clinical manifestations of diabetes insipidus
polydipsia, polyphagia, polyuria, fatigue, vision disturbances
nursing interventions for diabetes insipidus
patient education on glycemic index, nutrition, exercise promotion, monitoring
bg, foot care
diagnostic testing for diabetes insipidus
water deprivation test (a subcutaneous injection of vasopressin produces urine
output with an increased specific gravity and osmolality)
signs and symptoms of diabetes insipidus
polyuria (urinary output of 3 to 20 L/day of dilute urine), polydipsia, weight
loss, fatigue, nocturia
causes diabetes insipidus
results from a deficiency of ADH. decreased ADH reduces the ability of the
distal renal tubules in the kidneys to collect and concentrate urine.
cushing's syndrome safety education
instruct patient to follow lifelong hormone therapy replacement, obtain annual
flu vaccine, wear medical alert bracelet that lists cushings as a disease along
with medication, monitor bg and bp
lab changes with cushings syndrome
17-hydroxycorticosteroids, and 17-ketosteroids, plasma and urine levels are
elevated, elevated serum sodium, decreased serum potassium, and increased
blood glucose levels
cushings syndrome pathophysiology
caused by an oversecretion of the hormones the adrenal cortex produces
causes of cushings syndrome
, can be the result of a tumor in the pituitary gland, resulting in release of the
hormone ACTH, it can also be the result of hyperplasia of the adrenal cortex,
and it can also result from long-term use of glucocorticoids to treat other
conditions
nursing actions for addison's disease
prevent circulatory shock, monitor for fluid and electrolyte imbalance, monitor
and treat hyperkalemia and hyperhlycemia
clincial manifestations for addison's disease
weight loss, craving for salt, bronzed skin, weakness and fatigue, hyponatremia,
hyperkalemia, hypoglycemia, hypercalcemia
discharge teaching for addison's disease
avoid discontinuing medications abruptly, report manifestations of cushing
syndrome, take medications with food, report symptoms of adrenal
insufficiency, and report acute illness to provider
pathophysiology of primary addison's disease
due to destruction of adrenal cortex caused by autoimmune disease, tb,
histoplasmosis, adrenalectomy, cancer, radiation therapy of abdomen
pathophysiology of secondary addison's disease
surgical removal of adrenal glands, hemorrhagic infarction of glands,
medications, hypopiuitarism
medications for addison's disease
hydrocortisone, prednisone, cortisone
thyroidectomy
surgical removal of part or all of the thyroid gland
subtotal thyroidectomy
performed for the treatment of hyperthyroidism when medication therapy fails,
or radiation therapy is contraindicated. no hormone replacement therapy
needed.
polydipsia, polyphagia, polyuria, fatigue, vision disturbances
nursing interventions for diabetes insipidus
patient education on glycemic index, nutrition, exercise promotion, monitoring
bg, foot care
diagnostic testing for diabetes insipidus
water deprivation test (a subcutaneous injection of vasopressin produces urine
output with an increased specific gravity and osmolality)
signs and symptoms of diabetes insipidus
polyuria (urinary output of 3 to 20 L/day of dilute urine), polydipsia, weight
loss, fatigue, nocturia
causes diabetes insipidus
results from a deficiency of ADH. decreased ADH reduces the ability of the
distal renal tubules in the kidneys to collect and concentrate urine.
cushing's syndrome safety education
instruct patient to follow lifelong hormone therapy replacement, obtain annual
flu vaccine, wear medical alert bracelet that lists cushings as a disease along
with medication, monitor bg and bp
lab changes with cushings syndrome
17-hydroxycorticosteroids, and 17-ketosteroids, plasma and urine levels are
elevated, elevated serum sodium, decreased serum potassium, and increased
blood glucose levels
cushings syndrome pathophysiology
caused by an oversecretion of the hormones the adrenal cortex produces
causes of cushings syndrome
, can be the result of a tumor in the pituitary gland, resulting in release of the
hormone ACTH, it can also be the result of hyperplasia of the adrenal cortex,
and it can also result from long-term use of glucocorticoids to treat other
conditions
nursing actions for addison's disease
prevent circulatory shock, monitor for fluid and electrolyte imbalance, monitor
and treat hyperkalemia and hyperhlycemia
clincial manifestations for addison's disease
weight loss, craving for salt, bronzed skin, weakness and fatigue, hyponatremia,
hyperkalemia, hypoglycemia, hypercalcemia
discharge teaching for addison's disease
avoid discontinuing medications abruptly, report manifestations of cushing
syndrome, take medications with food, report symptoms of adrenal
insufficiency, and report acute illness to provider
pathophysiology of primary addison's disease
due to destruction of adrenal cortex caused by autoimmune disease, tb,
histoplasmosis, adrenalectomy, cancer, radiation therapy of abdomen
pathophysiology of secondary addison's disease
surgical removal of adrenal glands, hemorrhagic infarction of glands,
medications, hypopiuitarism
medications for addison's disease
hydrocortisone, prednisone, cortisone
thyroidectomy
surgical removal of part or all of the thyroid gland
subtotal thyroidectomy
performed for the treatment of hyperthyroidism when medication therapy fails,
or radiation therapy is contraindicated. no hormone replacement therapy
needed.
