SOLUTIONS GRADED A++ LATEST UPDATE
Not enough glucose?
Glycogenolysis
Gluconeogenesis
Lipolysis
Beta oxidation of fatty acids
Protein breakdown
Too much glucose?
Protein synthesis
Triglyceride synthesis
Glycogenesis
Glycolysis
Anabolism?
synthesis of a complex molecule from simpler molecules and stored energy
State of body: fed
Hormone involved: insulin
Catabolism?
breakdown of complex molecules to simpler molecules and energy
State of body: fasting
Hormones involved: cortisol, glucagon, epinephrine
,insulin
inhibit?
stimulate?
Increases anabolism
Inhibits:
Proteolysis
Lipolysis
Gluconeogenesis:make glucose out of other things
Glycogenolysis: breakdown of glucose
Beta oxidation of fatty acids
stimulates:
Triglyceride formation in fat cells
Protein synthesis in muscle
Glycogenesis: formation of glycogen
Glycolysis
Glucose uptake
Na+/K+ ATPase (take in K+, out Na+)
Triggered by: increased glucose
Glucagon (alpha cells of pancreas)
stimulates?
, Stimulates:
Glycogenolysis
Gluconeogenesis
Lipolysis
Beta oxidation of fatty acids
Triggered by: decreased glucose, exercise, infection, stress
Epinephrine (adrenal medulla)
inhibit?
stimulate?
Stimulates:
Glycogenolysis
Gluconeogenesis
Lipolysis
Glucagon secretion
Inhibition:
Insulin secretion
Protein degradation in muscle
Characteristic: maximal effect is short due to desensitization ( in diabetes, muscle stop
responding to epinephrine due to chronic stimulation→ start degrading muscle)
Cortisol (adrenal cortex)
stimulate?