TESTBANK q
RUBIN'S PATHOLOGY: CLIN
q q
ICOPATHOLOGIC FOUNDATI q
ONS OF MEDICINE q q
7th Edition By David S. Strayer, Emanuel Rubin
q q q q q q q
TESTBANK q
,TestqBankqRubin'sqPathology:qClinicopathologicqFoundationsqofqMedicineq7thq Edition
TableqofqContents:
Chapterq1:qCellqAdaptation,qInjuryqandqDeath
Chapterq2:qInflammation
Chapterq3:qRepair,qRegenerationqandqFibrosis
Chapterq4:qImmunopathology
Chapterq5:qNeoplasia
Chapterq6:qDevelopmentalqandqGeneticqDiseases
Chapterq7:qHemodynamicqDisorders
Chapterq8:qEnvironmentalqandqNutritionalqPathology
Chapterq9:qInfectiousqandqParasiticqDiseases
SectionqII:qPathogenesisqofqSystemicqConditionsqExpandableqsection
Chapterq10:qAging
Chapterq11:qSystemicqAutoimmuneqDiseases
Chapterq12:qSepsis
Chapterq13:qObesityqandqDiabetesqMellitus
Chapterq14:qTheqPathologyqofqPregnancy
Chapterq15:qTheqAmyloidoses
SectionqIII:qDiseasesqofqIndividualqOrganqSystemsExpandableqsection
Chapterq16:qBloodqVessels
Chapterq17:qTheqHeart
Chapterq18:qTheqRespiratoryqSystem
Chapterq19:qTheqGastrointestinalqTract
Chapterq20:qTheqLiverqandqBiliaryqSystem
Chapterq21:qTheqPancreas
Chapterq22:qTheqKidney
Chapterq23:qTheqLowerqUrinaryqTractqandqMaleqReproductiveqSystem
Chapterq24:qTheqFemaleqReproductiveqSystemqandqPeritoneum
Chapterq25:qTheqBreast
Chapterq26:qHematopathology
Chapterq27:qTheqEndocrineqSystem
Chapterq28:qTheqSkin
Chapterq29:qTheqHeadqandqNeck
Chapterq30:qBones,qJointsqandqSoftqTissue
Chapterq31:qSkeletalqMuscleqandqPeripheralqNervousqSystem
Chapterq32:qTheqCentralqNervousqSystem
Chapterq33:qTheqEye
Chapterq34:qForensicqPathology
,Rubin'sqPathology:qClinicopathologicqFoundationsqofqMedicineqChapte
rq1:qCellqAdaptation,qInjuryqandqDeath
Ischemiaqandqotherqtoxicqinjuriesqincreaseqtheqaccumulationqofqintracellularqcalciumqasqaqresultqof:
1.
A) releaseqofqstoredqcalciumqfromqtheqmitochondria.
B) improvedqintracellularqvolumeqregulation.
C) decreasedqinfluxqacrossqtheqcellqmembrane.
D) attractionqofqcalciumqtoqfattyqinfiltrates.
Theqpatientqisqfoundqtoqhaveqliverqdisease,qresultingqinqtheqremovalqofqaqlobeqofqhisqliver.qAdapt
2. ationqtoqtheqreducedqsizeqofqtheqliverqleadsqtoq_ ofqtheqremainingqliverqcells.
A) metaplasia
B) organqatrophy
C) compensatoryqhyperplasia
D) physiologicqhypertrophy
Aqpersonqeatingqpeanutsqstartsqchokingqandqcollapses.qHisqairwayqobstructionqisqpartiallyqcl
eared,qbutqheqremainsqhypoxicquntilqheqreachesqtheqhospital.qTheqprolongedqcellqhypoxiaqca
3. usedqaqcerebralqinfarctionqandqresulting _qinqtheqbrain.
A) caspaseqactivation
B) coagulationqnecrosis
C) rapidqphagocytosis
D) proteinqp53qdeficiency
Bacteriaqandqvirusesqcauseqcellqdamageqby
4. ,qwhichqisquniqueqfromqtheqintracellularq
damageqcausedqbyqotherqinjuriousqagents.
A) disruptingqtheqsodium/potassiumqATPaseqpump
B) interruptingqoxidativeqmetabolismqprocesses
C) replicatingqandqproducingqcontinuedqinjury
D) decreasingqproteinqsynthesisqandqfunction
Theqpatientqhasqaqprolongedqinterruptionqinqarterialqbloodqflowqtoqhisqleftqkidney,qcausingqhypoxic
5. qcellqinjuryqandqtheqreleaseqofqfreeqradicals.qFreeqradicalsqdamageqcellsqby:
A) destroyingqphospholipidsqinqtheqcellqmembrane.
B) alteringqtheqimmuneqresponseqofqtheqcell.
C) disruptingqcalciumqstorageqinqtheqcell.
, D) inactivationqofqenzymesqandqmitochondria.
6. Injuredqcellsqhaveqimpairedqflowqofqsubstancesqthroughqtheqcellqmembraneqasqaqresultqof:
A) increasedqfatqload.
B) alteredqpermeability.
C) alteredqglucosequtilization.
D) increasedqsurfaceqreceptors.
7. Reversibleqadaptiveqintracellularqresponsesqareqinitiatedqby:
A) stimulusqoverload.
B) geneticqmutations.
C) chemicalqmessengers.
D) mitochondrialqDNA.
8. Injuredqcellsqbecomeqveryqswollenqasqaqresultqof:
A) increasedqcellqproteinqsynthesis.
B) alteredqcellqvolumeqregulation.
C) passiveqentryqofqpotassiumqintoqtheqcell.
D) blebqformationqinqtheqplasmaqmembrane.
Aqdiabeticqpatientqhasqimpairedqsensation,qcirculation,qandqoxygenationqofqhisqfeet.qHeqstepsqonqaq
pieceqofqglass,qtheqwoundqdoesqnotqheal,qandqtheqareaqtissueqbecomesqnecrotic.qTheqnecroticqcellq
9. deathqisqcharacterizedqby:
A) rapidqapoptosis.
B) cellularqrupture.
C) shrinkageqandqcollapse.
D) chronicqinflammation.
Aq99-year-
10. oldqwomanqhasqexperiencedqtheqdeclineqofqcellqfunctionqassociatedqwithqage.qAqgroupqofqtheorie
sqofqcellularqagingqfocusqonqprogrammed:
A) changesqwithqgeneticqinfluences.
B) eliminationqofqcellqreceptorqsites.
C) insufficientqtelomeraseqenzyme.
D) DNAqmutationqorqfaultyqrepair.
Anq89-year-
oldqfemaleqpatientqhasqexperiencedqsignificantqdecreasesqinqherqmobilityqandqstaminaqduringqaq3
-
11. weekqhospitalqstayqforqtheqtreatmentqofqaqfemoralqheadqfracture.qWhichqofqtheqfollowingqphenom
enaqmostqlikelyqaccountsqforqtheqpatientsqdecreaseqinqmuscleqfunctionqthatqunderliesqherqreduced
RUBIN'S PATHOLOGY: CLIN
q q
ICOPATHOLOGIC FOUNDATI q
ONS OF MEDICINE q q
7th Edition By David S. Strayer, Emanuel Rubin
q q q q q q q
TESTBANK q
,TestqBankqRubin'sqPathology:qClinicopathologicqFoundationsqofqMedicineq7thq Edition
TableqofqContents:
Chapterq1:qCellqAdaptation,qInjuryqandqDeath
Chapterq2:qInflammation
Chapterq3:qRepair,qRegenerationqandqFibrosis
Chapterq4:qImmunopathology
Chapterq5:qNeoplasia
Chapterq6:qDevelopmentalqandqGeneticqDiseases
Chapterq7:qHemodynamicqDisorders
Chapterq8:qEnvironmentalqandqNutritionalqPathology
Chapterq9:qInfectiousqandqParasiticqDiseases
SectionqII:qPathogenesisqofqSystemicqConditionsqExpandableqsection
Chapterq10:qAging
Chapterq11:qSystemicqAutoimmuneqDiseases
Chapterq12:qSepsis
Chapterq13:qObesityqandqDiabetesqMellitus
Chapterq14:qTheqPathologyqofqPregnancy
Chapterq15:qTheqAmyloidoses
SectionqIII:qDiseasesqofqIndividualqOrganqSystemsExpandableqsection
Chapterq16:qBloodqVessels
Chapterq17:qTheqHeart
Chapterq18:qTheqRespiratoryqSystem
Chapterq19:qTheqGastrointestinalqTract
Chapterq20:qTheqLiverqandqBiliaryqSystem
Chapterq21:qTheqPancreas
Chapterq22:qTheqKidney
Chapterq23:qTheqLowerqUrinaryqTractqandqMaleqReproductiveqSystem
Chapterq24:qTheqFemaleqReproductiveqSystemqandqPeritoneum
Chapterq25:qTheqBreast
Chapterq26:qHematopathology
Chapterq27:qTheqEndocrineqSystem
Chapterq28:qTheqSkin
Chapterq29:qTheqHeadqandqNeck
Chapterq30:qBones,qJointsqandqSoftqTissue
Chapterq31:qSkeletalqMuscleqandqPeripheralqNervousqSystem
Chapterq32:qTheqCentralqNervousqSystem
Chapterq33:qTheqEye
Chapterq34:qForensicqPathology
,Rubin'sqPathology:qClinicopathologicqFoundationsqofqMedicineqChapte
rq1:qCellqAdaptation,qInjuryqandqDeath
Ischemiaqandqotherqtoxicqinjuriesqincreaseqtheqaccumulationqofqintracellularqcalciumqasqaqresultqof:
1.
A) releaseqofqstoredqcalciumqfromqtheqmitochondria.
B) improvedqintracellularqvolumeqregulation.
C) decreasedqinfluxqacrossqtheqcellqmembrane.
D) attractionqofqcalciumqtoqfattyqinfiltrates.
Theqpatientqisqfoundqtoqhaveqliverqdisease,qresultingqinqtheqremovalqofqaqlobeqofqhisqliver.qAdapt
2. ationqtoqtheqreducedqsizeqofqtheqliverqleadsqtoq_ ofqtheqremainingqliverqcells.
A) metaplasia
B) organqatrophy
C) compensatoryqhyperplasia
D) physiologicqhypertrophy
Aqpersonqeatingqpeanutsqstartsqchokingqandqcollapses.qHisqairwayqobstructionqisqpartiallyqcl
eared,qbutqheqremainsqhypoxicquntilqheqreachesqtheqhospital.qTheqprolongedqcellqhypoxiaqca
3. usedqaqcerebralqinfarctionqandqresulting _qinqtheqbrain.
A) caspaseqactivation
B) coagulationqnecrosis
C) rapidqphagocytosis
D) proteinqp53qdeficiency
Bacteriaqandqvirusesqcauseqcellqdamageqby
4. ,qwhichqisquniqueqfromqtheqintracellularq
damageqcausedqbyqotherqinjuriousqagents.
A) disruptingqtheqsodium/potassiumqATPaseqpump
B) interruptingqoxidativeqmetabolismqprocesses
C) replicatingqandqproducingqcontinuedqinjury
D) decreasingqproteinqsynthesisqandqfunction
Theqpatientqhasqaqprolongedqinterruptionqinqarterialqbloodqflowqtoqhisqleftqkidney,qcausingqhypoxic
5. qcellqinjuryqandqtheqreleaseqofqfreeqradicals.qFreeqradicalsqdamageqcellsqby:
A) destroyingqphospholipidsqinqtheqcellqmembrane.
B) alteringqtheqimmuneqresponseqofqtheqcell.
C) disruptingqcalciumqstorageqinqtheqcell.
, D) inactivationqofqenzymesqandqmitochondria.
6. Injuredqcellsqhaveqimpairedqflowqofqsubstancesqthroughqtheqcellqmembraneqasqaqresultqof:
A) increasedqfatqload.
B) alteredqpermeability.
C) alteredqglucosequtilization.
D) increasedqsurfaceqreceptors.
7. Reversibleqadaptiveqintracellularqresponsesqareqinitiatedqby:
A) stimulusqoverload.
B) geneticqmutations.
C) chemicalqmessengers.
D) mitochondrialqDNA.
8. Injuredqcellsqbecomeqveryqswollenqasqaqresultqof:
A) increasedqcellqproteinqsynthesis.
B) alteredqcellqvolumeqregulation.
C) passiveqentryqofqpotassiumqintoqtheqcell.
D) blebqformationqinqtheqplasmaqmembrane.
Aqdiabeticqpatientqhasqimpairedqsensation,qcirculation,qandqoxygenationqofqhisqfeet.qHeqstepsqonqaq
pieceqofqglass,qtheqwoundqdoesqnotqheal,qandqtheqareaqtissueqbecomesqnecrotic.qTheqnecroticqcellq
9. deathqisqcharacterizedqby:
A) rapidqapoptosis.
B) cellularqrupture.
C) shrinkageqandqcollapse.
D) chronicqinflammation.
Aq99-year-
10. oldqwomanqhasqexperiencedqtheqdeclineqofqcellqfunctionqassociatedqwithqage.qAqgroupqofqtheorie
sqofqcellularqagingqfocusqonqprogrammed:
A) changesqwithqgeneticqinfluences.
B) eliminationqofqcellqreceptorqsites.
C) insufficientqtelomeraseqenzyme.
D) DNAqmutationqorqfaultyqrepair.
Anq89-year-
oldqfemaleqpatientqhasqexperiencedqsignificantqdecreasesqinqherqmobilityqandqstaminaqduringqaq3
-
11. weekqhospitalqstayqforqtheqtreatmentqofqaqfemoralqheadqfracture.qWhichqofqtheqfollowingqphenom
enaqmostqlikelyqaccountsqforqtheqpatientsqdecreaseqinqmuscleqfunctionqthatqunderliesqherqreduced