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CCRN EXAM WITH CORRECT ANSWERS AND LATEST QUESTIONS GRADED A+

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CCRN EXAM WITH CORRECT ANSWERS AND LATEST QUESTIONS GRADED A+ Shock - ANS-a cellular disease due to inadequate perfusion or the inability for cells to use the oxygen given What happens with inadequate oxygen uptake by cells? - ANS-The body goes into anaerobic metabolism which causes lactate to rise and leads to acidosis What are the three stages of shock? - ANS-Compensatory Progressive Refractory Uncal herniation - ANS-displacement of the temporal lobe against the brain stem and third cranial nerve compresses the parasympathetic nervous system so dilated pupil on that side will be seen before LOC What is the glascow coma scale - ANS-used to asses a pt's level of consciousness and assess functional state of the brain as a whole Obtunded - ANS-the patient can speak, mumbles words Stuporous - ANS-the patient cannot speak, but they may moan or grimace homonymous hemianopsia - ANS-indicates cranial nerve three damage; occurs contralateral to the problem General rules for neurological assessment - ANS--eyes deviate to the pathology -pupil changes are ipsilateral -visual changes are contralateral -motor changes are contralateral -babinski is contralateral -if the pathology is on both sides, then babinski is present in both sides What mechanisms are activated in compensatory shock? - ANS-Sympathetic nervous system and reninangiotensin-aldosterone system (RAAS) How is BP maintained from Sympathetic nervous system? - ANS-Increased HR, vasoconstriction, and Contractility How is BP maintained from RAAS? - ANS-Renin secretion causes vasoconstriction Aldosterone release causes NA and H2O retention Signs of Compensatory Shock - ANS--Tachycardia -Tachypnea -Respiratory alkalosis -Oliguria -Cool, pale skin -Thirsty -BP MAINTAINED Progressive Stage of Shock ( Mechanisms Failing) - ANS--worsening Tachycardia HYPOTENSION -metabolic acidosis -changes in LOC mottled skin Refractory Stage of Shock (not responding to treatment) - ANS--severe hypoperfusion -Failure of organs Name the types of shock - ANS-Cardiogenic Hypovolemic Neurogenic Anaphylactic Septic Obstructive What are the causes of hypovolemic shock? - ANS-third-spacing, hemorrhage, GI bleed, burns, excessive diuresis What happens to the pulse pressure in hypovolemic shock? - ANS-Narrow Everything decreases in hypovolemic shock except? - ANS-SVR increases How do you treat hypovolemic shock? - ANS-aggressive fluid loading- LR is recommended by ACS replace blood/plts as needed avoid vasopressors How do you treat hemorrhagic shock? - ANS-Stop the bleeding

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CCRN EXAM 2024-2025 WITH CORRECT
ANSWERS AND LATEST QUESTIONS
GRADED A+




Shock - ANS-a cellular disease due to inadequate perfusion or the inability for cells to use the oxygen
given



What happens with inadequate oxygen uptake by cells? - ANS-The body goes into anaerobic metabolism
which causes lactate to rise and leads to acidosis



What are the three stages of shock? - ANS-Compensatory

Progressive

Refractory

Uncal herniation - ANS-displacement of the temporal lobe against the brain stem and third cranial nerve

compresses the parasympathetic nervous system so dilated pupil on that side will be seen before LOC



What is the glascow coma scale - ANS-used to asses a pt's level of consciousness and assess functional
state of the brain as a whole

,Obtunded - ANS-the patient can speak, mumbles words



Stuporous - ANS-the patient cannot speak, but they may moan or grimace



homonymous hemianopsia - ANS-indicates cranial nerve three damage; occurs contralateral to the
problem



General rules for neurological assessment - ANS--eyes deviate to the pathology

-pupil changes are ipsilateral

-visual changes are contralateral

-motor changes are contralateral

-babinski is contralateral

-if the pathology is on both sides, then babinski is present in both sides

What mechanisms are activated in compensatory shock? - ANS-Sympathetic nervous system and renin-
angiotensin-aldosterone system (RAAS)



How is BP maintained from Sympathetic nervous system? - ANS-Increased HR, vasoconstriction, and
Contractility



How is BP maintained from RAAS? - ANS-Renin secretion causes vasoconstriction

Aldosterone release causes NA and H2O retention



Signs of Compensatory Shock - ANS--Tachycardia

-Tachypnea

-Respiratory alkalosis

-Oliguria

-Cool, pale skin

-Thirsty

-BP MAINTAINED

,Progressive Stage of Shock ( Mechanisms Failing) - ANS--worsening Tachycardia

HYPOTENSION

-metabolic acidosis

-changes in LOC

mottled skin



Refractory Stage of Shock (not responding to treatment) - ANS--severe hypoperfusion

-Failure of organs



Name the types of shock - ANS-Cardiogenic

Hypovolemic

Neurogenic

Anaphylactic

Septic

Obstructive



What are the causes of hypovolemic shock? - ANS-third-spacing, hemorrhage, GI bleed, burns, excessive
diuresis



What happens to the pulse pressure in hypovolemic shock? - ANS-Narrow



Everything decreases in hypovolemic shock except? - ANS-SVR increases



How do you treat hypovolemic shock? - ANS-aggressive fluid loading- LR is recommended by ACS

replace blood/plts as needed

avoid vasopressors



How do you treat hemorrhagic shock? - ANS-Stop the bleeding

, PRBCs with FFP/PLTs/Cryo

prevent hypothermia

will need calcium and magnesium replacement



Massive Transfusion Protocol - ANS-10 units PRBCs in 24 hours or 5 units in less than 3



What is the triad of death? - ANS-Hypothermia

Acidosis

Coagulopathy



Criteria for Systemic Inflammatory Response Syndrome (SIRS) - ANS-2 or more of the following

-T >38C or <36C

-HR>90

RR>20 or PACO2<32

WBCs >12 <4



Sepsis - ANS-organ dysfunction caused by an abnormal host response to a microorganism

infection + organ dysfunction



qSOFA - ANS-2 or more of:

Hypotension: SBP less than or equal to 100 mmHg

Altered mental status (any GCS less than 15)

Tachypnoea: RR greater than or equal to 22



identified patients with suspected infection who are likely to have a prolonged ICU stay or to die in the
hospital



septic shock - ANS-sepsis and uncontrollable decreased blood pressure

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