Cardiac output ✔️✔️stroke volume x heart rate (amount of blood ejected per contraction)
Primary compensatory mechanism for low cardiac output in peds ✔️✔️Tachycardia
Late sign of low CO ✔️✔️Hypotension, bradycardia is ominous sign in peds
What is stroke volume influenced by ✔️✔️afterload, preload, contractility
Preload ✔️✔️volume evaluated by CVP
How to increase preload ✔️✔️volume (blood/fluids), pressprs
How to decrease preload ✔️✔️diuretics
What is afterload ✔️✔️resistance to ventricular emptying, measured by systemic vascular resistance
When is afterload decreased and how would you treat ✔️✔️distributive shocks (neurogenic, septic,
anaphylactic) and by vasodilators- give them vasopressors
How would you decrease afterload ✔️✔️vasodilators
when is afterload increased ✔️✔️HTN, shock compensation
SNS stimulation releases what ✔️✔️epinephrine, catecholamines, norepi-increases HR,
vasoconstriction, pupil dilation, catecholamines increase BSG through glycogenesis
, Parasympathetic system does what ✔️✔️Decreases HR
MAP equation ✔️✔️(SBP + 2DBP)/3
Pulse pressure ✔️✔️difference between systolic and diastolic pressure
Widened PP seen in... ✔️✔️increased ICP
Narrowed PP seen in.... ✔️✔️early stages of shock
Cushing's triad indicates what and what are the signs ✔️✔️Increased ICP, widened PP (or HTN),
irregular breathing pattern, and bradycardia
What do chronotropes do and what is an example ✔️✔️Control HR- cardizem is a negative
chronotrope, atropine positive
What do inotropes do and what is an example ✔️✔️Affect contractility- dobutamine and dopamine
are positive inotropes
What do dromotropes do and what is an example ✔️✔️Control automaticity- positive dromotrope is
epi, negative dromotrope is beta blockers
Beta blockers end in and do what ✔️✔️-olols, slow HR and lower BP, may mask signs of early shock
and hypoglycemia
Calcium channel blockers end in and do what ✔️✔️-dipine (and cardizem), control ventricular rate in a
fib and HTN
ACE inhibitors ✔️✔️"PRIL" Captopril, Enalapril, Afosiopril