Blood
liquid thatfills thevascularspaces flowsthrough thevessels with a certain pressure and
speed and passes through the vicinity of all cellsofbody hemodynamic function
plays a role in respiration because it carries 0 and coz
carries hormones from release site target organs
Bloodplasma transport nutrients that havebeen digested to andmetabolicwaste from
kidneys lungs and skin ofelimination
organs
in regulation ofbody's To receiving distributing heat H2Oinbloodplasma hashighthermalconductivity
intervenes
Throughplasmaproteincomponents andleukocytes bloodplaysrolein antimicrobial defense
has a constantpH mechanisms to maintain acid basebalance importantrole change in pH
change in the 0 transportcapacity affectbreathing changesplasma Ca neuromuscular excitabile
Stopping bleeding hemostasis maintain fluid coagulant balance plasmaproteins paletelets involved
1 Temperature
Humanbody homeotherm body constantTo maintained through thermoregulation mechanisms
1 participants blood endocrine system hypothalamic centers
Toof an organ depends on
intensity of metabolic R's moreintense moreheatreleased Tohigher
Thermoregulation mechanisms available thermogenesis thermolysis
Bloodmedium TO 37 C
Lung 36 3615 heatis lost through ventilation evaporationofH2oonsurfaceofairways
her 38 40 insuprahepaticveins blood To 39 41 Ro in liver
intense metabolic
L center ofmetabolic processes
Testicle 34 30 necessary for spermatogenesis
Body and blood To depends on age sex time day
of day i night
diet physical activity emotional state
Pathological increases in To may indicate inflammatory or infectious disease febrilesyndrom
Hyperthermia bloodTo 40 C 42 C Coma death can occur
Hypothermia symptoms appearwhen To drops 1 2 Cby 35 C below 30 C highrisk
2 Color
Blood red given by hemoglobin intraerythrocytic pigment 15g ok
heme group formed byprotopophyrin Ix and Fe ofhemoglobin
Fe absorb lightrays with wavelenghts corresponding to violetand green colors andrefl
red ones
Oxyhemoglobin hemoglobin saturated with 0 98 100 arterialblood brightred
60 75 venous blood
, Reducedhemoglobin deoxygenated hemoglobin 0 2 arterial blood
25 venousblood dark red
Thecolor venous blood
ismodified by
of
speed with whichbloodcirculates through an organ speedhigh extraction 0
of
of
color the venous blood is lighter
degree activity organ alsomatters in contracting muscles secretingglands
of
Morecapillaries of retrous
open and circulatoryflowincreases increasesamount hemoglobin in
of totalhemoglobin
oxy
reduced hemoglobin invenousblood tomorethan 5glok
of
Cyanosis increased
Normal levels
ofreduced hemoglobin 0,75 glok in arterial blood
24 capillary
4,5 venous
In heart failure decreased contraction force of the ventricular myocardium
systemic circulation is sloweddown desaturation of hemoglobin in tissues
reduced Hb increases and central cyanosis appears
There's also a peripheral mechanism the appearance cyanosis whendue to vasoconstriction
for of
hyperviscosity orbothforexample at thelevel ofthe extremities in coldconditions thecirculation
is sloweddown hemoglobin desaturation increases cyanosis appears
Plasma yellow bile pigment total bilirubin 1,2 mg ok maximumvalue
Light yellowcolor bile pigment bilirubin obtained by the metabolism Hb
Eliminated by liver through bile
of
opalescent lactescent in postprandial blood hyperlipidemia blood or in a patient
with diabetes or atherosclerosis
Jaundice increase in total bilirubin above 2,4 mgtoll
is a clinical sign
it means theyellowcoloringoftheconjunctiva salera eyes teguments
In carbonmonoxidepoisining carboxyhemoglobin Hbo of
pathologinalcompound
of Hb is
rapidlyformed affinityofHbfor co is 250 times greaterthan oz fixation is irreversib
Met hemoglobinemia Fe fromHb for
Fe nolongerfixes 02 but hydroxyl radicals OH
bond beingmorestable than 02
In case CO poisoning hypoxemia and tissuehypoxiaset in with clinicalsignsappearing
of
from 10 methemoglobin in total hemoglobin Blood is brownand blue gray cyanosis of
the mucous membranes andteguments appears
Maximum normalblood met Hb is it in adults
and 1,5 in children who don't have
sufficiently developed reduction mechanisms There's alsohereditary methemoglobinemia
caused bythe lack synthesis of theinvolved reductase
of