Material
NUR2063 Essentials of
Pathophysiology Pathophysiology Final
Rasmussen University
1. •Neither recessive nor dominant—co-dominant.
• Hemoglobin S causes erythrocytes to be abnormally shaped.
• Abnormal erythrocytes carry less oxygen and clog vessels, causing hypoxia and
tissue ischemia.
Exercise-induced asthma
Chronic Bronchitis
Sickle Cell Anemia
Occupational asthma
2. blood flow reestablished to quickly
Distributive shock
Anaphylactic shock
Reprofusion injury
spinal cord injury
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3. •Life-threatening complication of many conditions
• Results from an inappropriate immune response
• Widespread coagulation followed by massive bleeding because of the
depletion of clotting factors
• Complications: shock and multisystem organ failure
Disseminated intravascular coagulation - DIC
Chronic Bronchitis Manifestations and Treatment
Disseminated Intravascular Coagulation DIC
Traumatic Brain Injury Complications
4. thyroid gland - T3, T4,
calcitonin parathyroid gland
- parathyroid hormone
during acute respiratory failure
stress incontinence
secondary brain injury
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Glands that regulate calcium
5. altered level of
consciousness BP
drops
HR
goes up
hypote
nsive
Glands that regulate calcium
spinal cord injury
shock sighs and symptoms
Reprofusion injury
6. chest pain that occurs while a person is at rest
and not exerting himself does not get better
hemorrhagic stroke
Bone electrolytes
sickle cell crisis
unstable angina
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7. Abnormally low white blood cell count
• Normal range = 5,000 to 10,000 cells/mL3 blood
beta cells
Leukopenia
Pneumonia
CT scan
8. damage to the alveoli
air can get in, but not out
emphysema lecture
Normal CO2 levels
anemia symptoms
Stabilize the patient
9. Manifestations
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