Motor Speech Disorders Exam 2
1.flaccid dysarthria relates to damage where?: lower motor neurons
(motor aspect of cranial nerves)
2.flaccid dysarthria site of lesion: final common pathway (nuclei, axons,
neuro- muscular junction)
3.status of reflexes for flaccid dysarthria: nonexistent, minimal
4.associated terms and confirmatory signs flaccid dysarthria: hypotonia,
atro- phy, diminished reflexive responses/voluntary movement, bulbar
palsy, fasiculations, fibrillations
5.flaccid dysarthria related to velopharyngeal: nasal emission,
hypernasality
6.flaccid dysarthria related to laryngeal: often breathy, audible inspirations
7.flaccid dysarthria related to oral articulation: imprecise labial control,
jaw movements, tongue (V, VII, XII)
8.flaccid dysarthria: characteristics will reflect CN function ( is a
theme): weakness
9.flaccid dysarthria bilateral vs. unilateral involvement: mandible -
bilateral (open mouth posture)
velum - unilateral (weak side rests lower and less
elevation) tongue - unilateral (atrophy and deviation to
weak side)
10.AMR patterns flaccid dysarthria: generally even
11.stress testing flaccid dysarthria: specific to neuromuscular junction
(muscles become progressively weak)
12.speech vs. nonspeech movements flaccid dysarthria: different
networks (may not reflect same outcome)
13.nonprogressive examples flaccid dysarthria: bell palsy, brainstem
stroke/trauma/tumor, locked-in syndrome, guillain-barre syndrome,
moebius syn- drome
14.isolated to nucleus of VII cranial nerve (usually just one side),
upper/lower facial muscles affected: bell palsy
15.trauma or infarction to ventral pons area, lesion isolates cognitive
from motor system: locked-in syndrome
16.inflammation of PNS nerves, demyelination of nerves, paralysis ascends-
: guillain-barre syndrome
17.bilateral paralysis of VII, VI: moebius syndrome
18.progressive examples of flaccid dysarthria: myasthenia gravis,
amyotrophic lateral sclerosis
19.breakdown at neuromuscular junction, muscles become less efficient
with repeated use: myasthenia gravis
1/
11
, Motor Speech Disorders Exam 2
20.for those with myasthenia gravis, listen for deterioration during:
lengthy talking tasks (stress testing)
2/
11
1.flaccid dysarthria relates to damage where?: lower motor neurons
(motor aspect of cranial nerves)
2.flaccid dysarthria site of lesion: final common pathway (nuclei, axons,
neuro- muscular junction)
3.status of reflexes for flaccid dysarthria: nonexistent, minimal
4.associated terms and confirmatory signs flaccid dysarthria: hypotonia,
atro- phy, diminished reflexive responses/voluntary movement, bulbar
palsy, fasiculations, fibrillations
5.flaccid dysarthria related to velopharyngeal: nasal emission,
hypernasality
6.flaccid dysarthria related to laryngeal: often breathy, audible inspirations
7.flaccid dysarthria related to oral articulation: imprecise labial control,
jaw movements, tongue (V, VII, XII)
8.flaccid dysarthria: characteristics will reflect CN function ( is a
theme): weakness
9.flaccid dysarthria bilateral vs. unilateral involvement: mandible -
bilateral (open mouth posture)
velum - unilateral (weak side rests lower and less
elevation) tongue - unilateral (atrophy and deviation to
weak side)
10.AMR patterns flaccid dysarthria: generally even
11.stress testing flaccid dysarthria: specific to neuromuscular junction
(muscles become progressively weak)
12.speech vs. nonspeech movements flaccid dysarthria: different
networks (may not reflect same outcome)
13.nonprogressive examples flaccid dysarthria: bell palsy, brainstem
stroke/trauma/tumor, locked-in syndrome, guillain-barre syndrome,
moebius syn- drome
14.isolated to nucleus of VII cranial nerve (usually just one side),
upper/lower facial muscles affected: bell palsy
15.trauma or infarction to ventral pons area, lesion isolates cognitive
from motor system: locked-in syndrome
16.inflammation of PNS nerves, demyelination of nerves, paralysis ascends-
: guillain-barre syndrome
17.bilateral paralysis of VII, VI: moebius syndrome
18.progressive examples of flaccid dysarthria: myasthenia gravis,
amyotrophic lateral sclerosis
19.breakdown at neuromuscular junction, muscles become less efficient
with repeated use: myasthenia gravis
1/
11
, Motor Speech Disorders Exam 2
20.for those with myasthenia gravis, listen for deterioration during:
lengthy talking tasks (stress testing)
2/
11
