lOMoAR cPSD| 37664106
Blood press
Antihypertensive medicatio
a ce-inhibiTORs b eTa blOckeRs ca channel blOcke
Suffix/Prefix Suffix/Prefix Suffix/Prefix
Suffix- pril (Example- enalapril) Suffix- lol (Example- atenolol) Suffix- dipine, amil, zem
(Example- Amlodipine,
Mechanism of Action Mechanism of Action
Verapamil, Diltiazem)
MOA: prevent conversion of MOA: blockers block the effect
angiotensin I to angiotensin II of epinephrine at the receptor Mechanism of Action
sites.
Side Effects MOA: Calcium channel blocke
prevent calcium ions movemen
A- Angioedema Selective BB: affects only the
C-Cough (dry cough across myocardial cell
beta1 adrenergic sites (heart)
E- Electrolyte (Elevated K) membrane.
Nonselective BB: (lungs and
This causes relaxation of sm
heart) acts on both beta 1 and
Nursing Actions muscle.
beta 2 adrenergic sites
1. Monitor SE Relaxation of
2.Monitor K+ level smooth muscles
3.Monitor BP + Pulse Selective Beta Blockers
a ngiOTensin II RecePTOR blOckeRs
Non Selective Beta Blockers
Suffix/Prefix
Suffix- sartan (Example- Losartan)
Mechanism of Action Side Effects Side Effects
Bradycardia 1. Orthostatic Hypotension
ARBs prevent aldosterone
Hypotension 2.Bradycardia, Dizziness
release and peripheral
Mask symptoms-hypoglycemia
vasoconstriction by selectively
Nursing Actions
, lOMoAR cPSD| 37664106
GERD/PEPTIC
Gastrointesinal medication
P ROTOn PumP inhibiTORs H isTamine H2 AnTagOnisT A
Suffix/Prefix Suffix/Prefix S
1. A
Suffix- -dine-tidine
Suffix- zole (Example- Omeprazole) 2.Ma
(Example-Ranitidine) 3.Cal
Mechanism of Action 4.Sod
Mechanism of Action
MOA: Proton pump inhibitors M
MOA: Histamine (H2) receptor
suppress the secretion of HCL in MOA:
antagonist blocks the action of
the stomach by inhibiting hydrogen- compo
histamine, which produces HCL
potassium adenosine triphosphate and p
secretion.
enzyme (the enzyme that generates pepsin
This action promotes ulcer healing.
HCL). stoma
OMEPRAZOLE RANATIDINE Alumin
3. Schedule drug toavoid
Side Effects
1. Abdominal Pain interactions 1.
2.Nausea 2.D
3.Vomiting
Blood press
Antihypertensive medicatio
a ce-inhibiTORs b eTa blOckeRs ca channel blOcke
Suffix/Prefix Suffix/Prefix Suffix/Prefix
Suffix- pril (Example- enalapril) Suffix- lol (Example- atenolol) Suffix- dipine, amil, zem
(Example- Amlodipine,
Mechanism of Action Mechanism of Action
Verapamil, Diltiazem)
MOA: prevent conversion of MOA: blockers block the effect
angiotensin I to angiotensin II of epinephrine at the receptor Mechanism of Action
sites.
Side Effects MOA: Calcium channel blocke
prevent calcium ions movemen
A- Angioedema Selective BB: affects only the
C-Cough (dry cough across myocardial cell
beta1 adrenergic sites (heart)
E- Electrolyte (Elevated K) membrane.
Nonselective BB: (lungs and
This causes relaxation of sm
heart) acts on both beta 1 and
Nursing Actions muscle.
beta 2 adrenergic sites
1. Monitor SE Relaxation of
2.Monitor K+ level smooth muscles
3.Monitor BP + Pulse Selective Beta Blockers
a ngiOTensin II RecePTOR blOckeRs
Non Selective Beta Blockers
Suffix/Prefix
Suffix- sartan (Example- Losartan)
Mechanism of Action Side Effects Side Effects
Bradycardia 1. Orthostatic Hypotension
ARBs prevent aldosterone
Hypotension 2.Bradycardia, Dizziness
release and peripheral
Mask symptoms-hypoglycemia
vasoconstriction by selectively
Nursing Actions
, lOMoAR cPSD| 37664106
GERD/PEPTIC
Gastrointesinal medication
P ROTOn PumP inhibiTORs H isTamine H2 AnTagOnisT A
Suffix/Prefix Suffix/Prefix S
1. A
Suffix- -dine-tidine
Suffix- zole (Example- Omeprazole) 2.Ma
(Example-Ranitidine) 3.Cal
Mechanism of Action 4.Sod
Mechanism of Action
MOA: Proton pump inhibitors M
MOA: Histamine (H2) receptor
suppress the secretion of HCL in MOA:
antagonist blocks the action of
the stomach by inhibiting hydrogen- compo
histamine, which produces HCL
potassium adenosine triphosphate and p
secretion.
enzyme (the enzyme that generates pepsin
This action promotes ulcer healing.
HCL). stoma
OMEPRAZOLE RANATIDINE Alumin
3. Schedule drug toavoid
Side Effects
1. Abdominal Pain interactions 1.
2.Nausea 2.D
3.Vomiting
