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BNKN602 EXAM QUESTIONS AND ANSWERS WITH COMPLETE SOLUTIONS GRADED A++

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BNKN602 EXAM QUESTIONS AND ANSWERS WITH COMPLETE SOLUTIONS GRADED A++ Autonomic dysreflexia patho Autonomic dysreflexia occurs in people with spinal cord injury at or above T6. 1) A noxious stimulant below the level of injury occurs, this stimulant could be a distended bladder or bowel, an ingrown toenail, kinked catheter. 2) This results in a extreme sympathetic response causing widespread vasoconstriction 3) The vasoconstriction of the blood vessels cause a rapidly increasing blood pressure 4) The brain is signaled by baroreceptors about the bodies hypertensive crisis and in an attempt to lower the blood pressure, the heart rate is decreased and the person can become bradycardic 5) inhibitory signals are unable to pass below the injury from being transmitted further down the spinal cord. If noxious stimulant is not removed, it can result in significant complications such as stroke, seizures, myocardial ischemia or death. Common causes of autonomic dysreflexia Urinary retention Over distended bladder or bowel Pressure ulcers Kidney stones Ingrown toenail Interventions for autonomic dysreflexia • Put the person into sitting position 90◦ to help lower the BP • Assess for the probable cause and remove • Continue to assess vital signs (Q2-3min BP) • Administer GTN as prescribed if SBP 150mmHg (short acting anti-hypertensive medications only) • Loosen clothing Cushing's triad Signs of increased intracranial pressure: 1. hypertension 2. bradycardia 3. irregular respirations Symptoms of a stroke Face--Sudden weakness and/or numbness of face, Arms--Sudden weakness of the arm (and/or leg) Speech--Difficulty speaking, words jumbled, or lost voice Interventions for a stroke • To monitor for further neurological deterioration and maintain patient safety • Maintain head of bed 30◦ • Maintain systolic blood pressure within prescribed parameters (often 180mmHg) • Continuous neurological observations • Ensure correct positioning of paralysed limbs • Ensure patient is kept NBM until swallow is assessed by Speech and Language Therapist • Administer prescribed aspirin orally or enterally • Turn the patient regularly Prolonged hypertension results in Permanent damage to the arterial walls Ischaemic heart disease A chronic disease caused by insufficient blood supply (thus O2) to the heart muscles via the coronary arteries typically resulting from Atherosclerosis. Ischaemic heart disease- Interventions -Monitor BP -Encourage low fat, high protein diet Angina chest pain due to obstruction of a major coronary artery resulting in decreased oxygen supply Myocardial infarction- signs and symptoms -Chest pain - may radiate down arm -shortness of breath -diaphoresis (sweating) -Palpitations -Increased Jugular venous distention if MI has caused HF -Weak thready pulse -Pale, cyanosis -Hypoxaemia

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BNKN602 EXAM QUESTIONS AND ANSWERS WITH COMPLETE

SOLUTIONS GRADED A++


Autonomic dysreflexia patho

Autonomic dysreflexia occurs in people with spinal cord injury at or above T6.

1) A noxious stimulant below the level of injury occurs, this stimulant could be a

distended bladder or bowel, an ingrown toenail, kinked catheter.

2) This results in a extreme sympathetic response causing widespread vasoconstriction

3) The vasoconstriction of the blood vessels cause a rapidly increasing blood pressure

4) The brain is signaled by baroreceptors about the bodies hypertensive crisis and in an

attempt to lower the blood pressure, the heart rate is decreased and the person can

become bradycardic

5) inhibitory signals are unable to pass below the injury from being transmitted further

down the spinal cord.

If noxious stimulant is not removed, it can result in significant complications such as

stroke, seizures, myocardial ischemia or death.

Common causes of autonomic dysreflexia

Urinary retention

Over distended bladder or bowel

Pressure ulcers

Kidney stones

Ingrown toenail

,Interventions for autonomic dysreflexia

• Put the person into sitting position 90◦ to help lower the BP

• Assess for the probable cause and remove

• Continue to assess vital signs (Q2-3min BP)

• Administer GTN as prescribed if SBP > 150mmHg (short acting anti-hypertensive

medications only) • Loosen clothing

Cushing's triad

Signs of increased intracranial pressure:

1. hypertension

2. bradycardia

3. irregular respirations

Symptoms of a stroke

Face--Sudden weakness and/or numbness of face,

Arms--Sudden weakness of the arm (and/or leg)

Speech--Difficulty speaking, words jumbled, or lost voice

Interventions for a stroke

• To monitor for further neurological deterioration and maintain patient safety

• Maintain head of bed 30◦

• Maintain systolic blood pressure within prescribed parameters (often < 180mmHg)

• Continuous neurological observations

• Ensure correct positioning of paralysed limbs

• Ensure patient is kept NBM until swallow is assessed by Speech and Language

Therapist

,• Administer prescribed aspirin orally or enterally

• Turn the patient regularly

Prolonged hypertension results in

Permanent damage to the arterial walls

Ischaemic heart disease

A chronic disease caused by insufficient blood supply (thus O2) to the heart muscles via

the coronary arteries typically resulting from Atherosclerosis.

Ischaemic heart disease- Interventions

-Monitor BP

-Encourage low fat, high protein diet

Angina

chest pain due to obstruction of a major coronary artery resulting in decreased oxygen

supply

Myocardial infarction- signs and symptoms

-Chest pain - may radiate down arm

-shortness of breath

-diaphoresis (sweating)

-Palpitations

-Increased Jugular venous distention if MI has caused HF

-Weak thready pulse

-Pale, cyanosis

-Hypoxaemia

, -Nausea

-SOB, dyspnoea

Ischaemic heart disease- Signs and symptoms

-angina

-dizziness

-nausea

-SOB

-^HR

Myocardial infarction (MI)

Heart attack; death of myocardial tissue (infarction) caused by ischemia (loss of blood

flow) as a result of an occlusion (plugging) of a coronary artery; usually caused by

atherosclerosis

Myocardial infarction interventions

-Interventions for MI



-Monitor vital signs record and report

-Administer prescribed 02

-Administer prescribed GTN to promote vasodilation and therefore increase 02 delivery

-Administer prescribed aspirin to inhibit platelet formation of thrombus

-Administer prescribed opioids or analgesia to decrease chest pain and regularly

elevate pt response

-Reassure pt and family to reduce level of anxiety, therefore decrease WOB and

decrease RR

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