is also referred to as Lou Gehrig's disease. The inability to trace the
critical etiology of the disease, ALS presents multiple pathways that depict similar disease
existence. The variable paths are either sporadic or hereditary. ALS extends its effects to the
lower and upper neurons with different onset patterns where often the initial degeneration occurs
to the lower motor neuron usually located within proximal limbs. The disease is progressive;
thus, it ruins the neurons' normal functioning, leading to paralysis, bringing forth untimely and
inevitable death. Currently, there is no designed cure for the disease only through the
collaboration of multiple medications and other interventions to manage the symptoms to
prolong the individual's life. The life extension hack can result in a duration of 10 or even more
years. (Zarei et al., 2015)
Symptoms
Amyotrophic lateral sclerosis defines a distinctive feature that depicts the lower and
upper motor neuron clinical manifestations' coexistence. Under the lower motor neuron (LMN)
findings, the disease shows muscle atrophy and fasciculations. Muscle atrophy is the loss of
muscle mass. On the other hand, fasciculations are spontaneous involuntary muscle contraction
and relaxation of the delicate muscle fibers. The findings of the upper motor neuron (UMN)
entail hyperreflexia and stratification. Hyperreflexia indicates the existence of the upper motor
neuron lesion that depicts the absence of the inhibitory mechanism to modulate the motor
pathways. Therefore, the general body weakness about the aftermath of the disease on the upper
