NURS 663 COMPREHENSIVE QUESTIONS AND
VERIFIED ANSWERS EXAM 1\STUDY GUIDE 2024
UPDATE
What is ECT?
The use of electrical shock current delivered to the brain to induce a seizure that treats depression. Goal
is to reverse atrophy.
ECT is FDA approved for what
Bipolar disorder, schizophrenia, schizoaffective disorder, catatonia, neuroleptic malignant syndrome,
treatment resistant refractory major depressive disorder
What is ECT schedule
Typically Monday Wednesday Friday for 6 to 12 sessions
Bilateral electrode placement in ECT indications
Classic placement increases cognitive side effects but has better efficacy. Reserved for urgency such as
life-threatening depression profound distress and catatonia
ECT side effects and Risks 8
Cognitive side effects such as memory loss, head, neck, jaw pain, nausea, myalgia's procedure is low risk
What is vagus nerve stimulation
It's an implant in the chest that stimulates the left Vegas nerve by Paul stations and it's controlled by an
on off switch that is activated by a magnet
How does vagus nerve stimulation work
Stimulates the brain stem nuclei which changes serotonin in the limbic and cortical systems
Vagal nerve stimulation side effects-4
Voice altered, breathlessness, neck pain, no cognitive side effects
Vagus nerve stimulation is FDA approved for what in under investigation for what
FDA approved for epilepsy and under investigation for major depressive disorder and bipolar disorder
What is trans cranial magnetic stimulation
Placement of rapid alternating magnets on scalp cause impulse to structures and is focused
Goal of trans cranial magnetic stimulation
Firing of neurons will alter pathology
, Bipolar one disorder DSM five criteria
Manic episode: 1+ week of a colon elevated, expansive or irritable mood and increase energy. 3+
symptoms from B: distractibility, indiscretion, irresponsible, grandiosity, flight of ideas, activity(Increase
goal-orient), decreased need for sleep, talkativeness or pressured speech.
Bipolar two DSM five criteria
Hypo mania and major depressive disorder: hypo mania same as mania with decreased severity and
duration and no functional impairment for episode of four or more days and no psychosis
Mixed episodes (bipolar)
Manic and depressive symptoms time by side usually with comorbid substance abuse increased risk of
suicide and psychosis
rapid cycling
Four or more cycles per year no greater than a week well period
Cyclothymia DSM-V Criteria
Two or more years of mood cycling with dysthymia and hypo mania decreased intensity than bipolar
disorder meets criteria for hypo mania but does not meet criteria for major depressive disorder
Dysthymia DSM five criteria
HE'S 2 SAD depressive symptoms lasting two or more years that is subsydromal characterized by
hopelessness decreased energy, decrease self-esteem for two years, abnormal sleep, abnormal appetite
impaired decision-making.
MAOÍ Mechanism of action generally
Catalyzes the deamination of monoamines intracellularly and MAO transport Reuptake extracellular
monoamines
MAO-A Mechanism of action
MAO-A Oxidizes serotonin norepinephrine and epinephrine
MAO-B Mechanism of action
Oxidizes phenylalanine
MAO-A and MAO-B mechanism of action
Oxidizes dopamine non-preferentially
MAOs Neumonic -2
Date with Tyra banks with wine and cheese in Maui— can cause hypertensive crisis related to tyramine
from aged food.
MAWIs= my arms weight increased= effective for atypical depression
VERIFIED ANSWERS EXAM 1\STUDY GUIDE 2024
UPDATE
What is ECT?
The use of electrical shock current delivered to the brain to induce a seizure that treats depression. Goal
is to reverse atrophy.
ECT is FDA approved for what
Bipolar disorder, schizophrenia, schizoaffective disorder, catatonia, neuroleptic malignant syndrome,
treatment resistant refractory major depressive disorder
What is ECT schedule
Typically Monday Wednesday Friday for 6 to 12 sessions
Bilateral electrode placement in ECT indications
Classic placement increases cognitive side effects but has better efficacy. Reserved for urgency such as
life-threatening depression profound distress and catatonia
ECT side effects and Risks 8
Cognitive side effects such as memory loss, head, neck, jaw pain, nausea, myalgia's procedure is low risk
What is vagus nerve stimulation
It's an implant in the chest that stimulates the left Vegas nerve by Paul stations and it's controlled by an
on off switch that is activated by a magnet
How does vagus nerve stimulation work
Stimulates the brain stem nuclei which changes serotonin in the limbic and cortical systems
Vagal nerve stimulation side effects-4
Voice altered, breathlessness, neck pain, no cognitive side effects
Vagus nerve stimulation is FDA approved for what in under investigation for what
FDA approved for epilepsy and under investigation for major depressive disorder and bipolar disorder
What is trans cranial magnetic stimulation
Placement of rapid alternating magnets on scalp cause impulse to structures and is focused
Goal of trans cranial magnetic stimulation
Firing of neurons will alter pathology
, Bipolar one disorder DSM five criteria
Manic episode: 1+ week of a colon elevated, expansive or irritable mood and increase energy. 3+
symptoms from B: distractibility, indiscretion, irresponsible, grandiosity, flight of ideas, activity(Increase
goal-orient), decreased need for sleep, talkativeness or pressured speech.
Bipolar two DSM five criteria
Hypo mania and major depressive disorder: hypo mania same as mania with decreased severity and
duration and no functional impairment for episode of four or more days and no psychosis
Mixed episodes (bipolar)
Manic and depressive symptoms time by side usually with comorbid substance abuse increased risk of
suicide and psychosis
rapid cycling
Four or more cycles per year no greater than a week well period
Cyclothymia DSM-V Criteria
Two or more years of mood cycling with dysthymia and hypo mania decreased intensity than bipolar
disorder meets criteria for hypo mania but does not meet criteria for major depressive disorder
Dysthymia DSM five criteria
HE'S 2 SAD depressive symptoms lasting two or more years that is subsydromal characterized by
hopelessness decreased energy, decrease self-esteem for two years, abnormal sleep, abnormal appetite
impaired decision-making.
MAOÍ Mechanism of action generally
Catalyzes the deamination of monoamines intracellularly and MAO transport Reuptake extracellular
monoamines
MAO-A Mechanism of action
MAO-A Oxidizes serotonin norepinephrine and epinephrine
MAO-B Mechanism of action
Oxidizes phenylalanine
MAO-A and MAO-B mechanism of action
Oxidizes dopamine non-preferentially
MAOs Neumonic -2
Date with Tyra banks with wine and cheese in Maui— can cause hypertensive crisis related to tyramine
from aged food.
MAWIs= my arms weight increased= effective for atypical depression
