https://www.stuvia.com/
Test bank for Davis Advantage for
v v v v v
Pathophysiology Introductory Concepts and
v v v v
Clinical Perspectives
v v
,https://www.stuvia.com/
Stuvia.com v- vThe vMarketplace vto vBuy vand vSell vyour
vStudy vMaterial
Chapter v1, vThe vCell vin vHealth vand vIllness
Multiple vChoice
Identify vthe vchoice vthat vbest vcompletes vthe vstatement vor vanswers vthe vquestion.
v 1. v Which vstatement vregarding vthe vsodium–potassium vpump vis vcorrect?
1. The vcell’s vplasma vmembrane vis vmore vsoluble vto vsodium vions vthan vpotassium vions.
2. The vconcentration vof vsodium vions vshould vbe vhigher vinside vthe vcell vcompartment.
3. The vconcentration vof vpotassium vions vshould vbe vhigher voutside
vthe vcellvcompartment.
4. The vactive vtransport vinvolves vpumping vout vthree vsodium vions vand vpumping vin vtwo
potassium vions.
v 2. vIn vthe vabsence vof voxygen, vwhich vcellular vfunction vcreates vthe vsame vamount vof venergy vas vis
vcreated vinvthe vpresence vof voxygen?
1. Dissipation vof vpyruvic vacid
2. Initiation vof vthe vcitric vacid vcycle
3. Activation vof vacetyl-coenzyme vA
4. Creation vof vacidosis vvia vlactic vacid
v 3. v How vmany vadenosine vtriphosphates v(ATPs) vare vproduced vin vaerobic venergy vmetabolism?
1. v vv2
2. v vv3
3. v vv 34
4. v vv 53
v 4. v Which vcell vorganelles vdiffer vin vtheir vnumber vaccording vto vthe vcell’s venergy vneeds?
1. Ribosomes
2. Mitochondria
3. Ribonucleic vacids
4. Deoxyribonucleic vacids
v 5. v Which voption vbest vsupports vthe vreason vmore venergy vis vproduced vwhen va vperson vis vexercising?
1. Exercise vcauses van vincrease vin vthe vsynthesis vof vprotein.
2. There vis van vincrease vin vthe vproduction vof vpyruvic vacid vin vthe vcells.
3. The vconversion vof vpyruvic vacid vto vlactic vacid vis vincreased vby vexercise.
4. Muscle vcells vhave vmore vmitochondria vto vmeet venergy vdemands.
v 6. v When vdoes vribosomal vprotein vsynthesis vcease?
1. During vendoplasmic vreticulum vstress
2. During vthe vsynthesis vof vadenosine vtriphosphate v(ATP)
3. During va vsevere vhypoxic vstate
4. During vthe vprocessing vof vprohormone
v 7. v Which vcellular vorganelles vare vresponsible vfor vpropelling vmucus vand vinhaled vdebris vout vof vthe
vlungs?
1. Cilia
2. Microfilaments
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3. Secretory vvesicles
4. Endoplasmic vreticula
v 8. v Which vare vthe vkey vproteins vin vthe vcontractile vunits vof vthe vmuscle vcells?
1. Actin vand vmyosin
2. Prohormone vand vtubulin
3. Tubulin vand vactin
4. Myosin vand vprohormone
v 9. v v vWhich vdeficiency vcauses vTay–Sachs vdisease?
1. Proteasome
2. Peroxisome
3. Macrophage
4. Lysosomal venzymes
v v v 10. v Which vis va vcharacteristic vof vadrenoleukodystrophy?
1. Accumulation vof vganglioside
2. Cessation vof vribosomal vprotein vsynthesis
3. Acceleration vof vcellular vproteasome vactivity
4. Accumulation vof vlong-chain vfatty vacids vin vthe vnervous vsystem
v v v v11. v Which vstatement vregarding vendoplasmic vreticulum v(ER) vstress vis vcorrect?
1. During vER vstress, vproteins vare vrapidly vdegraded.
2. During vER vstress, vlipids vcannot vtravel vto vtheir vproper vintracellular vlocations.
3. During vER vstress, vlong-chain vfatty vacids vaccumulate vin vthe vnervous vsystem.
4. During vER vstress, vnondegraded vsubstances vaccumulate vin vthe vcells.
v v v 12. v A vclient vis vdiagnosed vwith vtype v1 vdiabetes vmellitus. vAt va vcellular vlevel, vwhich vfunction vis
vlikely vto vbevinvolved?
1. Inability vof vribosomes vto vproduce va vspecific vtype vof vprotein
2. Incorrect vprocessing vof va vprotein vby vthe vGolgi vapparatus
3. Stagnation vof va vpreviously vdynamic vaction vin vmicrotubules
4. Obstruction vof vthe vsmooth vendoplasmic vreticulum
v v v 13. v A vnewborn vpatient vexhibits vcharacteristics vof vsevere vphysical vdeformities. vWhich vcellular
vcomponentvis vexamined vto vdetermine vthe vcause vand vprobability vof vthe vdisease vbeing
vgenetically vtransferred?
1. Transfer vRNA
2. Ribosomal vRNA
3. Double vhelix vof vDNA
4. Mitochondrial vDNA
v v v 14. v A vhiker vexperiences vmuscle vpain vand vacidosis vwhile vascending va vmountain vduring va
vlong, vsteepvclimb. vWhich vis vthe vreason vfor vthese vmanifestations?
1. Cellular vhypoxia
2. Autolysis
3. Heterolysis
4. Cellular vedema
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vStudy vMaterial
v v v 15. v Which vfactor vprovides vDNA vthe vunique vmolecular vability vto vreplicate?
1. The vpairing vof vnitrogenous vbases
2. The vpresence vof vpyrimidine vbases
3. The vpresence vof vnucleotides
4. The vnitrogenous vbase vand vphosphate vbond
v v v 16. v How vmany vnitrogenous vbases vcompose va vsingle vcodon?
1. v v v 2
2. v v v 3
3. v v v 4
4. v v v 5
v v v 17. v Which vcomponents vform vthe vstructure vof vDNA?
1. Nucleotides
2. Amino vacids
3. Fatty vacids
4. Phosphates
v v v 18. v Which vfactor vis vessential vin vorder vfor vprotein vsynthesis vto voccur?
1. Free-standing vribosomes vwithin vthe vcell
2. Protein vblueprint vfrom vthe vcell vof vthe vDNA
3. Specific vinformation vfrom vthe vnucleus vof vthe vcell
4. Transfer vRNA vto vmove vthe vprotein vout vof vthe vcell
v v v 19. v Tetracycline vantibiotic vis vprescribed vfor van vadult vclient vwith vchlamydia vinfection.
vWhich vis vthevmechanism vof vaction vof vthe vdrug?
1. It vprevents vthe vreplication vof vbacteria.
2. It valters vthe vconfiguration vof vbacterial vcytoplasm.
3. It vinterferes vwith vthe vfunction vof vbacterial vribosomes.
4. It vinhibits vthe vfunctions vof vbacterial vmitochondria.
v v v 20. v Where vdoes vthe vconversion vof va vprohormone vinto va vhormone vtake vplace?
1. In vthe vribosomes
2. In vthe vGolgi vapparatus
3. In vthe vsecretory vgranules
4. In vthe vendoplasmic vreticulum
v v v 21. v Which vis vthe vcell’s v“master vmind”?
1. Nucleus
2. Ribosome
3. Golgi vapparatus
4. Endoplasmic vreticulum
Multiple vResponse
Identify vone vor vmore vchoices vthat vbest vcomplete vthe vstatement vor vanswer vthe vquestion.
v v v 22. v Which vstatements vregarding vthe vmicrotubules vare vtrue? vSelect vall vthat vapply.
Copyright v© v2020 vF. vA. vDavis vCompany
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Distribution vof vthis vdocument vis villegal
Test bank for Davis Advantage for
v v v v v
Pathophysiology Introductory Concepts and
v v v v
Clinical Perspectives
v v
,https://www.stuvia.com/
Stuvia.com v- vThe vMarketplace vto vBuy vand vSell vyour
vStudy vMaterial
Chapter v1, vThe vCell vin vHealth vand vIllness
Multiple vChoice
Identify vthe vchoice vthat vbest vcompletes vthe vstatement vor vanswers vthe vquestion.
v 1. v Which vstatement vregarding vthe vsodium–potassium vpump vis vcorrect?
1. The vcell’s vplasma vmembrane vis vmore vsoluble vto vsodium vions vthan vpotassium vions.
2. The vconcentration vof vsodium vions vshould vbe vhigher vinside vthe vcell vcompartment.
3. The vconcentration vof vpotassium vions vshould vbe vhigher voutside
vthe vcellvcompartment.
4. The vactive vtransport vinvolves vpumping vout vthree vsodium vions vand vpumping vin vtwo
potassium vions.
v 2. vIn vthe vabsence vof voxygen, vwhich vcellular vfunction vcreates vthe vsame vamount vof venergy vas vis
vcreated vinvthe vpresence vof voxygen?
1. Dissipation vof vpyruvic vacid
2. Initiation vof vthe vcitric vacid vcycle
3. Activation vof vacetyl-coenzyme vA
4. Creation vof vacidosis vvia vlactic vacid
v 3. v How vmany vadenosine vtriphosphates v(ATPs) vare vproduced vin vaerobic venergy vmetabolism?
1. v vv2
2. v vv3
3. v vv 34
4. v vv 53
v 4. v Which vcell vorganelles vdiffer vin vtheir vnumber vaccording vto vthe vcell’s venergy vneeds?
1. Ribosomes
2. Mitochondria
3. Ribonucleic vacids
4. Deoxyribonucleic vacids
v 5. v Which voption vbest vsupports vthe vreason vmore venergy vis vproduced vwhen va vperson vis vexercising?
1. Exercise vcauses van vincrease vin vthe vsynthesis vof vprotein.
2. There vis van vincrease vin vthe vproduction vof vpyruvic vacid vin vthe vcells.
3. The vconversion vof vpyruvic vacid vto vlactic vacid vis vincreased vby vexercise.
4. Muscle vcells vhave vmore vmitochondria vto vmeet venergy vdemands.
v 6. v When vdoes vribosomal vprotein vsynthesis vcease?
1. During vendoplasmic vreticulum vstress
2. During vthe vsynthesis vof vadenosine vtriphosphate v(ATP)
3. During va vsevere vhypoxic vstate
4. During vthe vprocessing vof vprohormone
v 7. v Which vcellular vorganelles vare vresponsible vfor vpropelling vmucus vand vinhaled vdebris vout vof vthe
vlungs?
1. Cilia
2. Microfilaments
Copyright v© v2020 vF. vA. vDavis vCompany
Downloaded vby: vAriah v|
Distribution vof vthis vdocument vis villegal
,https://www.stuvia.com/
Stuvia.com v- vThe vMarketplace vto vBuy vand vSell vyour
vStudy vMaterial
3. Secretory vvesicles
4. Endoplasmic vreticula
v 8. v Which vare vthe vkey vproteins vin vthe vcontractile vunits vof vthe vmuscle vcells?
1. Actin vand vmyosin
2. Prohormone vand vtubulin
3. Tubulin vand vactin
4. Myosin vand vprohormone
v 9. v v vWhich vdeficiency vcauses vTay–Sachs vdisease?
1. Proteasome
2. Peroxisome
3. Macrophage
4. Lysosomal venzymes
v v v 10. v Which vis va vcharacteristic vof vadrenoleukodystrophy?
1. Accumulation vof vganglioside
2. Cessation vof vribosomal vprotein vsynthesis
3. Acceleration vof vcellular vproteasome vactivity
4. Accumulation vof vlong-chain vfatty vacids vin vthe vnervous vsystem
v v v v11. v Which vstatement vregarding vendoplasmic vreticulum v(ER) vstress vis vcorrect?
1. During vER vstress, vproteins vare vrapidly vdegraded.
2. During vER vstress, vlipids vcannot vtravel vto vtheir vproper vintracellular vlocations.
3. During vER vstress, vlong-chain vfatty vacids vaccumulate vin vthe vnervous vsystem.
4. During vER vstress, vnondegraded vsubstances vaccumulate vin vthe vcells.
v v v 12. v A vclient vis vdiagnosed vwith vtype v1 vdiabetes vmellitus. vAt va vcellular vlevel, vwhich vfunction vis
vlikely vto vbevinvolved?
1. Inability vof vribosomes vto vproduce va vspecific vtype vof vprotein
2. Incorrect vprocessing vof va vprotein vby vthe vGolgi vapparatus
3. Stagnation vof va vpreviously vdynamic vaction vin vmicrotubules
4. Obstruction vof vthe vsmooth vendoplasmic vreticulum
v v v 13. v A vnewborn vpatient vexhibits vcharacteristics vof vsevere vphysical vdeformities. vWhich vcellular
vcomponentvis vexamined vto vdetermine vthe vcause vand vprobability vof vthe vdisease vbeing
vgenetically vtransferred?
1. Transfer vRNA
2. Ribosomal vRNA
3. Double vhelix vof vDNA
4. Mitochondrial vDNA
v v v 14. v A vhiker vexperiences vmuscle vpain vand vacidosis vwhile vascending va vmountain vduring va
vlong, vsteepvclimb. vWhich vis vthe vreason vfor vthese vmanifestations?
1. Cellular vhypoxia
2. Autolysis
3. Heterolysis
4. Cellular vedema
Copyright v© v2020 vF. vA. vDavis vCompany
Downloaded vby: vAriah v|
Distribution vof vthis vdocument vis villegal
,https://www.stuvia.com/
Stuvia.com v- vThe vMarketplace vto vBuy vand vSell vyour
vStudy vMaterial
v v v 15. v Which vfactor vprovides vDNA vthe vunique vmolecular vability vto vreplicate?
1. The vpairing vof vnitrogenous vbases
2. The vpresence vof vpyrimidine vbases
3. The vpresence vof vnucleotides
4. The vnitrogenous vbase vand vphosphate vbond
v v v 16. v How vmany vnitrogenous vbases vcompose va vsingle vcodon?
1. v v v 2
2. v v v 3
3. v v v 4
4. v v v 5
v v v 17. v Which vcomponents vform vthe vstructure vof vDNA?
1. Nucleotides
2. Amino vacids
3. Fatty vacids
4. Phosphates
v v v 18. v Which vfactor vis vessential vin vorder vfor vprotein vsynthesis vto voccur?
1. Free-standing vribosomes vwithin vthe vcell
2. Protein vblueprint vfrom vthe vcell vof vthe vDNA
3. Specific vinformation vfrom vthe vnucleus vof vthe vcell
4. Transfer vRNA vto vmove vthe vprotein vout vof vthe vcell
v v v 19. v Tetracycline vantibiotic vis vprescribed vfor van vadult vclient vwith vchlamydia vinfection.
vWhich vis vthevmechanism vof vaction vof vthe vdrug?
1. It vprevents vthe vreplication vof vbacteria.
2. It valters vthe vconfiguration vof vbacterial vcytoplasm.
3. It vinterferes vwith vthe vfunction vof vbacterial vribosomes.
4. It vinhibits vthe vfunctions vof vbacterial vmitochondria.
v v v 20. v Where vdoes vthe vconversion vof va vprohormone vinto va vhormone vtake vplace?
1. In vthe vribosomes
2. In vthe vGolgi vapparatus
3. In vthe vsecretory vgranules
4. In vthe vendoplasmic vreticulum
v v v 21. v Which vis vthe vcell’s v“master vmind”?
1. Nucleus
2. Ribosome
3. Golgi vapparatus
4. Endoplasmic vreticulum
Multiple vResponse
Identify vone vor vmore vchoices vthat vbest vcomplete vthe vstatement vor vanswer vthe vquestion.
v v v 22. v Which vstatements vregarding vthe vmicrotubules vare vtrue? vSelect vall vthat vapply.
Copyright v© v2020 vF. vA. vDavis vCompany
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Distribution vof vthis vdocument vis villegal
